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    Hipoxaemia

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    Jaqueline Medeiros
    This study examined the effects of hypoxia, hyperoxia, and hypercapnia on renal blood flow in normal subjects and renal transplant recipients. Nine normal subjects and eight renal transplant recipients inhaled room air, varying levels of oxygen, and carbon dioxide. Renal blood flow was measured using Doppler ultrasonography. In normal subjects, renal blood flow increased with hypoxia and hypercapnia, and decreased with hyperoxia. Renal transplant recipients showed similar but attenuated responses. The study concluded that renal denervation does not completely abolish renal blood flow responses to inhaled oxygen and carbon dioxide.

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    Hipoxaemia

    Uploaded by

    Jaqueline Medeiros
    20 views1 page
    This study examined the effects of hypoxia, hyperoxia, and hypercapnia on renal blood flow in normal subjects and renal transplant recipients. Nine normal subjects and eight renal transplant recipients inhaled room air, varying levels of oxygen, and carbon dioxide. Renal blood flow was measured using Doppler ultrasonography. In normal subjects, renal blood flow increased with hypoxia and hypercapnia, and decreased with hyperoxia. Renal transplant recipients showed similar but attenuated responses. The study concluded that renal denervation does not completely abolish renal blood flow responses to inhaled oxygen and carbon dioxide.

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    This study examined the effects of hypoxia, hyperoxia, and hypercapnia on renal blood flow in normal subjects and renal transplant recipients. Nine normal subjects and eight renal transplant recipients inhaled room air, varying levels of oxygen, and carbon dioxide. Renal blood flow was measured using Doppler ultrasonography. In normal subjects, renal blood flow increased with hypoxia and hypercapnia, and decreased with hyperoxia. Renal transplant recipients showed similar but attenuated responses. The study concluded that renal denervation does not completely abolish renal blood flow responses to inhaled oxygen and carbon dioxide.

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    © All Rights Reserved
    Download as PDF, TXT or read online from Scribd
    Download as pdf or txt
    20 views1 page

    Hipoxaemia

    Uploaded by

    Jaqueline Medeiros
    This study examined the effects of hypoxia, hyperoxia, and hypercapnia on renal blood flow in normal subjects and renal transplant recipients. Nine normal subjects and eight renal transplant recipients inhaled room air, varying levels of oxygen, and carbon dioxide. Renal blood flow was measured using Doppler ultrasonography. In normal subjects, renal blood flow increased with hypoxia and hypercapnia, and decreased with hyperoxia. Renal transplant recipients showed similar but attenuated responses. The study concluded that renal denervation does not completely abolish renal blood flow responses to inhaled oxygen and carbon dioxide.

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    04/06/2019 Acute effects of hypoxaemia, hyperoxaemia and hypercapnia on renal blood flow in normal and renal transplant subjects.

    bjects. - PubMe…

    PubMed

    Format: Abstract Full text links

    Eur Respir J. 1998 Sep;12(3):653-7.

    Acute effects of hypoxaemia, hyperoxaemia and hypercapnia on


    renal blood flow in normal and renal transplant subjects.
    Sharkey RA1, Mulloy EM, O'Neill SJ.

    Author information

    Abstract
    The aim of this investigation was to study noninvasively the effects of hypoxaemia, hyperoxaemia
    and hypercapnia on renal blood flow in normal subjects and renal allograft recipients, i.e. with
    denervated kidneys. By comparing these two groups, the influence of renal innervation on any
    resulting changes in renal blood flow could be ascertained. Nine normal and eight renal allograft
    recipients were studied. Each subject inhaled the following gas mixtures in order: room air, 10%
    O2 (hypoxaemia), 10% O2 + baseline CO2 (isocapnic hypoxaemia), 10% O2 + high CO2
    (hypercapnic hypoxaemia), 100% O2 (hyperoxaemia), 100% O2 + baseline CO2 (isocapnic
    hyperoxaemia) and 100% O2 + high CO2 (hypercapnia hyperoxaemia). Using Doppler
    ultrasonography, the pulsatility index (PI), an index of renovascular resistance, was measured at
    the various gas inhalation levels. In normal subjects, the renovascular resistance increased in
    response to hypoxaemia, with a greater increase in response to hypercapnic hypoxaemia.
    Hyperoxaemia caused a decrease in renovascular resistance but this was abolished with the
    addition of CO2. There was a similar pattern in the PI response to the different gas inhalations in
    the renal transplant subjects, but these responses were attenuated in comparison with those of
    the normals. In conclusion, renal denervation does not completely abolish the renovascular
    responses to inhaled oxygen and carbon dioxide.

    PMID: 9762795

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    https://www.ncbi.nlm.nih.gov/pubmed/9762795 1/1

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