Health Check Up Reports

NAME: Mr.
RINKU Registration NO: 408494

Age/Gender: 34 Y/Male Registered: 08/May/2024 07:44AM
Lab NO: 032405080010 Analysed: 08/May/2024 01:08PM
BarcodeNo: 10411387 Reported: 08/May/2024 01:31PM
Referred BY: Dr. M.K MEHTA Panel: CANWINN AROGYA DHAM
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Test Name Value Unit Bio Ref.Interval
HEALTH PACKAGE ADVANCE

ESR 24 mm/1st 0 - 10
Method : Westergren
Hb A1C, GLYCOSYLATED Hb, K2 EDTA

Hb A1C, GLYCOSYLATED Hb ,EDTA 5.6 %
Method: HPLC - Cation Exchange
eAG(Estimated Average Glucose) 114.0 mg/dl 70 - 140
Last 3 month average sugar
SAMPLE TYPE : WHOLE BLOOD

Hemoglobin A1c(GIycated hemoglobin) is structrally related to adult hemoglobin (HbA) and has a glucose molecule attached to it.
HbAlc is continuously formed during the 120 day life of red blood ceII,and a single measurment of HbAlc reflects the average blood
glucose level d»ring the preceding 2-3 months.
HbAlc of 7.0% means that 7.0o/ of the total hemoglobin has glucose attached to it.
Criteria :- For patients are diagnosed with Normal values or Diabetes.
Less than 5.7 % – Normal

5.7 % to 6.4 % – Pre – Diabetes
6.5 % and above – Diagnosed Diabetes
6.5 % to 7.0 % – Very Good Control
7.0 % to 8.0 % – Fair To Good control
8.0 % to 10.0 % – Unsatisfactory Control
Above 10.0 % – Very Poor Control
For patients diagnosed with Dlabetes :-

* American Diabetes Association(ADA) recommends that for adequate glucose control a reasonable HbAlc goal for a non pregnant
adult is less than 7.0%.
Note :- ADA recommends that individuals with diabetes be tested a least twice each year for those in good controlled or whose
therapy has changed.
Page 1 of 15
NAME: Mr. RINKU Registration NO: 408494
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CBC, COMPLETE BLOOD COUNT

HAEMOGLOBIN 13.9 gm/dl 13.0 - 17.0
Method: Colorimetric Assay
TOTAL LEUCOCYTE COUNT (TLC) 11.00 th/cumm 4.0 - 10.0
Method: Electrical Impedance
DIFFERENTIAL LEUCOCYTE COUNT
NEUTROPHIL 59.8 % 34 - 64
Method: Flow Cytometry
LYMPHOCYTE 31.1 % 20-40
EOSINOPHIL 5.6 % 1-6
MONOCYTE 3.1 % 02 - 10
BASOPHIL 0.4 % 0-1
RBC COUNT 4.90 millions/cmm 4.5 - 5.5
Method: Electrical Impedance
PCV/ HAEMATOCRIT 43.10 % 40.0 - 50.0
Method : Electrical Impedance
MCV 87.4 fl. 83.0 - 101.0
Method : Calculated
MCH 28.20 picogram 27.0 - 32.0
Method : Calculated
MCHC 32.3 gm/dl 31.5 - 34.5
Method : Electrical Impedance
PLATELET COUNT 187 1000/cumm 150 - 410
Method: Elec.Impedance/Manual
ABSOLUTE NEUTROPHIL COUNT 6.58 th/cumm 2.0 - 7.0
ABSOLUTE LYMPHOCYTE COUNT 3.42 th/cumm 1.0 -3.0
ABSOLUTE EOSINOPHIL COUNT 0.62 th/cumm 0.02 - 0.5
ABSOLUTE MONOCYTE COUNT 0.34 th/cumm 0.2 - 1.0
ABSOLUTE BASOPHIL COUNT 0.04 1000/uL 0.02-0.1
MPV 16.20 fl. 6.5 - 11.0
Method: Calculated
RDW (SD) 46.30 F/L 35 - 56
Method: Calculated
*
Page 2 of 15
RDW (CV) 14.30 % 11.6-14.0

