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Weight
Term infants are those born after the beginning of week 38 and before week
42 of pregnancy (calculated from the first day of the last menstrual period).
Infants born before term (before the beginning of the 38th week of pregnancy)
are classified as preterm infants regardless of their birth weight. Infants born
after the end of week 41 of pregnancy are classified as postterm infants or
postmature.
Infants who fall between the 10th and 90th percentiles of weight for their
gestational age, whether they are preterm, term, or postterm, are considered
appropriate for gestational age (AGA). Infants who fall below the 10th
percentile of weight for their age are considered small for gestational age
(SGA). Those who fall above the 90th percentile in weight are considered
large for gestational age (LGA). Other terms used include:
• Low–birth-weight (LBW) infant: one weighing less than 2,500 g at birth
• Very-low-birth-weight (VLBW) infant: one weighing less than 1500 g at
birth
• Extremely-low-birth-weight (ELBW) infant: one weighing less than 1,000
g at birth
such as hypoglycemia
Weight gain in Rapid Slow
nursery
Possibly always be Not likely to be
Future restricted <10th restricted in growth
growth percentile because of because “catch-up”
poor growth occurs
organ development
Etiology
At least 50% of neonatal deaths are preterm. Infant mortality could be
reduced dramatically if the causes of preterm birth could be discovered and
corrected and all pregnancies could be brought to term. However, even with
the examples of possible causes listed in the following, the exact cause of
premature labor and early birth is rarely exactly known.
Assessment
When interviewing parents of a preterm infant, be careful not to convey
disapproval of reported pregnancy behaviors such as cigarette smoking that
may have contributed to preterm birth. An accurate but comforting answer to a
direct inquiry about why preterm birth occurs is, “No one really knows what
causes prematurity.”
The eyes of most preterm infants appear small in relation to term infants.
Although difficult to elicit, a pupillary reaction is present. A preterm infant has
varying degrees of myopia (nearsightedness) because of a lack of eye globe
depth.
The ears appear large in relation to the head. The cartilage of the ear is
immature and allows the pinna to fall forward. The level of the ears should be
carefully inspected to rule out chromosomal abnormalities
Neurologic function in the preterm infant is often difficult to evaluate
because the neurologic system is still immature. Observing the infant make
spontaneous or provoked muscle movements can be as important as formal
reflex testing. During an examination, a preterm infant is much less active
than a mature infant and rarely cries. If the infant does cry, the cry is weak
and high pitched.
Potential Complications
Because of immaturity, preterm infants are prone to several specific
conditions
Anemia of Prematurity
Many preterm infants develop a normochromic, normocytic anemia (normal
cells, just few in number), which can make infants appear pale, lethargic, and
anorectic. Anemia occurs from a combination of immaturity of the
hematopoietic system (the effective production of red cells with an elevated
reticulocyte count may not begin until 32 weeks of pregnancy) combined with
the destruction of red blood cells because of low levels of vitamin E, a
substance that normally protects red blood cells against oxidation. Excessive
blood drawing for electrolytes, complete blood counts, or blood gas analysis
after birth can potentiate the problem. Delaying cord clamping at birth to allow
a little more blood from the placenta to enter the infant may also help reduce
the development of anemia.
Periventricular/Intraventricular Hemorrhage
Preterm infants are prone to periventricular hemorrhage (bleeding into the
tissue surrounding the ventricles) or intraventricular hemorrhage (bleeding
into the ventricles) because of fragile capillaries and immature cerebral
vascular development. When there is a rapid change in cerebral blood
pressure, such as could occur with hypoxia, intravenous infusion, ventilation,
or pneumothorax (lung collapse), capillary rupture could occur; brain anoxia
then occurs distal to the rupture.
Nutrition problems can arise with a preterm infant because the infant’s body is
attempting to continue to maintain the rapid rate of intrauterine growth
appropriate for the gestational age.
