This document discusses the classification and types of musculoskeletal infections. It covers:
1. Classification systems for MSK infections based on duration, mechanism of infection, bone portion affected, and host status.
2. Specific types of bone infections including Garre sclerosing osteomyelitis, multifocal non-suppurative osteomyelitis, and infantile cortical hyperostosis.
3. The natural process of bone necrosis, sequestrum formation, and drainage of pus through a sinus tract in chronic osteomyelitis infections.
This document discusses the classification and types of musculoskeletal infections. It covers:
1. Classification systems for MSK infections based on duration, mechanism of infection, bone portion affected, and host status.
2. Specific types of bone infections including Garre sclerosing osteomyelitis, multifocal non-suppurative osteomyelitis, and infantile cortical hyperostosis.
3. The natural process of bone necrosis, sequestrum formation, and drainage of pus through a sinus tract in chronic osteomyelitis infections.
This document discusses the classification and types of musculoskeletal infections. It covers:
1. Classification systems for MSK infections based on duration, mechanism of infection, bone portion affected, and host status.
2. Specific types of bone infections including Garre sclerosing osteomyelitis, multifocal non-suppurative osteomyelitis, and infantile cortical hyperostosis.
3. The natural process of bone necrosis, sequestrum formation, and drainage of pus through a sinus tract in chronic osteomyelitis infections.
This document discusses the classification and types of musculoskeletal infections. It covers:
1. Classification systems for MSK infections based on duration, mechanism of infection, bone portion affected, and host status.
2. Specific types of bone infections including Garre sclerosing osteomyelitis, multifocal non-suppurative osteomyelitis, and infantile cortical hyperostosis.
3. The natural process of bone necrosis, sequestrum formation, and drainage of pus through a sinus tract in chronic osteomyelitis infections.
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LO WEEK 5 (2)
1. Classification of MSK infection
Duration (acute- subacute- chronic) and mechanism (haematogenous contagious focus of infection) Lewand Waldvogel Affected portion of bone, Host status and local environment (stage 1 medullary, 2 superficial,3 localized, 4 diffuse) Cierny and mader
Other types of bone infections
a. Garre Sclerosing Osteomyelitis Garré, in 1893, described a rare form of non- suppurative osteomyelitis which is characterized by marked sclerosis and cortical thickening. There is no abscess, only a diffuse enlargement of the bone at the affected site – usually the diaphysis of one of the tubu- lar bones or the mandible. b. Multifocal non-suppurative osteomyelitis 1) it is not as rare as initially suggested; (2) it comprises several different syndromes which have certain fea- tures in common; and (3) there is an association with chronic skin infection, especially pustular lesions of the palms and soles (palmo-plantar pustulosis) and pustular psoriasis. c. Infantile Cortical Hyperostosis (Caffey’s Disease) Infantile cortical hyperostosis is a rare disease of infants and young children. It usually starts during the first few months of life with painful swelling over the tubular bones and/or the mandible. The child may be feverish and irritable, refusing to move the affected limb. Infection may be suspected but, apart from the swelling, there are no local signs of inflammation. The ESR, though, is usually elevated.
d. Acute suppurative arthritis
(1) direct invasion through a penetrating wound, intra-articular injection or arthroscopy; (2) direct spread from an adjacent bone abscess; or (3) blood spread from a distant site. In infants it is often difficult to tell whether the infec- tion started in the metaphyseal bone and spread to the joint or vice versa.
2. How is the drainage of the sinus is done
Local spread of the infection within the medullary cavity further compromises the internal circulation The resultant area of bone necrosis eventually becomes separated from the living bone, therefore forming sequestra. Bacteria are able to survive and continue multiply within the tiny haversian canals and canaliculi of this island of avascular bone The surrounding pond of pus prevents revascularization of the sequestrum and thereby protects its bacterial inhabitants not only from the living leucocytes but also from the action of circulating antibiotics Extensive new bone formation from the deep layer of elevated periosteum produces an enveloping bony tube (involcrum), which maintains the continuity of the involved bone To remove the sequestra, there is natural process of spontaneous extrustion through an opening in the involcrum and hence through a sinus tract to the exterior. This leads to chronic phase of the disease 3. Saucerization and muscle flap transfer 4. How is IV done in osteomyelitis