Pages that link to "Q34318060"
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The following pages link to The candidate tumor suppressor gene BLU, located at the commonly deleted region 3p21.3, is an E2F-regulated, stress-responsive gene and inactivated by both epigenetic and genetic mechanisms in nasopharyngeal carcinoma. (Q34318060):
Displaying 50 items.
- BS69, a specific adaptor in the latent membrane protein 1-mediated c-Jun N-terminal kinase pathway (Q24537608) (← links)
- Transcriptional patterns, biomarkers and pathways characterizing nasopharyngeal carcinoma of Southern China (Q24647880) (← links)
- Chromosomal imbalances in nasopharyngeal carcinoma: a meta-analysis of comparative genomic hybridization results (Q25255613) (← links)
- Functional epigenetics identifies a protocadherin PCDH10 as a candidate tumor suppressor for nasopharyngeal, esophageal and multiple other carcinomas with frequent methylation (Q28278843) (← links)
- A novel isoform of the 8p22 tumor suppressor gene DLC1 suppresses tumor growth and is frequently silenced in multiple common tumors (Q28385864) (← links)
- OPCML is a broad tumor suppressor for multiple carcinomas and lymphomas with frequently epigenetic inactivation. (Q33361612) (← links)
- Promoter methylation inhibits BRD7 expression in human nasopharyngeal carcinoma cells (Q33367808) (← links)
- Diagnostic Capacity of RASSF1A Promoter Methylation as a Biomarker in Tissue, Brushing, and Blood Samples of Nasopharyngeal Carcinoma (Q33603685) (← links)
- Distinct Epigenetic Domains Separated by a CTCF Bound Insulator between the Tandem Genes, BLU and RASSF1A (Q33705017) (← links)
- Epigenetic identification of ADAMTS18 as a novel 16q23.1 tumor suppressor frequently silenced in esophageal, nasopharyngeal and multiple other carcinomas (Q33874587) (← links)
- The epigenetic modifier PRDM5 functions as a tumor suppressor through modulating WNT/β-catenin signaling and is frequently silenced in multiple tumors. (Q34075626) (← links)
- The ubiquitin peptidase UCHL1 induces G0/G1 cell cycle arrest and apoptosis through stabilizing p53 and is frequently silenced in breast cancer. (Q34141646) (← links)
- Methylation profiling of Epstein-Barr virus immediate-early gene promoters, BZLF1 and BRLF1 in tumors of epithelial, NK- and B-cell origins (Q34212911) (← links)
- Tumor suppressor BLU inhibits proliferation of nasopharyngeal carcinoma cells by regulation of cell cycle, c-Jun N-terminal kinase and the cyclin D1 promoter (Q34315482) (← links)
- Tumor suppressive role of a 2.4 Mb 9q33-q34 critical region and DEC1 in esophageal squamous cell carcinoma. (Q34373674) (← links)
- RBMS3 at 3p24 inhibits nasopharyngeal carcinoma development via inhibiting cell proliferation, angiogenesis, and inducing apoptosis (Q34408248) (← links)
- Identifying cancer driver genes in tumor genome sequencing studies (Q34480097) (← links)
- The tumor suppressor Wnt inhibitory factor 1 is frequently methylated in nasopharyngeal and esophageal carcinomas. (Q34612189) (← links)
- Frequent hypermethylation of RASSF1A and TSLC1, and high viral load of Epstein-Barr Virus DNA in nasopharyngeal carcinoma and matched tumor-adjacent tissues. (Q34769720) (← links)
- Oncogenic induction of cellular high CpG methylation by Epstein-Barr virus in malignant epithelial cells (Q35020438) (← links)
- Epigenetic silencing of a Ca(2+)-regulated Ras GTPase-activating protein RASAL defines a new mechanism of Ras activation in human cancers. (Q35927921) (← links)
- Deregulation of secreted frizzled-related proteins is associated with aberrant β-catenin activation in the carcinogenesis of oral submucous fibrosis. (Q36176784) (← links)
