Scribd Logo
Upload

Welcome to Scribd!

  • 101 views

    Cerebellar Ataxia

    Uploaded by

    efafsadfa
    This document describes various abnormal and pathological gait patterns, including ataxic, antalgic, apopulsive, bouncing, broad-based, festinant/shuffling, hallux limitus, high-steppage, normal, rheumatoid, and scissors gaits. For each gait type, it provides the characteristic features and pathology that would cause that particular gait pattern. Some examples include that an ataxic gait results from cerebellar dysfunction and is uncoordinated with jerky movements, while an antalgic gait occurs due to pain and shows reduced velocity and double limb support.

    Copyright:

    © All Rights Reserved
    Download as DOCX, PDF, TXT or read online from Scribd

    Cerebellar Ataxia

    Uploaded by

    efafsadfa
    101 views2 pages
    This document describes various abnormal and pathological gait patterns, including ataxic, antalgic, apopulsive, bouncing, broad-based, festinant/shuffling, hallux limitus, high-steppage, normal, rheumatoid, and scissors gaits. For each gait type, it provides the characteristic features and pathology that would cause that particular gait pattern. Some examples include that an ataxic gait results from cerebellar dysfunction and is uncoordinated with jerky movements, while an antalgic gait occurs due to pain and shows reduced velocity and double limb support.

    Copyright

    © © All Rights Reserved

    Available Formats

    DOCX, PDF, TXT or read online from Scribd

    Did you find this document useful?

    Is this content inappropriate?

    This document describes various abnormal and pathological gait patterns, including ataxic, antalgic, apopulsive, bouncing, broad-based, festinant/shuffling, hallux limitus, high-steppage, normal, rheumatoid, and scissors gaits. For each gait type, it provides the characteristic features and pathology that would cause that particular gait pattern. Some examples include that an ataxic gait results from cerebellar dysfunction and is uncoordinated with jerky movements, while an antalgic gait occurs due to pain and shows reduced velocity and double limb support.

    Copyright:

    © All Rights Reserved
    Download as DOCX, PDF, TXT or read online from Scribd
    Download as docx, pdf, or txt
    101 views2 pages

    Cerebellar Ataxia

    Uploaded by

    efafsadfa
    This document describes various abnormal and pathological gait patterns, including ataxic, antalgic, apopulsive, bouncing, broad-based, festinant/shuffling, hallux limitus, high-steppage, normal, rheumatoid, and scissors gaits. For each gait type, it provides the characteristic features and pathology that would cause that particular gait pattern. Some examples include that an ataxic gait results from cerebellar dysfunction and is uncoordinated with jerky movements, while an antalgic gait occurs due to pain and shows reduced velocity and double limb support.

    Copyright:

    © All Rights Reserved
    Download as DOCX, PDF, TXT or read online from Scribd
    Download as docx, pdf, or txt
    of 2

    Cerebellar ataxia

    Position sense loss (stamping)


    Distal weakness (slapping)
    Proximal weakness (waddling)
    Apraxia of gait
    Gait pattern
    Apropulsive gait

    Antalgic gait

    Ataxic gait

    Bouncing gait

    Broad-based gait
    Childhood gait

    Festinant or shuffling
    gait

    Hallux limitus gait

    High-steppage gait

    Normal gait
    Rheumatoid gait

    Scissors gait

    Characteristics
    Typical of someone who pronates excessively during the stance phase of gait
    Characteristically inefficient gait pattern; subject does not achieve supination at
    toe off
    Typical of the elderly
    Characteristically shows loss of gait fluidity and ease of movement, due to foot
    pain, generalized pain and disability
    Typical of someone with cerebellar pathology
    Characteristically an uncoordinated gait pattern
    Shows dyssynergy (loss of smoothly coordinated voluntary movements),
    dysrhythmia (abnormal timing and coupling of movements), dysmetria (the
    inability to gauge distance, speed, strength and velocity of movements)
    This results in a wide-based gait with a slow, jerky and irregular cadence,
    variability of stride length and foot placement from step to step, numerous
    postural adjustments and easy loss of balance
    Typical of someone with ankle equinus
    Characteristically shows early heel off (lift) due to soft-tissue contracture of
    Achilles tendon and gastrocnemius complex, shortened stride length, external
    hip rotation, extended knee extension throughout the stance phase, abductory
    twist of heel and forefoot, excessive pronation, elongated propulsive phase,
    forefoot subluxation and medial column collapse

    Typical of a child aged 2-6 years


    Characteristically shows developmental knock knee, abdomen less prominent
    than the toddler gait; foot type approaches the adult form with a more evident
    medial longitudinal arch
    Typical of someone with Parkinson's disease
    Characteristically shows difficulty in initiating gait; rapid, short, shuffling steps
    once mobile, with loss of arm swing, stooped stance, propulsion and retropulsion (small backward steps)
    Typical of someone with functional or actual reduced dorsiflexion at the first
    metatarsophalangeal joint
    Characteristically shows reduced heel lift, obligatory pronation about the
    longitudinal axis of the metatarsal joint, leading to abductory twist and
    increased abducted angle of gait, internal tibial torsion, internal rotation and
    increased transverse plane motion at knee, internal hip rotation, forward pelvic
    tilt and increased lumbar lordosis, thoracic kyphosis and a forward tilt of the
    cervical spine
    Typical of someone with foot drop and/or distal sensory neuropathy
    Characteristically shows increased hip and knee flexion during the swing phase
    of gait in order for the foot, which is plantarflexed at the ankle, to clear the
    ground

    Typical of someone with rheumatoid disease


    Characteristically shows an antalgic pattern with reduced gait velocity due to
    shortened stride length and reduced cadence, with an increased double-support
    phase and reluctance to load the forefoot and altered velocity of the centre of
    pressure profile
    Typical of someone with cerebral palsy
    Characteristically shows spasticity of hip flexors and adductors, with

    Spasticity /
    hemiparesis

    Toddler gait

    hyperreflexia and clonus, internal hip rotation, contraction of the knee flexors
    leading to toe walking and cavovarus foot deformity
    Spasticity: more pronounced in extensor muscles, +/- weakness,
    causes stiff & jerky walking. Toes & shoes scuffed, catching
    level ground. Pace shortens, narrow base maintained. Clonus
    may occur.
    Hemiparesis: Weak leg is circumducted & drags.
    Parkinsons disease muscular rigidity throughout extensors &
    flexors. Power preserved but pace shortens & slows to a shuffle,
    base remains narrow. A stoop & diminished arm swinging
    become apparent.
    Typical of a 12-15-month-old child who has just begun to walk
    Characteristically shows a broad-based gait, with apparent flat foot and bow
    legs, partially flexed knees, lordosis and bulging of the abdomen, forward tilt of
    the body, arms extended sideways at the shoulders and/or the arms lifted up

    You might also like