Blounts Disease

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BLOUNTS

DISEASE
Moderators:Dr.K.G
Kamath
Dr.Ronald M
Presenter:Dr.Gururaj
Date:30/06/09

History
Erlacher

reported the first case of tibia vara

in 1922.

1937, Blount reported 13 more cases and

reviewed all of the 15 cases that were


reported in the literature up to that time.
Blount suggested the term tibia vara.

Normal Tibio -femoral


angles

Physiological genu
varum

In utero molding of
the lower
extremities

Definition
Disease of unknown etiology that affects the

metaphysis,,the growth plate and the


epiphysis of the posteromedial aspect of
proximal tibia resulting in a varus and internal
rotation deformity(Blount and Milwakee 1937)

Etiology
Exact cause unknown
Combination of hereditory and developmental

factors
Abnormal stress placed on the posteromedial

proximal tibial epiphysis that leads to growth


suppression

Obese pt (body weight)


Abnormal stress
Static varus knee
Compr ession of medial
lateral
physis

tensile strsses on
physis

progressive genu varum

pathology
1)densely packed cartilage cells displaying
hypertrophy
2)islands of acellular cartilage
3)large clusters of vessels
Disrupted enchondral ossification.

Age
The infantile ( 1-3 years)
juvenile (4-10 years)
adolescent(11-14 years)

Clinical features
Infantile tibia vara

-females
- blacks
-marked
obesity
-no pain

-palpable metaphyseal beak


-leg length descrepancy
-lateral thrust to the knee during the stance
phase of gate
-bilateral in approximately 80%

Adolesent tibia vara


complain of pain
typically overweight

or obese
unilateral in 80% of
cases

Radiological features
Langenskiold staging

Metapyseal diaphyseal angle

Levine and drennan angle


>11 degrees risk of

blounts disease is high


>16 degrees diagnostic

Infantile tibia vara


Widened irregular

physeal line
Increased meta-diap
angle >11 degrees
Sloped irregularly
occupied epiphysis
Metaphyseal beak

Adolescent tibia vara

Slope relatively

normal
Lack of meta beak
Widened medial
physeal plate

Differential diagnosis
Physiological genu varum

-gradual curve involving both tibia and


femur.
-non progressive, spontaneously resolves.
Hypophosphatemic rickets
-short stature and genu varum
-x rays rachitic changes in physis
-s.phospharus is low
Metaphyseal chondrodysplasia
Congenital bowing

Treatment
Langenskiold stage 1 and 2

<3 yrs: orthotics


>3 yrs: corrective osteotomy

Langenskiold stage3: corrective osteotomy,

delay in surgery after 4 yrs more chances of


recurrence.

Langenskiold stage4 and 5: physiologic

physeal arrest

In pt younger than 8 years or with 2 yrs


skeletal growth remaining Realignment and
medial physeal resection with placement of
interpositioning material .

Langenskiold stage6: established bony bridge


if pt has < 2 yrs growth remaining and normal

joint surface
corrective osteotomy with complete
physeal closure
If > 2 yrs of growth is remaining ,
resection of bony bridge (asymmetrical
physeal distraction)with replacement of
interposition graft

incongruity of joint surface - intraarticular

directed ostetomy

adolescent tibia vara:

- corrective osteotomy
- lateral epiphysiodesis
-realignment by external fixation.

Orthotics

KAFO

ELASIC BLOUNT BRACES.


Can be used 23 hrs a day,

or partial time wear (only


during nights ) both are
equally successful

osteotomy
Distal to the tibial tubercle.
internal tibial torsion corrected by external

rotation of tibial component .


fibular osteotomy should be performed

through separate incision.

Rabs osteotomy

Greene chevron
osteotomy

Epiphyseal and metaphyseal osteotomy


Intra epiphyseal osteotomy
Hemi elevation of epiphysis and osteotomy

with leg lengthening using ilizarao

Thank
you

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