Method: Calculated
PCT 0.30 % 0.17 - 0.4
Method: Calculated
P-LCR 67.50 % 17.5 - 42.3
Method: Calculated
Page 3 of 15
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Absolute Eosinophil Count ,EDTA 620 /ul 20 - 500

Page 4 of 15
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Complete Urine Analysis (CUE)

Specimen: Urine
COLOUR YELLOW
Method: Physical
APPEARANCE S. HAZY -
Method: Physical
pH 6.50 5.0-8.0
Method: Multistix
SPECIFIC GRAVITY 1.020 1.001-1.030
Method: Multistix
KETONES NIL NIL
Method : Multistix
GLOCOSE NIL NIL
Method : Multistix
BLOOD NIL NIL
Method: Multistix
BILIRUBIN NIL NIL
Method : Multistix
UROBILINOGEN NEGATIVE NEGATIVE
Method: Multistix
NITRITE NEGATIVE NEGATIVE
Method : Multistix
LEUKOCYTE NEGATIVE NEGATIVE
Method: Multistix
PROTEIN + NIL
Method: Multistix
MICROSCOPIC EXAMINATION
PUS CELLS 6-8 /HPF 1-2
Method : Microscopy
RBC NIL /HPF NIL
Method: Microscopy
EPITHELIAL CELLS 8-10 /HPF 2-3
Method : Microscopy
CASTS NIL NIL
Method : Microscopy
CRYSTALS NIL NIL
Method: Microscopy
BACTERIA NIL NIL
Method: Microscopy
YEAST CELLS NIL NIL
Method: Microscopy
*
Page 5 of 15
OTHERS NIL -
Method: Microscopy
Page 6 of 15
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Specimen: Serum
CRP;SERUM 16.80 mg/l 0.0 - 6.0
Immunoturbidimetry
Comments
CRP is an acute phase reactant which is used in inflammatory disorders for monitoring course and effect of therapy. It is most useful as an indicator of activity in
Rheumatoid arthritis, Rheumatic fever, tissue injury or necrosis and infections. As compared to ESR, CRP shows an earlier rise in inflammatory disorders which
begins in 4-6 hrs, the intensity of the rise being higher than ESR and the recovery being earlier than ESR. Unlike ESR, CRP levels are not influenced by hematologic
conditions like Anemia, Polycythemia etc.,
IRON PROFILE;SERUM
IRON ,Serum 118.9 ug/dL 50.0-175.0
Method: Spectrophotometry
UNSATURATED IRON BINDING CAPACITY 289.50 ug/dL 110.0 - 370.0
Method: Spectrophotometry
TOTAL IRON BINDING CAPACITY 408.4 ug/dL 228.0 - 448.0
Method: Calculated
TRANSFERRIN SATURATION 29.11 % 16.0 - 50.0
Method: Calculated
Serum Iron
Serum iron measures the amount of circulating iron that is bound to transferrin. Clinicians order this laboratory test when they are concerned about iron
deficiency, which can cause anemia and other problem.
Total Iron Binding Capacity

The test measures the extent to which iron-binding sites in the serum can be saturated. Because the iron-binding sites in the serum are almost entirely dependent
on circulating transferrin, this is really an indirect measurement of the amount of transferrin in the blood.
Taken together with serum iron and percent transferrin saturation clinicians usually perform this test when they are concerned about anemia, iron deficiency or
iron deficiency anemia. However, because the liver produces transferrin, liver function must be considered when performing this test. It can also be an indirect
test of liver function, but is rarely used for this purpose.
Note : Kindly correlate clinically.
Page 7 of 15
Specimen: Na F PLASMA
GLUCOSE FASTING 90.80 mg/dl 74.0 - 100.0
Method : GOD-POD
American Diabetes Association Reference Range :-

