Case Series Cap
Case Series Cap
Case Series Cap
CAP-LR
GENERAL DATA
• F.S, is 26 years old female, unemployed, Iglesia
ni Cristo, born on May 21, 1991 in Zamboaga
del sur, currently residing in Novaliches
Quezon City, consulted for the 1st time in our
institutioin
CHIEF COMPLAINT
• Productive cough
HISTORY OF PRESENT ILLNESS
• 11 days prior to consult, patient felt itchiness of the throat causing
nonproductive cough without associated fever, nausea, vomiting
and difficulty of breathing. It was associated with sore throat and
colds. No medication taken. No consult done.
EOSINOPHILS 1.8
BASOPHILS 0.8
• CHEST XRAY
– Pneumonia, Right Middle Lobe
• O> Patient is conscious, coherent, not in cardio
respiratory distress
• VITAL SIGNS:
– BP:140/90mmHg HR: 91 bpm RR: 18 cpm Temp:36.8 C
• SKIN: Skin is fair in color, warm to touch, with good
skin turgor. The nails are trimmed, with capillary refill
time of <2secs. No lesions, no scaling or thickenings
noted.
• HEENT: Anicteric sclera, pink palpebral
conjunctiva, no nasoaural discharge, no
tonsillopharyngeal congestion, no cervical
lymphadenopathy
• Chest&Lungs: Symmetrical chest expansion, no
retractions, no lagging, vesicular breath sounds
• HEART: Adynamic precordium, PMI at 5th ICS,
LMCL, normal rate, regular rhythm, no murmur
• EXTREMITIES: Grossly normal extremities, no
cyanosis, no edema, no pallor, full equal
peripheral pulses. No limitations in doing the
range of motions.
• A> CAP- LR
Hypertension Suspect
Decrease compliance
Hypoxemia
respiratory alkalosis
Death
PATTERNS OF PNEUMONIA
A. Congestion
• Usually seen in the 1st 24 hours
• Not usually seen by the physician
• Grossly appears red & doughy
• Microscopically: (+) vascular congestion & alveolar edema +
microorganism can spread via pores of Kohn (within the
alveoli)
B. Red Hepatization
• After 24 hours to 2 days
• Inflammatory mediators cause leakiness of the alveolar capilliary causing the
RBC to be able to get through the alveoli including the neutrophils
• Many RBC, neutrophil, desquamated epithelial cells & fibrin
C. Gray Hepatization
• Last 2-3 days
• Lung is dry, friable and gray-brown to yellow as a consequence of a persistent
fibrinopurulent exudates, progressive disintegration of RBC, hemosiderin,
marcophages and neutrophills.
• No RBC but lot of fibrin, start of the rebuilding / reconstitution of the lung
D. Resolution
• Enzymatic digestion of alveolar exudates, resorption and phagocytosis
• Lots of phagocytes, cleans up the debris
• After this, patient recovers from pneumonia
PATTERNS OF PNEUMONIA
2. Bronchopneumonia-margin usually poorly
demarcate neutrophilic infiltrates center in
bronchi, and bronchiole
• 4. Miliary Pneumonia
Etiology
Routes of Micro-organism
• Remarks:
• Essentially normal chest
xray
• Findings:
• Hazy infiltrates seen in the
right upper and both lower
lobes
• Heart is not enlarged
• Aorta is prominent
• Diaphragm, sulci and
bony thorax is intact
• Impression:
• Pneumonia, right upper
and both lower lobes
• Atheromatous aorta
Should a chest radiograph be repeated
routinely?
• a repeat chest x-ray is NOT needed for patients
with low-risk CAP who are recovering
satisfactorily
• if the patient with CAP is not clinically improving
or shows progressive disease
• no need to repeat a chest radiograph prior to
hospital discharge in a patient who is clinically
improving
• repeat radiograph is recommended during a
follow-up office visit
Microbiologic Studies
Low-risk CAP (with or without comorbid
conditions)
• most common etiologic agents are bacterial
(S. pneumoniae, H. influenzae) and atypical
pathogens (M. pneumoniae, C. pneumoniae)
• sputum Gram stain and culture may not be
done, EXCEPT:
– failure of clinical response to previous antibiotics
– clinical conditions in which drug resistance may be
an issue
Moderate and High-risk CAP
• more pathogens to consider (enteric Gram
negatives, P. aeruginosa, S. aureus, L.
pneumophila)
• two sites of blood cultures are recommended
prior to starting any antibiotic treatment
Atypical pathogens (M. pneumoniae, C.
pneumoniae and L. pneumophila)
• most common methods for diagnosis include
serology [a fourfold increase in IgG or IgM
titers or an initially high IgG or IgM titer],
culture, and PCR of respiratory specimens
• L. pneumophila, urine antigen test (UAT) to
detect serotype 1 and direct fluorescent
antibody test (DFA)
TREATMENT
Empirical treatment
STREAMLINING OF ANTIBIOTIC
THERAPY
Indications
Antibiotic dosage of oral agents for
streamlining or switch therapy
Prognosis of Community-Acquired
Pneumonia
Negative prognostic factors in community-acquired
pneumonia (CAP) include preexisting lung disease,
underlying cardiac disease, poor splenic function,
advanced age, multilobar involvement, and delayed
initiation of appropriate antimicrobial therapy.
http://emedicine.medscape.com/article/234240-overview#a5
Recommended hospital discharge criteria