If these nutrients are not supplied, an infant can develop hypocalcemia
(decreased serum calcium) or azotemia (low protein level in the blood).
Delayed feeding and a resultant decrease in intestinal motility may also add to
hyperbilirubinemia, a problem infants already are at high risk of developing
when fetal red blood cells begin to be destroyed.
Digestion and absorption of nutrients in a preterm infant’s stomach and
intestine may be immature, making the digestion of milk difficult. Nutrition
problems are further compounded by a preterm infant’s immature reflexes,
which make swallowing and sucking difficult. An immature cardiac sphincter
(between the stomach and esophagus) allows regurgitation to occur readily.
The lack of a cough reflex may lead an infant to aspirate regurgitated formula.
Breast Milk. There is increasing evidence that although preterm infants grow
well on commercial formulas, the best milk for them, the same as with term
infants, is breast milk. The immunologic properties of breast milk may play a
major role in preventing neonatal necrotizing enterocolitis as well as an
increase in immune defenses.
Mothers can express breast milk manually or with a breast pump for
their infant’s gavage feedings. It is better for infants to receive their own
mother’s breast milk rather than banked milk if possible. This high level of
sodium seems to be necessary for fluid retention in the preterm infant.
The skin of a preterm infant is easily traumatized and therefore offers less
resistance to infection than the skin and mucous membrane of a mature
infant. In addition, preterm infants have a lowered resistance to infection
because they have difficulty producing phagocytes to localize infection as well
as a deficiency of immune globulin M (IgM) antibodies because of insufficient
production.
Nursing Diagnosis: Risk for impaired parenting related to interference with
parent– infant attachment resulting from hospitalization of infant at birth
Outcome Evaluation: Parents visit frequently and hold the infant; parents
speak of their child in positive terms.
- Learn the name of your child’s primary healthcare provider and primary
nurse or care manager. Make a point of talking to them when you visit so the
information you receive is consistent and so these important people can get to
know you.
- Discuss with your child’s primary nurse the time you will usually visit so she
or he can schedule your baby’s procedures and rest times other than when
you visit so there is time for you to hold your child and interact with him
uninterrupted
- Ask for explanations of any equipment or medications being used with your
child so you understand the plan of care.
- Any day you are unable to visit, call the nursery and ask to talk to your
child’s primary care nurse.
- Ask if you can supply expressed breast milk for your infant as soon as
feedings are started so you can feel you’re having a greater part in your
baby’s care.
- You might supply a tape recording of your voice so your baby can learn to
recognize it, as well as supply a small toy for your baby’s bed.
- Use your baby’s name when you talk about him (not “the baby”) to help you
gain a firm feeling that this is your baby, not the nursery’s.
- If your child is hospitalized a distance from home, ask if transfer to a local
hospital in a less technical environment will be possible as soon as he’s not
so ill.
Before effective bonding can be established, parents may need time to come
to terms with their feelings of disappointment that the infant is so small or guilt
that they were not able to prevent the preterm birth. Helping them air these
feelings and develop a more positive attitude toward their preterm infant is an
important nursing responsibility.
Because parents may not be psychologically ready for birth when a
preterm infant is born, it may be more difficult for them to believe they have a
child and to begin interacting than if the infant had been born at term.
Encourage the mother to come to the nursery and hold the infant before and
after gavage feedings and to breastfeed or bottle feed as soon as the baby is
ready for this.
If the baby is going to be transferred to a regional center, make sure
the parents have an opportunity to see the infant before the transfer. A
photograph of the infant for them to keep is helpful in making the birth more
real.
On days they cannot visit, parents can still stay in touch by telephone,
video, or nursery e-mail. By these means, by the time the baby is ready for
discharge, the parents should be able to feel they are taking home “their”
baby, one whom they know and have already begun to love.