Normal : < 100 mg/dl
Impaired fasting glucose(Prediabetes) : 100 - 126 mg/dl
Diabetes : >= 126 mg/dl
Glucose PP : 70 – 140 mg/dl
Interpretation:-
Conditions that can result in an elevated blood glucose level include: Acromegaly, Acute stress (response to trauma, heart attack, and stroke
for instance), Chronic kidney disease, Cushing syndrome, Excessive consumption of food, Hyperthyroidism, Pancreatitis
A low level of glucose may indicate hypoglycemia, a condition characterized by a drop in blood glucose to a level where first it causes
nervous system symptoms (sweating, palpitations, hunger, trembling, and anxiety), then begins to affect the brain (causing confusion,
hallucinations, blurred vision, and sometimes even coma and death). A low blood glucose level (hypoglycemia) may be seen with:Adrenal
insufficiency, Drinking excessive alcohol, Severe liver disease, Hypopituitarism, Hypothyroidism, Severe infections, Severe heart failure,
Chronic kidney (renal) failure, Insulin overdose, Tumors that produce insulin (insulinomas), Starvation.
Page 8 of 15
LIVER FUNCTION TEST(LFT)

Specimen: Serum
BILIRUBIN TOTAL 0.90 mg/dl 0.0 - 2.0
Method : DCA Method
CONJUGATED (D. Bilirubin) 0.30 mg/dL 0.0 - 0.20
Method : DCA Method
UNCONJUGATED (I.D.Bilirubin) 0.60 mg/dL 0.0 - 1.00
Method : Calculated
SGOT (AST) 38.60 IU/L 0.0-40.0
UV With P5P
SGPT (ALT) 42.10 U/L 0.0 - 45.0
Method : IFCC
ALKALINE PHOSPHATASE 120.00 U/L 53.0 - 128.0
Method : AMP Method
GAMMA GT 20.10 U/L 0.0 -55.0
Method : GLUPA - C
TOTAL PROTEIN 7.70 g/dL 6.4 - 8.3
Method : Colorimetric
ALBUMIN 4.30 g/dL 3.5 - 5.2
Method : BCG Method
GLOBULIN 3.40 g/dL 2.0 - 4.0
Method : Calculated
A/G Ratio 1.26 0.8 - 2.1
Method : Calculated
SGOT / SGPT Ratio 0.92 > 2.0 : Alcoholic liver Disease
Method: Calculated 1.0 - 1.5 : Viral Infection
Note
1. In an asymptomatic patient, Non alcoholic fatty liver disease (NAFLD) is the most common cause of increased AST, ALT levels. NAFLD is considered as hepatic
manifestation of metabolic syndrome.
2. In most type of liver disease, ALT activity is higher than that of AST; exception may be seen in Alcoholic Hepatitis, Hepatic Cirrhosis, and Liver neoplasia. In a
patient with Chronic liver disease, AST:ALT ratio>1 is highly suggestive of advanced liver fibrosis.
3. In known cases of Chronic Liver disease due to Viral Hepatitis B & C, Alcoholic liver disease or NAFLD, Enhanced liver fibrosis (ELF) test may be used to evaluate
liver fibrosis.
4. In a patient with Chronic Liver disease, AFP and Des-gamma carboxyprothrombin (DCP)/PIVKA II can be used to assess risk for development of Hepatocellular
Carcinoma.
Page 9 of 15
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KIDNEY FUNCTION TEST (KFT / RFT)

