Finals Maternal
Finals Maternal
Finals Maternal
Health Problems Common in Toddlers • Chemical burns – acids, alkali, organic compounds
· Are tissue injuries caused by contact with dry heat • Child abuse – immersion burns, contact burns (cigarettes)
(fire), moist heat (hot liquid or steam), chemicals, electricity,
radiation, lightening or extreme cold.
1. Extent of injury – measured by the percentage of total - result in destruction of the epidermis.
body surface area (TBSA) involved.
- physiologic functions remain intact and tissue damage
2. Depth of injury – based on the extent of destruction is minimal
§ superficial partial-thickness injury (First- degree burn) - the damage epithelium peels off in about 5-10 days
without scarring
§ Partial-thickness and deep partial-thickness injuries
(Second-degree burn) v Second-degree burns
§ Full thickness injury – third-degree burn a. Partial-thickness injuries result in destruction of the
epidermis and some of the dermis.
3. Severity of Injury – determined primarily by the extent
and depth of injury, but also - capillary damage occurs
by location of the injury - this type of burn usually heals spontaneously in about
14 days
· Major injury requires treatment at a specialized burn unit.
- scarring is minimal
· Moderate injury can be treated in a hospital.
b. Deep-partial thickness injuries result in destruction of
· Minor injuries can be treated on an outpatient basis. the epidermis and dermis
1
FINALS MATERNAL LECTURE
b. Burn shock – hypovolemic shock can occur when burns - moderate pain with severe pain on exposure to air or
affect more than 15% to 20% of TBSA due to massive water
capillary leakage
- third-degree burns
c. Growth retardation – growth hormone levels are
suppressed § Dry, leathery skin surface
d. Accelerated metabolic rate – energy expenditure § Cherry red, white, or black skin color
increases from 40% to 100% above basal levels associated
with increased catecholamine levels, hyperglycemia, and § Edema
increased nutritional needs
§ Blisters (rare)
e. Local infection and sepsis – burns create moist, warm
environments for bacteria, including the body’s own flora. § No pain in area burn; very painful surrounding areas
Gram-positive organisms s.a. staphylococcus, and gram-
Classifications:
negative organisms, particularly pseudomonas aeruginosa,
colonize by the third day
1. Minor
Nursing Process
• First-degree burns or second-degree burns less than
10% TBSA
1. Assessment Findings
2. Moderate
• dry skin area
3. Severe
- Second-degree burns
- red to pale ivory skin color
2
FINALS MATERNAL LECTURE
• Interrupted family processes 10. Encourage optimal physical functioning and minimize
scarring
• Imbalanced nutrition: less than body requirements
- conduct ROM exercises
- encourage mobility
Care for Major Burns
- splint the joints in extension when sleeping to prevent
1. Assess for signs of respiratory distress, burn shock, fluid, contractures
and electrolyte imbalance, altered metabolism, and infection.
Assess for signs of vascular heat loss (coolness, - encourage self-help
acrocyanosis, and mottling)
- wrap healing tissue with elastic bandages to minimize
2. Administer prescribed medications scarring
- topical or IV antimicrobials and antibiotics 12. Prepare the child and family for discharge
- vitamins A, B, and C, and iron and zinc - reinforce the need for follow-up care
- antipyretics
- assess diminished pulses and prolonged capillary refill - results from inhaling or ingesting lead-containing
substances
4. Prevent infection and promote wound healing
· Highest incidence: late infancy and toddlerhood
- maintain infection control precautions
- debride the eschar, crust, and blisters
Etiology
5. Maintain integrity of the skin graft
The child can be poisoned in 3 ways:
- maintain splints and dressings
1. Eating contaminated food or non-food substances
3
FINALS MATERNAL LECTURE
c. GI – acute crampy abdominal pain, vomiting, 3. Encourage fluids to enhance lead excretion
constipation, and anorexia
4. Monitor fluid I and O to evaluate kidney function
d. Musculoskeletal – short stature and lead lines in bones on
x-ray films 5. Prepare the child and family for interventions, which vary
according to lead level scores.
e. Neurologic
a. Rescreen for lead in 1 year when the lead level is less
· Low-dose lead exposure: behavioral changes s.a. than 10 mcg/dl.
distractibility, hyperactivity, impulsivity, learning problems,
hearing impairment, and mild intellectual deficits b. Rescreen and provide family with lead education materials
when the lead level is 10-14 mcg/dl.
