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    Cyanotic Congenital Heart Diseases

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    Gubara Mohamed Zain
    Cyanotic congenital heart diseases cause blue discoloration of the skin and mucous membranes due to high concentrations of deoxygenated hemoglobin. There are two types: central cyanosis affecting the mouth and tongue, often due to circulatory or respiratory issues; and peripheral cyanosis in the fingers and extremities. Cyanotic congenital heart defects produce cyanosis by allowing deoxygenated blood to bypass the lungs and enter systemic circulation, such as through right ventricular outflow obstructions with right-to-left shunts, bidirectional shunts, or malpositioned great arteries. One such defect, Tetralogy of Fallot, consists of pulmonary stenosis, a ventricular septal defect, overriding aorta

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    Cyanotic Congenital Heart Diseases

    Uploaded by

    Gubara Mohamed Zain
    144 views9 pages
    Cyanotic congenital heart diseases cause blue discoloration of the skin and mucous membranes due to high concentrations of deoxygenated hemoglobin. There are two types: central cyanosis affecting the mouth and tongue, often due to circulatory or respiratory issues; and peripheral cyanosis in the fingers and extremities. Cyanotic congenital heart defects produce cyanosis by allowing deoxygenated blood to bypass the lungs and enter systemic circulation, such as through right ventricular outflow obstructions with right-to-left shunts, bidirectional shunts, or malpositioned great arteries. One such defect, Tetralogy of Fallot, consists of pulmonary stenosis, a ventricular septal defect, overriding aorta

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    Cyanotic congenital heart diseases cause blue discoloration of the skin and mucous membranes due to high concentrations of deoxygenated hemoglobin. There are two types: central cyanosis affecting the mouth and tongue, often due to circulatory or respiratory issues; and peripheral cyanosis in the fingers and extremities. Cyanotic congenital heart defects produce cyanosis by allowing deoxygenated blood to bypass the lungs and enter systemic circulation, such as through right ventricular outflow obstructions with right-to-left shunts, bidirectional shunts, or malpositioned great arteries. One such defect, Tetralogy of Fallot, consists of pulmonary stenosis, a ventricular septal defect, overriding aorta

    Copyright:

    Attribution Non-Commercial (BY-NC)
    Download as PPT, PDF, TXT or read online from Scribd
    Download as ppt, pdf, or txt
    144 views9 pages

    Cyanotic Congenital Heart Diseases

    Uploaded by

    Gubara Mohamed Zain
    Cyanotic congenital heart diseases cause blue discoloration of the skin and mucous membranes due to high concentrations of deoxygenated hemoglobin. There are two types: central cyanosis affecting the mouth and tongue, often due to circulatory or respiratory issues; and peripheral cyanosis in the fingers and extremities. Cyanotic congenital heart defects produce cyanosis by allowing deoxygenated blood to bypass the lungs and enter systemic circulation, such as through right ventricular outflow obstructions with right-to-left shunts, bidirectional shunts, or malpositioned great arteries. One such defect, Tetralogy of Fallot, consists of pulmonary stenosis, a ventricular septal defect, overriding aorta

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    Cyanotic congenital

    heart diseases
    Cyanosis: is blue discoloration of the
    skin & mucous membranes & requires
    a concentration of deoxygenated
    haemoglobin of >5g/dl; of 2 types
     (1) central: cyanosis of mucous membranes &
    tongue ; often due to circulatory or ventilatory
    problems (hyperoxia test)
     Common causes :
     Respiratory

     CVS

     CNS

     Haematological
     Peripheral : blue tint in finger &
    extremities
     Causes
     All of central Causes of central
     Hypothermia
     Raynuads phenomenon
     HF , hypovolemia
     Hypo glycaemia
     CHD produce cyanosis due to deoxygenated blood
    bypassing the lungs & entering the systemic
    circulation , this can be caused by:
    – Rt ventricular out flow obstruction with Rt – to -
    Lt shunt
    – Bidirectional shunt
    – Malposition of the great arteries
     CCHD account for 25% of all CHDs include :
    1. 1 tetra logy of fallot (T O F)
    2. Total anomalous pulmonary venous connection
    3. Transposition of great arteries
    4. Truncus arteriosus
    5. Tricuspid atresia
    6. Pulmonary atresia
    7. Interrupted aortic arch
    8. Hypoblastic Lt heart syndrome
    9. Epstein's anomalies
    Tetra logy of fallot (T O F )

     Consist of:
    1. Pulmonary stenosis( Rt V outflow obstruction)
    2. VSD
    3. Over-riding of the aorta )
    4. Rt V hypertrophy

    The most common CCHD representing 55-70% of


    CCHD.
     Pathophysiology:
     Determined by the degree of Rt V outflow
    obstruction (the degree of cyanosis, the timing of
    the onset of symptoms &the degree of hypertrophy
     Is usually at both Rt V infndibulum (sub aortic
    area),& pulmonary valve
     Infundibular muscle is hypertrophied gradually with
    age
     Complete obstruction of Rt V ( P Artesia with VSD )
    is classified as sever form of TOF.
     Systemic venous return to Rt A & Rt V is normal
     When the V contracts in presence of stenosis the
    blood shunted through the VSD into the aorta
     Persistent arterial desaturation occur& cyanosis
    result .
     When the obst. Is mild to moderate a balanced
    shunt is present across the VSD & the Pt. may not
    be cyanosed ( a cyanotic [pink] TOF)
     Clinical manifestations :-
     Mild may present initially with HF due to shunt .
     Often cyanosis is not present at birth , with
    increase hypertrophy occurs latter in the first year
    of life .
     In sever cases it is noted immediately .
     In these cases when DA closed circulatory collapse
    may occur .
     Clubbing ,dyspnoea ,paroxysmal hyper cyanotic

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