ORAL PATHOLOGY Practical Specimens

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PRACTICAL – CAST SPECIMEN.

DR. BHAVANI.S.N.
OVER RETAINED DECIDUOUS TEETH
A primary tooth is retained beyond the time of normal exfoliation
Cause:
– Congenitally absence of successor teeth in individuals with persistent
primary teeth
– Impaction of successor teeth
– Translation or transmigration of successor teeth
– Existence of pathology, such as cysts, tumours, and odontoma under
the primary tooth that results in the impaction of successor teeth
– Microdontia of permanent dentition, partially or totally
Clinical features:
The mandibular second primary molar, followed by the maxillary deciduous canine
Treatment
– Extraction
– Filling
– Root treatment
– Periodontal treatment
– Prosthetic restoration

[ Dr Ali Murat Aktan European Journal of Orthodontics 34 (2012) 208–212]
.
TORI
Exostosis of bone
Cause:
Genetic
Environmemtal factors
TORUS MANDIBULARIS:
Bony protuberance on the lingual aspect of Mandible.
Clinical features:
Elderly adults
Male
Types
Single
multiple nodule.
Overlying mucosa may become ulcerated.
Radiopacity superimposed on the roots of the teeth.
Treatment:
Not needed.
Surgical removal
TORI
TORUS PALATINUS:
Exostosis that occurs in the midline of the vault of the hard palate.
Clinical features:
Bony hard mass that arise along the midline suture of the hard palate.
Types
Flat
Spindle
Lobular.
H/P:
Mass of dense, lamellar, cortical bone.
Treatment:
Surgical removal
MICRODONTIA
Unusually small teeth

Etiology : Genetic factors


Environmental factors
TYPES:
The term “microdontia” should be applied only when the teeth are physically
smaller than usual
Relative microdontia - Macrognathia
Diffuse true microdontia – commonly seen in down syndrome, pituitary
dwarfism
Isolated microdontia – commonly seen in maxillary lat .incisors –peg
shaped laterals

Treatment
not necessary
peg shaped laterals – porcelain crowns
SUPERNUMERARY TEETH /HYPERDONTIA
CAUSE:
• Hyper activity of dental lamina
• Due to third tooth bud arising from dental lamina.
• Splitting of permanent tooth bud.

Syndromes
a) Gardner
b) Sturge – weber
c) Cleidocranial dysplasia
d) Oral – Facial – Digital , type I
SUPERNUMERARY TEETH
Clinical features:
Single tooth hyperdontia
Permanent dentition - 90% in the maxillary anterior region
Most common site –Maxillary incisor region ,
followed by Maxillary 4th molar
Mandibular 4th molar
Premolars
Canines and
Lateral incisors
SUPERNUMERARY TEETH
First two decades of life
Male
Permanent tooth

Classification depending on morphology and location:


– Conical
– Tuberculate
– Supplemental
– Odontoma
SUPERNUMERARY TEETH
Multiple supernumerary teeth – most often in the premolar region followed
by molar and anterior region

. Supernumerary teeth in the maxillary anterior region –


mesiodens

. Accessory 4th molar – distomolar or distodens

. Supernumerary tooth – lingual or buccal to molar - paramolar


.
.
OLIGODONTIA
• ANODONTIA : Total lack of tooth development
• HYPODONTIA : Lack of development of one or more teeth
• OLIGODONTIA : Lack of development of six or more teeth

Cause:
Environmental factors:
Trauma ,infection, radiation, chemotherapeutic medications, endocrine disturbances
and severe intrauterine disturbances have been associated with missing teeth.
Genetic factors eg. Syndromes associated
a) Down
b) Crouzon
c) Ectodermal dysplasia
d) Ehlers – Danlos
e) Gorlin
Hypodontia –
TRUE
FALSE

. Hypodontia in deciduous dentition – mandibular incisors

. Missing teeth in permanent dentition – 3rd molars (most common)


followed by
second premolars and lat. Incisors

. Females
Hypodontia is associated with
Microdontia
Reduced alveolar development
Retained primary tooth.
THANK YOU

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