BLOOD UREA 30.30 mg/dl 19.0 - 45.0
Method : Urease - GLDH
SERUM CREATININE 0.80 mg/dl 0.7 - 1.3
Method : enzymatic
SERUM URIC ACID 4.80 mg/dL 3.5 - 7.2
Method : Uricase POD
CALCIUM 8.7 mg/dl 8.6 - 10.2
Method : Arsenazo III
SODIUM 138.5 mmol / L 136.0 - 145.0
Method: ISE
POTASSIUM 4.22 mmol / L 3.5 - 5.5
Method: ISE
TOTAL PROTEIN 7.70 g/dL 6.4 - 8.3
Method : Colorimetric
ALBUMIN 4.30 g/dL 3.5 - 5.2
Method : BCG Method
GLOBULIN 3.40 g/dL 2.0 - 4.0
Method : Calculated
INTERPRETATION:
Urea is the end product of protein metabolism.It reflects on funcioning of the kidney in the body. Creatinine is the end product of creatine metabolism.It is a measure
of renal function and eleveted levels are observed in patients typically with 50% or greater impairment of renal function.Sodium is critical in maintaining water &
osmotic equilibrium in extracellular fluids.Disturbances in acid base and water balance are typically reflected in the sodium concentrations .Potassium is an essential
element involved in critical cell functions. Potassium levels are influenced by electrolyte intake ,excretion and other means of elemination ,exercise ,hydration and
medications. Calcium imbalance my cause a spectrum of disease. High concentrations are seen in Hyperparathyroidism,Malignancy & Sarcoidosis. Low levels may be
due to protein eficiency,renal insufficiency and Hypoparathyroidism.Repeat measurement is recommended if the values are outside the reference range.
Page 10 of 15
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LIPID PROFILE
TOTAL CHOLESTEROL 195.0 mg/dL 0.0 - 200.0
Method : CHOD-PAP
TRIGLYCERIDES 135.7 mg/dL 0.0 - 161.0
Method : GPO
H D L CHOLESTEROL DIRECT 40.2 mg/dL 35.3 - 79.5
Method : Direct Method
NON-HDL CHOLESTEROL 154.80 0 - 130
Method: Calculated
L D L CHOLESTEROL DIRECT 127.66 mg/dL 70.0 - 165.0
Method: Calculated
VLDL 27.1 mg/dL 15.0 - 40.0
Method: Calculated
TOTAL CHOLESTEROL /HDL RATIO 4.85 Ratio 0.0 - 4.97
Method : Calculated
LDL / HDL CHOLESTEROL RATIO 3.18 Ratio 0.00 - 3.55
Method : Calculated
HDL / LDL CHOLESTEROL RATIO 0.31 Ratio 0.30 - 0.40
Method : Calculated
INTERPRETATION:
TRIGLYCERIDE level > 250mg/dL is associated with an approximately 2-fold greater risk of coronary vascular disease. Elevation of triglycerides can be seen with
obesity, medication, fast less than 12 hrs., alcohol intake, diabetes melitus,and pancreatitis. CHOLESTEROL, its fractions and triglycerides are the important plasma
lipids indefining cardiovascular risk factors and in the managment of cardiovascular disease.Highest acceptable and optimum values of cholesterol values of cholesterol
vary with age. Values above 220 mgm/dl are associated with increased risk of CHD regardless of HDL & LDL values. HDL-CHOLESTEROL level <35 mg/dL is
associated with an increased risk of coronary vascular disease even in the face of desirable levels of cholesterol and LDL - cholesterol. LDL - CHOLESTEROL&
TOTAL CHOLESTEROL levels can be strikingly altered by thyroid, renal and liver disease as well as hereditary factors. Based on total cholesterol, LDL-
cholesterol, and total cholesterol/HDL - cholesterol ratio, patients may be divided into the three risk categories.
Page 11 of 15
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VITAMIN B12, Serum