Parents visiting a high-risk nursery often need a great deal of support
from nursing personnel. In such a high-tech setting, a parent may want very
much to touch his or her infant but is so afraid touching might set off an alarm
that he or she stands with arms folded
Because preterm infants can be hospitalized for long periods, parents can feel
baffled by receiving information from a parade of different healthcare
providers or a different person every time they visit. Consistent caregiver
helps to reduce the number of people who contact the parents and who
communicate the parents’ needs to the rest of the staff.
Try to make a baby’s siblings as welcome in a high-risk nursery as the
baby’s parents in order to build family unity. Check to be certain siblings do
not have an upper respiratory infection or fever. Also, their immunizations
should be up to date and they should not have been recently exposed to a
communicable disease, such as chickenpox, before they visit
Nursing Diagnosis: Deficient diversional activity (lack of stimulation) related
to preterm infant’s rest needs
The amount of rest and stimulation required by preterm infants for healthy
development is best individualized. Developmental care (care designed to
meet the specific needs of each infant) can lead to increased weight gain and
decreased crying and apnea spells in preterm infants.
Developmental Care
Discharge from a NICU is a major transition for parents as well as their infant.
Before discharge, the parents of a preterm infant need to learn and practice
any specialmethods of care necessary for their infant and interventions to help
maximize their child's development. Some parents tend to overprotect preterm
infants, such as notallowing visitors or not taking an infant outside. Let parents
know their concern is normal but overprotection is not necessary.
Ongoing health maintenance of a preterm infant follows the usual pattern of
well-child care. Basic immunizations are given according to the chronologic
age of aninfant. In many communities, NICUs maintain their own well-child
settings for infants who were hospitalized there.However, preterm infants can
be followed by any healthcare provider for well-child care.When plotting the
height and weight of preterm infants at well-child visits, remember to account
for early birth on the growth chart by double charting-that is, plotting the child's
weight and height accoding to the chronologic age (a pattern that, in the early
months, probably places the child below the 10th percentile). Then, in another
color, plot the height and weight according to an infant's adjusted age, or plot
the weight of a baby born 2 months early 2 months earlier on the graph. A
preterm baby typically gains "catch-up" weight in the first 6 months of life, so
by 1 year of age, a baby plots over the 10th pereentile on a growth chart
without accounting for a setback age.
An infant is SGA (also called microsomia) if the birth weight is below the 10th
percentile on an intrauterine growth curve for that age. Such infants may be
born:
Preterm: before week 38 of gestation
Term: between weeks 38 and 42
Postterm: past 42 weeks
Etiology
A woman's nutrition during pregnancy plays a major role in fetal growth, so a
lack of adequate nutrition may be a major contributor to IUGR (Ota, Tobe-Gai,
Mori, et Al. 2012). Adolescents are prone to having a high incidence of SGA
infants because if they eat only enough to meet their own nutritional and
growth needs, the needs of a growing fetus can be compromised. In still other
instances, the placental supply of nutrients is adequate but an infant cannot
use them because of a chromosomal abnormality or anintrauterine infection
such as rubella or toxoplasmosis.Even in light of these nutritional influences,
the most common cause of IUGR is a placental issue: either the placenta did
not obtain sufficient nutrients from the uterine arteries or uit was inefficient at
transporting nutrients to the fetus.
Assessment
The SGA infant may be detected in utero when fundal height during
pregnancy
becomes progressively less than expected. However, if a woman is unsure of
the date of her last menstrual period, this discrepancy can be hard to
substantiate; a sonogram can then demonstrate the decreased size. A
biophysical profile including a nonstress test, placental grading, amniotic fluid
amount, and an ultrasound examination documents additional information on
placental function and fetal growth.
Appearance
Generally, an infant who suffers nutritional deprivation early in pregnancy,
when fetal growth consists primarily of an increase in the number of body
cells, is below average in weight, length, and head circumference. An infant
who suffers deprivation late in pregnancy, when growth consists primarily of
an increase in cell size, may have only a reduction in weight. Regardless of
when deprivation occurs, the infant tends to have an overall wasted
appearance :
The infant may have poor skin turgor and generally appears to have a
large head because the rest of the body is so small.
