Unstable Angina Pectoris
Unstable Angina Pectoris
Unstable Angina Pectoris
Name: Mahfuzah Hazirah STB: C 111 07 307 Supervisor: dr. Muzakkir Amir, SpJP.FIHA.FICA
PATIENTS IDENTITY
Name:
Ny. Widyaningsih
Sex/age: Female/63 yo Ward : CVCU Medical Record: 385330 Date of admission: 07th July 2012 Fee : Askes
HISTORY TAKING
Chief
complaint:
Chest pain
Guided anamnesis:
Occurred 3 days prior admission. Getting worse 1 day ago. The patient complain of pain on the left side of chest. Nonradiated. Dull pain with burning sensation. Duration less then 20 minutes. Frequency of recurrent attack: 6-7 times per day with the increasing intensity. The pain does not triggered by activity. Shortness of breath (+) even at resting state. History of sudden wake at night (-). Patient able to sleep with one pillow. No cough. No nausea and vomit. No epigastric pain.
RISK FACTORS
History of coronary artery disease 3 years ago History of hypertension 2 years ago History of 1st degree family with coronary disease and hypertension : mother History of dyslipidemia
PHYSICAL EXAMINATION
GENERAL Moderate
VITAL SIGN Blood pressure : 130/90 mmHg Pulse : 60 bpm Breathing : 26 x/I Temperature: 36.5
LOCAL EXAMINATION
EYE:
anemic (-) jaundice (-) isochors pupil 2.5mm/2.5mm. Palpebra edema (-/-) NECK: JVP +2cm H2O. Lymph nodes enlargement (-) Thorax:
Inspection: symmetry both right and left hemithorax, Palpation: vocal fremitus P: symmetry, tumor (-) tenderness (-) Percussion : sonor for both hemithorax. Auscultation: vesicular breathing, ronchi (-/-) wheezing (-/-)
Cor: Heart sound S1/S2 reguler Abdomen: peristaltic (+) normal, hepar-lien are not palpated Extremities : edema (-/-)
ELECTROCARDIOGRAM (09/07/2012)
Interpretation :
Rhythm:
sinus rhythm
QRS rate: HR 65 bpm P wave : 0.04 sec, poor P-wave at aVR PR interval: 0.16 sec QRS complex: 0.04 sec Axis: Left Axis Deviation ST segment: isoelectric T-wave: normal Conclusion: sinus rhythm Hr= 65 bpm
RADIOLOGY FINDING
FOTO THORAX AP (07 JULY 2012)
Lung bronchovascular is within normal limit. No spesific process can be detected at both side of the lung. Heart enlarged with CTI 13/19.6=0.66, apex embedded. Right sinus , left sinus and both diaphragma is normal Intact bones Summary: cardiomegaly (HHD)
TREATMENT
O2 2-3 lpm k/p IVFD NaCl 0.9% 10 tpm Isosorbid dinitrate 1mg/h/SP Diuretic 40 mg 1-0-0 Amlodipin 5 mg 0-0-1 Clopidogrel 75 mg 0-1-0 Alprazolam 0.5 mg 0-0-1
DISCUSSION
UNSTABLE ANGINA PECTORIS
DEFINITION
Angina pectoris, or angina, is a symptom of chest pain or pressure that occurs when the heart is not receiving enough blood and oxygen to meet its needs. Unstable angina occurs in unexpected or unpredictable times, such as at rest. Unstable angina symptoms are a medical emergency, and may be a precursor for a heart attack. Thus, medical attention should be sought immediately.
http://www.cardiosmart.org/HeartDisease/
CLINICAL MANIFESTATION
Unstable angina pain can last between 5 and 20 minutes. Sometimes symptoms can come and go, Many people describe unstable angina as:
Pain
or pressure
Tightness A heavy, crushing feeling in the chest, neck, throat, jaw, shoulder and/or arm Discomfort just below the breastbone Burning similar to heartburn or indigestion Shortness of breath
Because unstable angina occurs without warning and during rest, it can cause severe anxiety. Unstable angina sometimes brings about other symptoms such as nausea, light headedness, or profuse sweating. The pain from angina may subside if a person takes nitroglycerin.
http://www.cardiosmart.org/HeartDisease
RISK FACTORS
Unstable angina results from coronary artery disease (CAD). Thus, risk factors for the development of CAD are also risk factors for unstable angina:
Smoking
Havinghigh cholesterollevels(hypercholesterolemia) Low HDL cholesterolemia (<40 mg/dl) Hypertrigleseridemia (>200 mg/dl) Hypertension Diabetes mellitus Obesity Having family members (especially parents or siblings) who have had coronary artery disease (CAD) or a stroke (<65 yo)
http://www.cardiosmart.org/HeartDisease
A classification has been proposed by Braunwald to facilitate the assignation of patients to a particular risk group. This classification takes into account the severity of symptoms, the clinical circumstances surrounding the anginal episode, and the intensity of treatment.
Classification
PATHOGENESIS
Plaque rupture Thrombus formation Incomplete/ intermittent occlusion of the infact-related vessel to the presence of collateral channels/ to small size of affected vessel.
Cardiology, Desmond G. Julian, J.Campbell Cowan, James M. McLenachan, 8th edition, Elsevier, 2005
DIAGNOSIS
Cardiology, Desmond G. Julian, J.Campbell Cowan, James M. McLenachan, 8th edition, Elsevier, 2005
DIAGNOSIS
Clinical
Increase
Pre-existing angina Last longer than 10 minutes to several hours Not related to activities Pain may be intermitten Not relieve by nitrate
Cardiology, Desmond G. Julian, J.Campbell Cowan, James M. McLenachan, 8th edition, Elsevier, 2005
ECG ST
changes
segment depression/ T-wave inversion Serial ECG tracing should be recorded Cardiac enzyme level CK and CK-MB levels may be mildly raised Troponin-T may have a slight increased.
Cardiology, Desmond G. Julian, J.Campbell Cowan, James M. McLenachan, 8th edition, Elsevier, 2005
PRINCIPLE MANAGEMENT
any plaques that may have ruptured in order to prevent a heart attack, relieving symptoms treating the underlying coronary artery disease (CAD).
http://www.cardiosmart.org/HeartDisease
MANAGEMENT
http://www.cardiosmart.org/HeartDisease
CORONARY INTERVENTION
PCI Coronary angioplasty (a balloon-tipped catheter is inserted into a blood vessel in the arm or groin and is advanced through blood vessels and into the heart) Coronary artery bypass grafting surgery(CABG)
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