Breast cancer cell proliferation is inhibited by BAD: regulation of cyclin D1. (Q40099414)

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2 January 2020

title

Breast cancer cell proliferation is inhibited by BAD: regulation of cyclin D1 (English)

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2 January 2020

3

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Arnold M Saxton

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2 January 2020

Seung Joon Baek

8

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2 January 2020

Maria Cekanova

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Richard G Pestell

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2 January 2020

author name string

Romaine Fernando

1

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2 January 2020

James S Foster

2

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2 January 2020

Anders Ström

4

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2 January 2020

Mahadev Rao

6

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2 January 2020

Kiyoshi Yamaguchi

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2 January 2020

Robert Donnell

10

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2 January 2020

Jay Wimalasena

12

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2 January 2020

language of work or name

English

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publication date

1 August 2007

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Journal of Biological Chemistry

2 January 2020

published in

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volume

282

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issue

39

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2 January 2020

page(s)

28864-28873

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A role for proapoptotic BID in the DNA-damage response

2 January 2020

cites work

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Bax and Bid, two proapoptotic Bcl-2 family members, inhibit homologous recombination, independently of apoptosis regulation.

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BAD is a pro-survival factor prior to activation of its pro-apoptotic function

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Bcl-2 exerts a pRb-mediated cell cycle inhibitory function in HEC1B endometrial carcinoma cells.

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Mono- and multisite phosphorylation enhances Bcl2's antiapoptotic function and inhibition of cell cycle entry functions

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BAD/BCL-[X(L)] heterodimerization leads to bypass of G0/G1 arrest

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Distinct BH3 domains either sensitize or activate mitochondrial apoptosis, serving as prototype cancer therapeutics

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Bad Is a BH3 Domain-Containing Protein That Forms an Inactivating Dimer with Bcl-XL

21 January 2018

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Serine phosphorylation of death agonist BAD in response to survival factor results in binding to 14-3-3 not BCL-X(L)

21 January 2018

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Estradiol abrogates apoptosis in breast cancer cells through inactivation of BAD: Ras-dependent nongenomic pathways requiring signaling through ERK and Akt

21 January 2018

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Estrogen-induced activation of Cdk4 and Cdk2 during G1-S phase progression is accompanied by increased cyclin D1 expression and decreased cyclin-dependent kinase inhibitor association with cyclin E-Cdk2.

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Cyclin D1 in breast cancer

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Cyclin D1 is required for transformation by activated Neu and is induced through an E2F-dependent signaling pathway

21 January 2018

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Inhibition of JNK activation through NF-kappaB target genes

21 January 2018

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The LIM-only protein Lmo2 is a bridging molecule assembling an erythroid, DNA-binding complex which includes the TAL1, E47, GATA-1 and Ldb1/NLI proteins

21 January 2018

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Multifaceted regulation of cell cycle progression by estrogen: regulation of Cdk inhibitors and Cdc25A independent of cyclin D1-Cdk4 function

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Estrogens and cell-cycle regulation in breast cancer

21 January 2018

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Induction of cyclin D1 by simian virus 40 small tumor antigen

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Opposing action of estrogen receptors alpha and beta on cyclin D1 gene expression

21 January 2018

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JNK phosphorylation and activation of BAD couples the stress-activated signaling pathway to the cell death machinery

21 January 2018

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AP-1 in cell proliferation and survival

21 January 2018

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AP-1 as a regulator of cell life and death

21 January 2018

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c-Jun regulates cell cycle progression and apoptosis by distinct mechanisms

21 January 2018

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Proapoptotic BID is an ATM effector in the DNA-damage response

21 January 2018

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Insulin/insulin-like growth factor-I and estrogen cooperate to stimulate cyclin E-Cdk2 activation and cell Cycle progression in MCF-7 breast cancer cells through differential regulation of cyclin E and p21(WAF1/Cip1).

21 January 2018

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Signal transduction through the Ras/Erk pathway is essential for the mycoestrogen zearalenone-induced cell-cycle progression in MCF-7 cells

21 January 2018

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Bad-deficient mice develop diffuse large B cell lymphoma

21 January 2018

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Survival factor-mediated BAD phosphorylation raises the mitochondrial threshold for apoptosis

21 January 2018

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BAD and glucokinase reside in a mitochondrial complex that integrates glycolysis and apoptosis

21 January 2018

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Cell survival promoted by the Ras-MAPK signaling pathway by transcription-dependent and -independent mechanisms

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The growth-promoting activity of the Bad protein in chicken embryo fibroblasts requires binding to protein 14-3-3

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Proapoptotic BAX and BAK modulate the unfolded protein response by a direct interaction with IRE1alpha

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21 January 2018

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