VITAMIN B12 LEVEL ,Serum 248.00 pg/mL 211.0 - 946.0
ECLIA
Comment
Vitamin B12 belongs to the corrin family it is a cofactor for the conversion of methylemalonyl coenzyme A to succinyl
coenzyme A, synthesis of methionine from homocysteine and formation of myelin. It is required along with folate for DNA
synthesis. The major source of vitamin B12 for human beings is meat, while herbivorous animals get their requirement
from contaminated vegetable matter and coprophagy. Megaloblastic anaemia can be due to cobalamin & or folic acid
deficiency.
Vitamin B12 or Cyanocobalamin, is a complex corrinoid compound containing four pyrrole rings
that surround a single cobalt atom. Humans obtain vitamin B12 exclusively from animal dietary sources,
such as meat, eggs and milk.
Clinical and laboratory findings for Vitamin B12 deficiency include neurological abnormalities,
decreased serum B12 levels and increased excretion of methylmalonic acid. The impaired DNA synthesis
associated with Vitamin B12 deficiency causes macrocytic anaemias. These anaemias are characterized by abnormal
maturation of erythrocyte precursors in the Bone-Marrow, which results in the presence of magaloblasts and in decreased
erythrocyte survival.
Pernicious anaemia is a macrocytic anaemia caused by Vitamin B12 deficiency that is due to lack of intrinsic factor. Low
Vitamin B12 intake, gastrectomy, diseases of small intestine, malabsorption and trans-cobalamin deficiency can also
cause Vitamin B12 deficiency.
Increased Levels :-
1. Renal failure
2. Liver disease
Decreased Levels :-
1. Megaloblastic anemia
2. Vegetarianism
Comment :
Vitamin B12 deficiency causes macrocytic /Megaloblastic anaemia and neurological
abnormalities .Pernicious anaemia is also caused by vitamin B12 deficiency that is due to
lack of interinsic factor.Vitamin B12 deficiency is caused by low intake of vitamin B12 ,
Gastrectomy , Disease of small intestine , malabsorption.
Page 12 of 15
VITAMIN D (25 OH),SERUM

VITAMIN D(25 OH) ,Serum 23.54 ng/ml DEFICIENCY:- <20.0 ng/ml
Method: eCLIA INSUFFICIENCY:- 20-30 ng/ml
SUFFICIENCY:- 30-100 ng/ml
TOXICITY: >100 ng/mL
COMMENTS:-
1. Cholicalciferol (vitamin D3) is synthesized in the skin from 7 dehydrocholestrol in
response to sunlight. Ergocalciferol (vitamin D2) comes essentially from diet and supplements.
The assay measures both D2 (Ergocalciferol) and D3 (Cholecalciferol) metabolites of vitamin D.
Vitamin D status is best determined by measurement of 25 hydroxy Vitamin D, as it is the major
circulating form and has longer half life (2-3 weeks) than 1,25 Dihydroxy vitamin D (5-8 hrs).
2. Both Cholicalcifecal and Ergocalciferol are converted in liver to 25 OH Vitamin D.
3. 25 OH Vitamin D is considered the best indicator of Vitamin D nutritional status.
4. Vitamin D toxicity is recognized, but is a rare occurance.
5. Vitamin D level shows seasonal variation, with values being 40-50% lower in winter than in summer.
Decreased levels:
* Inadequate exposure to sunlight.
* Dietary deficiency.
* Vitamin D malabsorption.
* Severe Hepatocellular disease.
Kindly correlate all result clinically.
Page 13 of 15
THYROID PROFILE
T3 ,Serum 170.20 ng / dl 60.0 - 181.0
T4 ,Serum 6.25 ug / dl 4.5 - 10.9
TSH, ULTRASENSITIVE, Serum 2.60 uIU/ml 0.35 - 5.50
THE ABOVE REFERENCE RANGE ARE FOR ADULTS ONLY
AGE RELATED GUIDLINES FOR REFENCES RANGE FOR T3, T4 & TSH
T3 CORD BLOOD NEW BORN 1-5 YEARS 5-10 YEARS 10-15 YEARS
(ng/dl) 30 - 70 75 -260 100 - 260 90 -240 80 -210
T4 1-3 DAYS 3 DAYS -1 MONTHS 1-12 MONTH 1-3 YEARS 3-10 YEARS
(ng/dl) 8.2 - 19.9 6.0-15.9 6.1-14.9 6.8-13.5 5.5 - 12.8
TSH NEW BORN INFANT CHILD

