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    Management of Cardiac Arrest

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    punctumlacrimale
    Cardiac arrest can be caused by four rhythms: ventricular fibrillation, pulseless ventricular tachycardia, pulseless electric activity, and asystole. For ventricular fibrillation and pulseless ventricular tachycardia, defibrillation within minutes of collapse can significantly increase survival chances, especially for witnessed arrests. While other ACLS therapies may increase rates of return of spontaneous circulation, they have not been shown to increase survival to hospital discharge. High quality CPR, therapeutic hypothermia, and early percutaneous coronary intervention can improve outcomes when combined, but do not necessarily do so individually.

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    Management of Cardiac Arrest

    Uploaded by

    punctumlacrimale
    176 views12 pages
    Cardiac arrest can be caused by four rhythms: ventricular fibrillation, pulseless ventricular tachycardia, pulseless electric activity, and asystole. For ventricular fibrillation and pulseless ventricular tachycardia, defibrillation within minutes of collapse can significantly increase survival chances, especially for witnessed arrests. While other ACLS therapies may increase rates of return of spontaneous circulation, they have not been shown to increase survival to hospital discharge. High quality CPR, therapeutic hypothermia, and early percutaneous coronary intervention can improve outcomes when combined, but do not necessarily do so individually.

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    Cardiac arrest

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    Cardiac arrest can be caused by four rhythms: ventricular fibrillation, pulseless ventricular tachycardia, pulseless electric activity, and asystole. For ventricular fibrillation and pulseless ventricular tachycardia, defibrillation within minutes of collapse can significantly increase survival chances, especially for witnessed arrests. While other ACLS therapies may increase rates of return of spontaneous circulation, they have not been shown to increase survival to hospital discharge. High quality CPR, therapeutic hypothermia, and early percutaneous coronary intervention can improve outcomes when combined, but do not necessarily do so individually.

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    © All Rights Reserved
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    Download as pptx, pdf, or txt
    176 views12 pages

    Management of Cardiac Arrest

    Uploaded by

    punctumlacrimale
    Cardiac arrest can be caused by four rhythms: ventricular fibrillation, pulseless ventricular tachycardia, pulseless electric activity, and asystole. For ventricular fibrillation and pulseless ventricular tachycardia, defibrillation within minutes of collapse can significantly increase survival chances, especially for witnessed arrests. While other ACLS therapies may increase rates of return of spontaneous circulation, they have not been shown to increase survival to hospital discharge. High quality CPR, therapeutic hypothermia, and early percutaneous coronary intervention can improve outcomes when combined, but do not necessarily do so individually.

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    Management of

    Cardiac Arrest
    Presented By
    Budi Yuli Setianto, MD, PhD, FIHA,
    FINASIM, FAsCC, FAPSIC
     Cardiac arrest can be caused by 4 rhythms: ventricular fibrillation (VF),
    pulseless ventricular tachycardia (VT), pulseless electric activity (PEA), and
    asystole.

     VF represents disorganized electric activity, whereas pulseless VT represents


    organized electric activity of the ventricular myocardium. Neither generates
    significant forward blood flow.

     PEA encompasses a heterogeneous group of organized electric rhythms


    associated with either absence of mechanical ventricular activity or
    mechanical ventricular activity that is insufficient to generate a clinically
    detectable pulse.

     Asystole ( ventricular asystole ) represents absence of detectable ventricular


    electric activity with or without atrial electric activity.
     For VF/pulseless VT, attempted defibrillation within minutes of collapse. For
    victims of witnessed VF arrest, early CPR and rapid defibrillation can
    significantly increase the chance for survival to hospital discharge.

     Other ACLS therapies such as some medications and advanced airways,


    although associated with an increased rate of ROSC, have not been shown to
    increase the rate of survival to hospital discharge.

     Combination of higher quality CPR and post arrest interventions such as


    therapeutic hypothermia and early percutaneous coronary intervention (PCI),
    doesn’t necessarily improves the outcome.

     Periodic pauses in CPR should be as brief as possible and only as necessary to


    assess rhythm, shock VF/VT, perform a pulse check when an organized
    rhythm is detected, or place an advanced airway.
     Monitoring and optimizing quality of CPR is encouraged and includes:

     Optimizing chest compression rate and depth, adequacy of relaxation, and


    minimization of pauses.
     Monitoring partial pressure of end-tidal CO2 [PETCO2], arterial pressure
    during the relaxation phase of chest compressions, or [ScvO2] when feasible.

     In the absence of an advanced airway, a synchronized compression–


    ventilation ratio of 30:2 is recommended at a compression rate of at least 100
    per minute.

     After placement of an advanced airway, the provider performing chest


    compressions should deliver at least 100 compressions per minute without
    pauses for ventilation.

     The provider delivering ventilations should give 1 breath every 6 to 8 seconds


    (8 to 10 breaths per minute) and should avoid delivering an excessive number
    of ventilations.
    CARDIAC ARREST ALGORITHM

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