Acute Conditions of The Neonates
Acute Conditions of The Neonates
Acute Conditions of The Neonates
Nursing Implementation
Prevent infection by:
• Hand washing
• Wearing proper attire
• Antibiotics as ordered
• Avoiding exposure to infected personnel Meconium can cause severe respiratory
• Isolating infected Newborns distress in 3 ways:
Keep warm • Causes Inflammation of bronchioles
• Maintain in isolette w/ high humidity o Because it is a foreign substance
✔ Incubator: 40 -70% • Can block small bronchioles by mechanical
✔ High humidity: 55 – 65% plugging
• Monitor temp per axilla o Ball-valve action: air is allowed in but
• Prevent heat loss cannot be exhaled
o hyperinflated lungs
Give supportive care to parents o ed pulmonary perfusion
• Allow verbalization of feelings and concern o ed hypoxia
• Explain special procedures • Causes in surfactant production thru lung
• Inform of results and progress trauma
• Encourage participation in care; provide
positive feedbacks Assessment
• May demonstrate signs of fetal distress during
Meconium Aspiration Syndrome (MAS) labor and delivery
• Group of symptoms that occur when the • Apgar score less than 6 @ 1 and 5 minutes o
fetus or newborn aspirates meconium- Immediate signs of respiratory distress @
stained amniotic fluid into the lungs • delivery (cyanosis, tachypnea, retractions) o
• Etiology: Over-distended, barrel-shaped chest
• prolonged labor fetus expels • Diminished breath sounds
meconium into amniotic fluid (esp. • Yellow staining of skin, nails, umbilical cord
w/ cord compression)
• If Asphyxia and acidosis occur Priority Dx: Ineffective gas exchange
fetus gasps drawing meconium- Interventions:
stained amniotic fluid into lungs • Suction oropharynx then nasopharynx after
• 1st breath before nose and mouth is neonate’s head is born to remove as much
suctioned meconium-stained fluid meconium as possible
in upper airway passages is drawn • Place infant under radiant warmer
into the lungs • Administer O2 to maintain adequate PO2 and
• Aspiration of meconium into the • O2 saturation
tracheobronchial tree in utero or during the • Perform chest physiotherapy routinely
Sepsis
• Systemic response to infection with bacteria o Risk factors
Can also result from viral or fungal infections • Resolution of enclosed hemorrhage
• Causes SYSTEMIC Inflammatory Response (cephalhematoma, large amount of bruising
Syndrome (SIRS) due to the endotoxin of the from difficult delivery)
bacteria that causes tissue damage • Infection/sepsis
• If untreated septic shock, multi-organ • Dehydration
dysfunction syndrome, DEATH • Breastfeeding – pregnanediol in breast milk
renders glucorynyl transferase ineffective in
Manifestations: conjugating bilirubin
• Fever, chills, tachypnea, tachycardia, • Poor meconium/stool passage
neurological signs (lethargy)
• Hypotension – ominous/threatening sign Assessment
✔ Indicates body is unable to compensate • Pathologic jaundice
adequately ✔ Occurs in the 1st 24 hours
and cardiorespiratory arrest is about to occur ✔ Duration: lasts more than a week
• Lab tests: ✔ Dangerous levels @ w/c kernicterus may set
✔ (+) blood cultures it:
✔ Reduced fibrinogen and thrombocyte levels • Full-term: 20 mg/100 mL or above
✔ Presence of immature WBC • Preterm: 15 mg/ 100 mL or above
✔ Neutropenia (neutrophil 1000/mm3)
Assessment: Kernicterus
Nursing Responsibilities ✔ Signs of kernicterus:
• Monitoring neurological status and VS • Sluggish-to-absent Moro reflex
• Observing for shock; • Opisthotonus
• Maintaining strict standard and expanded • Severe lethargy
precautions (masks, gowns, gloves) • Projectile vomiting
• Antibiotics IV • Tense, bulging fontanel; high-pitched cry •
Apnea
• Immunization against H. influenzae (Hib)
• Convulsion – late sign
bet. 2 mos. – 4 years
• Irritability
HYPERBILIRUBINEMIA • Increasing serum bilirubin
• HYPER - “excess”; BILIS – “bile”; RUBOR
– “red”; EMIA – “blood” Nursing Diagnoses :
• Excessive levels of serum bilirubin greater • Fluid volume deficit r/t decreased intake,
than 12 – 13 mg/100 mL loose stools, and increased insensible
▪ Normal: 2 – 6 mg/100 mL, not to water loss
exceed 12 mg/100 mL • Impaired parenting r/t interruption in
bonding between infant and parents
Physiology secondary to separation
Clinical features
• Death occurs during sleep, and
• Infant does not cry or make other sounds of
• distress
• How it happens?
• Current theories focus on neurologic
immaturity related to the infant’s inability to
sense and regulate oxygenation status
ultimately leading to respiratory arrest