Heart Atau Cardiac Failure
Heart Atau Cardiac Failure
Heart Atau Cardiac Failure
FAILURE
dr.Bobi Ahmad S, S.Kep
DEFINITION
3. VALVULAR DISEASE
CAUSE OF RIGHT VENT.
FAILURE
1. LEFT VENT. FAILURE
3. PULMONARY HYPERTENSION
LOW-OUTPUT VS HIGH-
OUTPUT SYNDROME
• INCREASED METABOLIC NEEDS
- HYPERTHYROIDISM
- FEVER
- PREGNANCY
• HYPERKINETIC CONDITIONS
- ARTERIOVENOUS FISTULAS
- PAGET’S DISEASE
FUNCTIONAL STATUS
• CLASS I
- CLIENTS WITH CARDIAC DISEASE, BUT
WITHOUT RESULTING LIMITATIONS OF
PHYSICAL ACTIVITY
- ORDINARY PHYSICAL ACTIVITY DOES
NOT CAUSE UNDUE FATIGUE
PALPITATION, DYSPNEA, OR ANGINAL
PAIN
FUNCTIONAL STATUS
• CLASS II
- CLIENTS WITH C.D RESULTING IN
SLIGHT LIMITATION OF PHYSICAL
ACTIVITY
- THEY ARE COMFORTABLE AT REST
- ORDINARY PHYSICAL ACTIVITY
RESULTS IN FATIGUE, PALPITATION,
DYSPNEA, OR ANGINAL PAIN
FUNCTIONAL STATUS
• CLASS III
- CLIENTS WITH CD RESULTING IN
MARKED LIMITATION OF PHYSICAL
ACTIVITY
- THEY ARE COMFORTABLE AT REST
- LESS THAN ORDINARY PHYSICAL ACT
CAUSES FATIGUE, PALPITATION,
DYSPNEA, OR ANGINAL PAIN
FUNCTIONAL STATUS
• CLASS IV
- CLIENTS WITH CD RESULTING IN
INABILITY TO CARRY ON ANY PHYSICAL
ACT WITHOUT DISCOMFORT
- SYMPTOMS OF CARDIAC INSUFFICIENCY
OR OF THE ANGINAL SYNDROME MAY BE
PRESENT EVEN AT REST
- IF ANY PHYSICAL ACT IS UNDERTAKEN,
DISCOMFORT IS INCREASED
ETIOLOGY
VOLUME OVERLOAD: PRESSURE OVERLOAD:
- AORTIC INCOMPETENCE - HYPERTENTION
- MITRAL INCOMPETENCE - AORTIC STENOSIS
- TRICUSPID INCOMPETENCE - HYPERTROPHIC CARDIOMYOPATHY
- OVERTRANSFUSION FILLING DISORDERS :
- LEFT-TO-RIGHT SHUNTS - MITRAL STENOSIS
- HYPERVOLEMIA - TRICUSPID STENOSIS
MYOCARDIAL DYSFUNCTION: - CARDIAC TEMPONADE
- CARDIOMYOPATHY - RESTRICTIVE PERICARDITIS
- MYOCARDITIS INCREASED METABOLIC DEMAND :
- CORONARY ARTERY DISEASE - ANEMIAS
- ISCHEMIA - FEVER
- INFARCTION - BERIBERI
- DYSRYTHMIAS - PAGET’S DISEASE
- TOXIC DISORDERS - ARTEROVENOUS FISTULA
INCIDENCE / PREVALENCE
• 75 % HEART FAILURE OLDER THAN 60
YEARS
• 60 / 1000 POPULATION IN THE AGE
GROUP 65 & OLDER
• INCREASES TO 100 / 1000 IN THE AGE 7
5 YEARS & OLDER
• MEN > WOMEN
• ± 50 % WITH MODERATE SEVERE
HEART FAILURE SURVIVE 2 YEARS
ASSESSMENT
HISTORY : KEEP IN MIND :
MANY CONDITIONS CAN LEAD TO
HEART FAILURE
CAREFULLY QUESTIONS THE CLIENT
ABOUT PAST MEDICAL HISTORY
ASK CLENT’S PERCEPTION OF HIS/HER
ACTIVITY TOLERANCE, BREATHING &
URINARY PATTERN, FLUID VOLUME
STATUS, CLIENT’S KNOWLEDGE ABOUT
HEART FAILURE
ASSESSMENT
LEFT VENTRICULAR FAILURE :
REPORT UNUSUAL FATIGUE
DESCRIBE THE COUGH AS IRRITATING, NOCTURNAL,
USUALLY NON-PRODUCTIVE
CONFUSION MAY OCCUR
REPORT CHEST DISCOMFORT
DESCRIBE PALPITATION, SKIPPED BEATS, FAST
HAERTBEAT
PRESENCE OF DYSPNEA, ORTHOPNEA
DESCRIBE SUDDEN AWAKENING WITH A FEELING
OF BREATHLESSNESS 2 – 5 HOURS AFTER FALLING
ASLEEP HAVE PAROXYSMAL NOCTURNAL DYSPNEA
ASSESSMENT
RIGHT VENTRICULAR FAILURE :
WEIGHT GAIN (EDEMA)
GASTROINTESTINAL COMPLAINTS
(NAUSEA & ANOREXIA)
DIURESIS AT REST
EXPERIENCE INCREASED THIRS &
TAKE IN EXESSIVE FLUID (4000-5000 ml)
ALDOSTERON SECRETION
PHYSICAL ASSESSMENT / CLINICAL
MANIFESTATIONS OF
LEFT SIDED HEART FAILURE :
1. DECREASED CARDIAC OUTPUT :
* FATIGUE
* OLIGURIA DURING THE DAY
* ANGINA
* CONFUSION
* DIZZINESS
* TACHYCARDIA, PALPITATIONS
2. PULMONARY CONGESTION :
* HACKING COUGH, WORSE AT NIGHT
* DYSPNEA
* CRACKLES IN LUNGS
* FROTHY PINK-TINGED SPUTUM
* TACHYPNEA
PHYSICAL ASSESSMENT / CLINICAL
MANIFESTATIONS OF
RIGHT SIDED HEART FAILURE :
• JUGULAR VENOUS DISTENTION
• ENLARGED LIVER
• ANOREXIA & NAUSEA
• DEPENDENT EDEMA (LEGS & SACRUM)
• POLYURIA AT NIGHT
PSYCHOLOGICAL ASSESSMENT
FEELINGS OF :
• REJECTION
• INSECURITY
• FRUSTRATION
• RAGE
LABORATORY ASSESSMENT
ELEVATED :
• BLOOD UREA NITROGEN
• SERUM CREATININE LEVEL
• CREATININE CLEARANCE LEVEL
• PROTEINURIA
• HIGH SPECIFIC GRAVITY
• HYPOXIA
• RESPIRATORY ALKALOSIS
• RESPIRATORY ACIDOSIS
RADIOGRAPHIS ASSESSMENT