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    TUGAS KEL.1 Abstract Descriptive

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    veronika winda saskia
    This study aims to evaluate the effects of exercise-induced muscle damage (EIMD) on cardiovascular, metabolic and perceived exertion responses during cycling exercise and time-trial performance. Twenty recreational athletes performed tests to determine VO2 max and power output. They were randomly assigned to a treatment group, which performed muscle-damaging plyometric jumps, or a control group. Before and after the treatment, all participants completed a submaximal cycling test and 4km time-trial while various measurements were recorded. The study hopes to analyze differences between the groups over time using ANOVA to better understand how EIMD impacts endurance exercise performance.

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    TUGAS KEL.1 Abstract Descriptive

    Uploaded by

    veronika winda saskia
    12 views2 pages
    This study aims to evaluate the effects of exercise-induced muscle damage (EIMD) on cardiovascular, metabolic and perceived exertion responses during cycling exercise and time-trial performance. Twenty recreational athletes performed tests to determine VO2 max and power output. They were randomly assigned to a treatment group, which performed muscle-damaging plyometric jumps, or a control group. Before and after the treatment, all participants completed a submaximal cycling test and 4km time-trial while various measurements were recorded. The study hopes to analyze differences between the groups over time using ANOVA to better understand how EIMD impacts endurance exercise performance.

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    contoh abstrak descriptive

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    This study aims to evaluate the effects of exercise-induced muscle damage (EIMD) on cardiovascular, metabolic and perceived exertion responses during cycling exercise and time-trial performance. Twenty recreational athletes performed tests to determine VO2 max and power output. They were randomly assigned to a treatment group, which performed muscle-damaging plyometric jumps, or a control group. Before and after the treatment, all participants completed a submaximal cycling test and 4km time-trial while various measurements were recorded. The study hopes to analyze differences between the groups over time using ANOVA to better understand how EIMD impacts endurance exercise performance.

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    © All Rights Reserved
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    Download as docx, pdf, or txt
    12 views2 pages

    TUGAS KEL.1 Abstract Descriptive

    Uploaded by

    veronika winda saskia
    This study aims to evaluate the effects of exercise-induced muscle damage (EIMD) on cardiovascular, metabolic and perceived exertion responses during cycling exercise and time-trial performance. Twenty recreational athletes performed tests to determine VO2 max and power output. They were randomly assigned to a treatment group, which performed muscle-damaging plyometric jumps, or a control group. Before and after the treatment, all participants completed a submaximal cycling test and 4km time-trial while various measurements were recorded. The study hopes to analyze differences between the groups over time using ANOVA to better understand how EIMD impacts endurance exercise performance.

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    GROUP 2

    VERONIKA WINDA SASKIA ( 183112540120003 )


    YUNITA SARI ( 183112540120004 )
    MERIAM AMRULLAH ( 183112540120015 )
    REGINA SEPTY APRILIA ( 183112540120016)
    HIDA FAUZIAH ( 183112540120023 )
    HOSANNA R.S HUTAGALUNG (183112540120024 )
    LENNI TRISTIANA ( 183112540120025 )

    “This abstract is Descriptive Abstract”

    The effects of exercise-induced muscle damage on cycling


    endurance performance
    The effects of exercise-induced muscle damage (EIMD) on Background
    endurance performance have been studied infrequently, although
    mechanisms by which EIMD might affect endurance performance
    have been identified in some studies (Asp et al., 1998; Journal of
    Physiology, 509, 305-313; Gleeson et al., 1995; Journal of Sport
    Sciences, 13, 471-479).
    The purpose of this study was to evaluate the effects of EIMD on Purpose & Aim
    cardiovascular, metabolic and perceived exertion responses during
    fixed-load cycling exercise and on 4 km time-trial. With
    institutional ethics approval, 20 recreational athlete volunteers (age
    22.9 ± 4.6 years, stature 1.73 ± 0.09 m, body mass 72.3 ± 8.6 kg)
    performed an incremental test to exhaustion on a cycle ergometer
    to determine VO2 max and the power output corresponding to VO2
    max (Pmax).

    Participants were then randomly allocated to a treatment (n = Particular interest / Focus of


    10) and a control group (n = paper
    10). After a minimum of 48 h, participants exercised for 5 min at
    60% Pmax (Pmax60 ) followed immediately by a 4 km time-trial.
    Cardio-respiratory, metabolic and perceptual responses
    were recorded during all tests. Performance measures were
    accompanied by measurements of perceived muscle soreness,
    creatine kinase (CK) and peak isokinetic torque at 60 deg⋅s-1.
    The treatment group then performed muscle-damaging exercise
    comprising of 10 x 10 plyometric jumps while the control group
    performed no exercise for a similar time period. At 48 h
    following the treatment or control condition, participants then
    repeated all measurements. Data will be analysed using a two-
    way repeated measures ANOVA to determine any interaction
    effect of time and group on indirect markers of muscle damage,
    fixed-load exercise and time-trial performance. It is hoped that
    the findings of this study will reaffirm those of Marcora and
    Bosio (2007; Scandinavian Journal of Medicine and Science in
    Sport, 17, 662–671).

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