Biology Project: Sensory Organs
Biology Project: Sensory Organs
Biology Project: Sensory Organs
Sensory
Organs
Name: JOHN SOJAN
Roll No.: 08
Acknowledgement
Index
A. Sensory organs
1. olfactory receptors
2. hearing receptors
3. the sense of sight
4. taste receptors
5. sensory receptors
B. Conclusion
C. bibliography
s
Sensory organs
Olfactory Receptors
Choanal Atresia
According to the National Institutes of Health's
MedlinePlus medical encyclopedia, choanal atresia is the
most common nasal defect affecting newborns, occurring
in one out of every 7,000 births. Its main symptom is
breathing difficulties which result in a bluish color to the
skin, lips and fingernails due to a lack of oxygen. This
disease also leads to problems nursing, nasal blockage
affecting one side of the nose and chest retractions
(sinking of the chest as the baby breathes inward). The
cause of choanal atresia is not known.
Meniere’s Disease
Meniere’s Disease is a disease that affects the inner ear,
specifically the endolymphatic sac, also called the
labyrinth. This semi-circular canal processes sound
signals to the brain and is also responsible for the
perception of balance.
Cholesteatoma
Cholesteatoma is characterized by abnormal growth of
skin in the middle ear. It is usually caused by chronic
infection. Its symptoms include growth of small pouches
and cysts in the inner ear, leading to hearing loss. In
severe cases, paralysis of facial muscles and dizziness
may occur, but only rarely.Any infection present is
treated with antibiotics.
Otosclerosis
Otosclerosis is the buildup of spongy or bone-like tissue
in the middle ear that prevents the ossicles, namely the
stapes in the middle ear, from working properly. The
impaired movement and function reduces the sound that
actually reaches the ear. Otosclerosis usually results
in conductive hearing loss, a hearing loss caused by a
problem in the outer or middle ear.
If the buildup of tissue spreads to the inner ear, it is
called Cochlear Otosclerosis. This can cause
permanent sensorineural hearing impairment due to
interference with how the nerves in this part of the ear
work.
Scientists aren’t sure about the exact cause but there is
some research suggesting a relationship between
otosclerosis and the hormonal changes associated with
pregnancy and also with viruses.
Treatment for people who are diagnosed with
otosclerosis depends on the extent of hearing loss and
may include surgery to replace some or all of the ossicles
with artificial ones. It is important to discuss the risks
and possible complications of this and any procedure, as
well as the benefits, with a doctor and a surgeon.
If the hearing loss is mild, surgery may not be an option
but a properly fitted hearing aid may help some people
with otosclerosis. A hearing aid is designed to
compensate for a hearing loss by amplifying sound.
C.
When you look at an eye, the iris is the colored part. The
iris actually is a pigmented muscle that controls the size
of the pupil, which dilates to allow more light into the
eye or contracts to allow less light into the eye. The iris
and pupil are covered by the cornea.
The eye is the organ of vision. It has a complex structure
consisting of a transparent lens that focuses light on the
retina. The retina is covered with two basic types of
light-sensitive cells-rods and cones. The cone cells are
sensitive to color and are located in the part of the retina
called the fovea, where the light is focused by the lens.
The rod cells are not sensitive to color, but have greater
sensitivity to light than the cone cells. These cells are
located around the fovea and are responsible for
peripheral vision and night vision. The eye is connected
to the brain through the optic nerve. The point of this
connection is called the "blind spot" because it is
insensitive to light. Experiments have shown that the
back of the brain maps the visual input from the eyes.
Myopia
Myopia is the term used to define short sightedness.
Light from a distant object forms an image before it
reaches the retina. This could be because the eye is too
long, or the cornea or crystalline lens is too strong.
Hypermetropia
Myasthenia Gravis
Myasthenia Gravis is an autoimmune disease of the
muscles leading to weakness and easy fatigability.