uIU/ml 1.0 - 38.9 1.7 - 9.1 0.7- 6.4
Note : TSH level are subject to circadian variation ,reaching peak levels between 2-4 AM and at a minimum between 6 - 10 PM .
The variation is of the order of 50 % ,
hence time of the day has influence on the measures serum TSH concentration . Dose and time of drug intake also influence the test
result.
Ultrasensitive Kit are used
Serum FT3 FT4 and TSH measurements from three components of thyroid screening panel and are useful in diagnosing various disorder of
thyroid gland function.
1. Primary hyperthyroidism is accompanied by elevated serum T3 & T4 value along with depressed TSH level.
2. Primary hypothyroidism is accompanied by deressed serum T3 & T4 value and elevated serum TSH level.
3. Normal T4 level accompanied by high T3 level and low TSH are seen in patient with T3 thyrotoxicosis.
4. Normal or low T3 & high T4 level indicate T4 thyrotoxicosis ( problem in conversion of T4 to T3 ).
5. Normal T3 & T4 along with low TSH indicate mild / subclinical HYPERTHYRODISM .
6. Normal T3 & low T4 along with high TSH indicate mild / subclinical HYPOTHYRODISM
7. Normal T3 & T4 LEVEL with high TSH indicate mild / subclinical HYPOTHYRODISM.
8. Slightly elevated T3 Levels may be found in pregnancy and in estrogen therapy while depressed levels may be
encountered in severe illness , malnutrition , renal failure and during therapy with drugs like propanolol.
9. Although elevated TSH levels are nearly always indicative of primary hypothroidism . rarely they can result from
TSH secreting pituitary tumours ( seconday hyperthyroidism )
Result is to be correllated clinically.
Page 14 of 15
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TYPHIDOT (IgG & IgM);SERUM

TYPHIDOT (IgG) NEGATIVE . Negative
RAPID
TYPHIDOT (IGM) NEGATIVE . Negative
RAPID
COMMENT:-
IgM positive means acute typhoid fever.
IgM & IgG positive means acute typhoid fever in middle stage of
infection.
IgG positive means relapse or reinfection or previous infection.
IgM & IgG negative means probably not typhoid. sensitivity,
specificity, negative predictive values & positive predictive
value is 95.
Rarely high IgG concentration may give false negative for IgM
because of speific IgM to the antigen.
To be correlated clinically.
*** End Of Report ***
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NAME: Mr.

RINKU Registration NO: 408494

Test Name Value Unit Bio Ref.Interval

HEALTH PACKAGE ADVANCE

Hb A1C, GLYCOSYLATED Hb, K2 EDTA

SAMPLE TYPE : WHOLE BLOOD

Criteria :- For patients are diagnosed with Normal values or Diabetes.

Less than 5.7 % – Normal

For patients diagnosed with Dlabetes :-

Test Name Value Unit Bio Ref.Interval

CBC, COMPLETE BLOOD COUNT

Test Name Value Unit Bio Ref.Interval

RDW (CV) 14.30 % 11.6-14.0

Test Name Value Unit Bio Ref.Interval

Absolute Eosinophil Count ,EDTA 620 /ul 20 - 500

Test Name Value Unit Bio Ref.Interval

Complete Urine Analysis (CUE)

Test Name Value Unit Bio Ref.Interval

Test Name Value Unit Bio Ref.Interval

Total Iron Binding Capacity

Note : Kindly correlate clinically.

Test Name Value Unit Bio Ref.Interval

American Diabetes Association Reference Range :-

Test Name Value Unit Bio Ref.Interval

LIVER FUNCTION TEST(LFT)

Test Name Value Unit Bio Ref.Interval

KIDNEY FUNCTION TEST (KFT / RFT)

Test Name Value Unit Bio Ref.Interval

Test Name Value Unit Bio Ref.Interval

VITAMIN B12, Serum

Vitamin B12 deficiency causes macrocytic /Megaloblastic anaemia and neurological

Gastrectomy , Disease of small intestine , malabsorption.

Test Name Value Unit Bio Ref.Interval

VITAMIN D (25 OH),SERUM

Kindly correlate all result clinically.

Test Name Value Unit Bio Ref.Interval

TSH NEW BORN INFANT CHILD

BarcodeNo: 10411387 Reported: 08/May/2024 03:01PM

Test Name Value Unit Bio Ref.Interval

TYPHIDOT (IgG & IgM);SERUM

*** End Of Report ***

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