- The human cadherin 11 is a pro-apoptotic tumor suppressor modulating cell stemness through Wnt/β-catenin signaling and silenced in common carcinomas (Q36186624) (← links)
- 3p21.3 tumor suppressor cluster: prospects for translational applications (Q36429393) (← links)
- Epigenetic silencing of the 3p22 tumor suppressor DLEC1 by promoter CpG methylation in non-Hodgkin and Hodgkin lymphomas. (Q36517724) (← links)
- Anti-angiogenic pathway associations of the 3p21.3 mapped BLU gene in nasopharyngeal carcinoma. (Q36601451) (← links)
- Nasopharyngeal carcinoma: molecular pathogenesis and therapeutic developments (Q36810352) (← links)
- Evaluation of the 3p21.3 tumour-suppressor gene cluster (Q36833529) (← links)
- DNA methylation in hepatocellular carcinoma (Q37113559) (← links)
- DLEC1 is a functional 3p22.3 tumour suppressor silenced by promoter CpG methylation in colon and gastric cancers (Q37123409) (← links)
- Tumor suppressor genes on frequently deleted chromosome 3p in nasopharyngeal carcinoma (Q37183789) (← links)
- Role of DNA methylation in head and neck cancer (Q38019157) (← links)
- An update on viral association of human cancers (Q38082388) (← links)
- Mouse olfactory sensory neurons express 10,000 genes (Q38302590) (← links)
- Positional expression profiling indicates candidate genes in deletion hotspots of hepatocellular carcinoma (Q38309560) (← links)
- Epigenetic Alterations in Epstein-Barr Virus-Associated Diseases (Q38667123) (← links)
- Protocadherin20 Acts as a Tumor Suppressor Gene: Epigenetic Inactivation in Nasopharyngeal Carcinoma (Q38903947) (← links)
- Characterization of the nasopharyngeal carcinoma methylome identifies aberrant disruption of key signaling pathways and methylated tumor suppressor genes. (Q38931940) (← links)
- FEZF2, a novel 3p14 tumor suppressor gene, represses oncogene EZH2 and MDM2 expression and is frequently methylated in nasopharyngeal carcinoma (Q39151547) (← links)
- NOR1 is an HSF1- and NRF1-regulated putative tumor suppressor inactivated by promoter hypermethylation in nasopharyngeal carcinoma (Q39497820) (← links)
- Underlying mechanisms for LTF inactivation and its functional analysis in nasopharyngeal carcinoma cell lines (Q39576688) (← links)
- Phospholipase C delta 1 is a novel 3p22.3 tumor suppressor involved in cytoskeleton organization, with its epigenetic silencing correlated with high-stage gastric cancer. (Q39850176) (← links)
- Epigenetic disruption of interferon-gamma response through silencing the tumor suppressor interferon regulatory factor 8 in nasopharyngeal, esophageal and multiple other carcinomas. (Q39983693) (← links)
- Characterization of a novel epigenetically-silenced, growth-suppressive gene, ADAMTS9, and its association with lymph node metastases in nasopharyngeal carcinoma (Q39986557) (← links)
- Epigenetic inactivation of the deleted in lung and esophageal cancer 1 gene in nasopharyngeal carcinoma. (Q40209231) (← links)
- The major 8p22 tumor suppressor DLC1 is frequently silenced by methylation in both endemic and sporadic nasopharyngeal, esophageal, and cervical carcinomas, and inhibits tumor cell colony formation. (Q40252205) (← links)
- Hypermethylation of two consecutive tumor suppressor genes, BLU and RASSF1A, located at 3p21.3 in cervical neoplasias (Q40271074) (← links)
- Inactivation of Wnt inhibitory factor-1 (WIF1) expression by epigenetic silencing is a common event in breast cancer. (Q40311505) (← links)
- Tumor suppressor BLU promotes TRAIL-induced apoptosis by downregulating NF-κB signaling in nasopharyngeal carcinoma (Q40393121) (← links)
- Hypermethylation of RARβ2 correlates with high COX-2 expression and poor prognosis in patients with colorectal carcinoma (Q43008143) (← links)