Commonly (80% to 90% of cases), the disorder presents
itself with drooping upper eyelids (ptosis) and double
vision. The symptoms are typically highly variable,
intermittent, and may be asymmetrical between the two
eyes. There may also be speech and swallowing
difficulties, problems with facial expression, and
weakness of the muscles of the arms and legs. The
symptoms tend to improve after rest and in the morning,
and tend to be worse later in the day and after exercise.
The underlying problem is the development of antibodies
to neuromuscular receptors present in all muscles
(acetylcholine receptors). There is some relationship with
the thymus gland, and there is a positive family history in
about 5% of cases. Symptoms tend to appear in the
middle age years for men and women. Some cases have
occurred after bone marrow transplantation.
Patients with prominent ocular symptoms often develop
a fairly severe drooping of one or both upper eyelids,
worse when tired, that may interfere with vision. The
double vision associated with myasthenia can be variable,
and does not usually fit into a typical pattern of any one
specific eye muscle being involved. If the drooping
lessens by 2 millimeters or more, myasthenia may be a
cause. There are other office tests that can be performed
to aid in diagnosis (Tensilon test).
Treatment of the disorder has typically been with drugs
that block the enzyme that degrades the neurotransmitter
acetylcholine at the neuromuscular junction. Thus, the
neurotransmitter will stay in the junction longer, and is
better able to stimulate the muscle to contract. Steroids
and other immune suppressants such as azathioprine
have been successful in reducing ocular symptoms, and
in slowing the worsening of the disease over time. These
medications are not without side effects, and need to be
closely monitored by the treating physician. In some
cases, removal of the thymus gland may help the
disorder.
Lyme Disease
Lyme disease is a multi-system disorder caused by an
immune reaction to the spirochete Borrelia burgdorferi
transmitted by the Dear tick (Ixodes dammini). There are
common ocular complications during all stages of Lyme
disease. The disease is most common in the Northeast
and upper Midwest United States. It involves joint pain
and dermatological and ophthalmic findings, as well as
neurologic and cardiac abnormalities. Three stages of
Lyme disease have been described, which may overlap:
Stage 1: The earliest stage includes a flu-like illness with
a typical expanding "bull's-eye" rash. This may or may
not be associated with a known tick bite. Some patients
can develop conjunctivitis during this stage.
Stage 2: The second stage of the disease (after weeks to
months of the disease) includes cardiac involvement (8%)
and neurologic involvement (15%). This can include
meningitis and paralysis of cranial nerves. Paralysis of
the third or sixth cranial nerves affect eye movement, and
lead to double vision. Paralysis of the seventh cranial
nerve causes Bell's palsy, or drooping of one side of the
face. During this stage inflammatory ocular disorders
such as iritis, retinal vasculitis, chorioretinitis, and optic
disc edema can occur. These conditions can cause a loss
of vision.
Stage 3: The last stage of the disease, starting within 2
weeks to 2 years of the infection, include arthritis and
chronic neurologic syndromes. This includes fatigue
syndromes and focal central nervous system disorders.
Ocular findings in this stage include corneal
inflammation (keratitis) and double vision.
Treatment of Lyme disease is by commonly available
antibiotics. Longer treatment is required for cases with
neurological involvement, and intravenous antibiotics are
required for severe neurological involvement and
arthritis.
Taste recopters
Leukoplakia
Sensory Receptors
Psoriasis
Steroid creams.
Moisturizers for dry skin.
Coal tar (a common treatment for scalp psoriasis;
available in lotions, creams, foams, shampoos, and
bath solutions)
Vitamin D cream (a strong kind ordered by your
doctor; vitamin D in foods and pills has no effect)
Retinoid creams
Eczema
Moisturize frequently.
Avoid sudden changes in temperature or humidity.
Avoid sweating or overheating.
Reduce stress.
Avoid scratchy materials, such as wool.
Avoid harsh soaps, detergents, and solvents.
Skin Cancer
Conclusion
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