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Moduleiii:Summativeevaluation: Pheochromocytoma. (2020) - N Ational Library of M Edicine

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M OD U LE I I I : SU M M AT I V E EVA LUAT I ON

LEGEN DS:

Risk Factors Non-modifiable Factors hyperpigmentation


·Endocrine disturbances (Adrenal stimutes cortex activate ACTH (axillae and intertriginou
pathophysiology Mass) areas)
·Pheochomocytoma blurred vision
Diagnostic

Clinical Manifestation dysfunction of various tumor


suppressor and or oncogene
proteins headache and
SECONDARY nape paiins
HYPERTENSION decreased O2

uncontrolled proliferation of the


chromaffin cells in the medulla of
the adrenal glands revelaed adrenal
mass increased BP
Dizziness

Adenoma formation abdominal ultrasounds

BP: 190/120
sustained paroxysmal mmHg, 170-180 mmHg
overproduction of epinephrine and hypertension SBP, 100-110 mmHg
noripheniphrine from the adenoma DBP
increase vasoconstriction of
peripheral blood vessel

hyperstimulation of adrenergic
episodic hyper activity of the
hypertimultion of G protein coupled receptor receptors of the cardiac
sympathetic nervous system result to elvation in
incolved in metabolic processes myocytes
arterial pressure

tachycardia
(102 bpm, +3) and
increase secretions from the diaphoresis
palpitations
eccrine sweat glands (excessive sweats)
increase ability to mobilize glucose into the increase right ventricle afterload
bloodstream through enhanced lipolysis
glycogeolysis and gluconuogenesis

decrease right ventricle cardiac output

weakness

increased residual volume in right heart


backup of blood in systemic circulatiion
after contractions

increase blood volume venous system

increase volume and BP capillaries

fluid pushed from vessel into


interstistial of tissue

mild non pitting


bipedal edema

Reference:
Pheochromocytoma. (2020). N ational Library of M edicine.
https://medlineplus.gov/ency/article/000340.htm

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