Skull sutures may be widely separated. Hair may be dull and lusterless
The infant may have a small liver, which can cause difficulty regulating
glucose, protein, and bilirubin levels after birth.
The abdomen may be sunken. The umbilical cord often appears dry
and may be stained yellow.
Laboratory Findings
Blood studies at birth usually show a high hematocrit level (less than
normal amounts of plasma in proportion to red blood cells are present
because of a lack of fluid) and an increase in the total number of red blood
cells (polycythemia). The increase in red blood cells occurs because anoxia
during intrauterine life stimulated excess development of them. An immediate
cffect of polycythemia is to cause increased blood viscosity, a condition that
puts extra work on the infant's heart because it is more difficult to effectively
circulate thick blood. As a consequence, acrocyanosis (blueness of the hands
and feet) may be prolonged and persistently more marked than usual. If the
polycythemia is extreme, vessels may actually become blocked and thrombus
formationcan result. If the hematocrit level is more than 65% to 70%, an
exchange transfusion to dilute the blood may be necessary.
Birth asphyxia is a common problem for SGA infants, both because they have
underdeveloped chest muscles and because they are at risk for developing
meconium aspiration syndrome (MAS) as a result of meconium release, which
occurs when fetal anoxia develops during labor to cause reflex relaxation of
the anal sphincter. When gasping for breath in utero, the fetus draws
meconium discharged from the intestine into the amniotic fluid down into the
trachea and bronchi. Acting as a foreign substance, this blocks airflow into the
alveoli and causes the SGA infant to need resuscitation at birth. Closely
observe both respiratory rate and characterinthe first few hours of life as
underdeveloped chest muscles not only make taking the first breath difficult
but can make SGA infants unable to sustain an adequate newborn respiratory
rate.
Etiology
Infants who are LGA have been subjected to an overproduction of
nutrients and ggrowth hormone in utero. This happens most often to infants of
women who are obese or wwh have diabetes mellitus (Sjaarda et al., 2014).
Assessment
A fetus is suspected of being LGA when a woman's uterus appears to be
unusually
large for the date of pregnancy. Abdominal size can be deceptive, however.
Because a fetus lies in a flexed fetal position, he or she does not occupy
significantly more space at10 b than at 7 lb. If a fetus does seem to be
growing at an abnormally rapid rate, a sonogram can confirm the suspicion. A
nonstress test to assess the placenta's ability tt sustain a large fetus during
labor may be prescribed. Lung maturity may be assessed bB aamniocentesis If
an infant's large size was not detected during pregnancy, it may be ffirs
recognized during labor when the baby appears too large to descend through
the pelvic rim. If this happens, a cesarean birth may be necessary because
shoulder dystocia (tth wide fetal shoulders cannot pass; or needs significant
manipulation to pass through tth outlet of the pelvis) would halt vaginal birth at
that point.
Appearance
At birth, LGA infants may show immature reflexes and low scores on
gestational age examinations in relation to their size. They may have
extensive bruising or a birth injury such as a broken clavicle or Erb-Duchenne
paralysis from trauma to the cervical nerves if they were stressed in order for
the wide shoulders to be born vaginally. Because the head is large, it may
have been exposed to more thán the usual amount of pressure during birth,
causing a prominent caput succedaneum, cephalohematoma, or
molding.often immature, they require cautious.
Hypoglycemia
LGA infants also need to be carefully assessed for hypoglycemia in the early
hours of life because large infants require large amounts of nutritional stores
to sustain their weight. If the mother had diabetes that was poorly controlled
(the cause of the large size), the infant would have had an increased blood
glucose level in utero to match the mother's glucose level; this caused the
infant to produce elevated levels of insulin. After birth, these increased insulin
levels will continue for up to 24 hours of life, possibly causing rebound
hypoglycemia.