4
FINALS MATERNAL LECTURE
c. Rescreen, look for sources, and educate parents when the - low self-esteem
lead level is 15-19 mcg/dl. If this lead level persists, initiate
actions for a lead level of 20-44 mcg/dl. - substance abuse
f. Begin treatment and environmental clearance immediately - illness, disability, developmental delay
when the lead level is 70 mcg/dl and over.
- illegimate or unwanted pregnancy
6. Perform prescribed serial urine testing during chelation
therapy to monitor kidney status and the rate and volume of - hyperkinesis
lead excretion.
- failure to bond
- resemblance to someone the parent does not like
4. Child Abuse
3. Environmental Factors
· Used to describe acts of commission or omission by
- chronic stress
caregivers that prevent a child from actualizing his or her
potential growth and development.
- poverty, poor housing, and unemployment
Types
- divorce
1. Physical Abuse – the intentional infliction of injury to a
- frequent relocation
child
4. Factors Specific to Sexual Abuse
2. Emotional Abuse – the deliberate attempt to destroy
the child’s self-esteem or competence
- the abuser is typically a male whom the victim knows
· Emotional Abuse
- poor social-emotional support system
5
FINALS MATERNAL LECTURE
- Behavioral indicators
- Carefully assess the child’s emotional status and
behavior.
v Under 5 years old: regression, feeding or toileting
disturbances, temper tantrums, requests for frequent
- Provide the child with positive attention and age-
underwear changes, and seductive behavior.no
appropriate play and activities.
6
FINALS MATERNAL LECTURE
2. The child experiences less fear and anxiety. 1. Cerebral palsy commonly results from existing prenatal
brain abnormalities.
3. The parents demonstrate positive interactions with the
child. 2. Prematurity is the single most important determinant of
cerebral palsy.
3. Other prenatal or perinatal risk factors include asphyxia,
5. Cerebral Palsy ischemia, perinatal trauma, congenital and perinatal
infections, and perinatal metabolic problems, such as
· This is a group of disabilities caused by injury to the brain hyperbilirubinemia and hypoglycemia.
either before or during birth, or in early infancy.
4. Infection, trauma, and tumors can cause cerebral palsy in
· The most common permanent disability of childhood. early infancy.
b. Clinical hallmarks include hypertonicity with poor control 4. Disabilities usually result from injury to the cerebellum,
of posture, balance and coordinated movement, and the basal ganglia, or the motor cortex.
impairment of fine and gross motor skills. Active attempts at
motion increase the abnormal postures and lead to overflow 5. It is difficult to establish the precise location of neurologic
of movement to other parts of the body. lesions because there is no typical pathologic picture. In
some cases, the brain has gross malformations, in others,
c. Common types: vascular occlusion, atrophy, loss of neurons, and
degeneration may be evident.
· Hemiparesis – one side of the body is affected
6. Cerebral palsy is nonprogressive but may become more
· Quadriparesis – all four extremities are affected apparent as the child grows older.
7
FINALS MATERNAL LECTURE
6. Severe cases may be observed at birth; mild and d. Technology such as computer use, may help children
moderate cases usually are not detected until the child is 1 with severe articulation problems.
or 2 years old. Failure to achieve milestones may be the first
sign. 9. As necessary, seek referrals for corrective lenses and
hearing devices to decrease sensory deprivation related to
7. Diagnosis is based on the following: vision and hearing losses.
a. Prenatal, birth, and postnatal history 10. Help promote a positive self-image in the child.
c. Assessment of muscle tone, behavior and abilities. b. Set realistic and attainable goals.
2. Prevent physical deformity by ensuring correct use of 12. Prepare the child and family for procedures, treatments
prescribed braces and other devices, and by performing and surgeries, if needed.
ROM exercises.
Health Problems Common in Pre-schoolers
3. Promote mobility by encouraging the child to perform
age- and condition-appropriate motor activities. A. Leukemia
5. Foster relaxation and general health by providing rest It is a proliferation of abnormal WBCs
periods.
Several different types of leukemia exist, and
6. Administer prescribed medications, which may include classification has become a complex process.
sedatives, muscle relaxants, and anti-convulsion.
The most common leukemia in children is acute
7. Encourage self-care by urging the child to participate in lymphocytic leukemia (ALL), which is a
activities of daily living (ADLs) (e.g. using utensils and proliferation of blast cells (immature lymphocytes.
implements that are appropriate for the child’s age and ALL is classified by form, structure and
condition). morphology of the blast cells.
8
FINALS MATERNAL LECTURE
Several genetic diseases have been associated e. CNS symptoms (if there is CNS metastasis) – headache,
with increase incidences of leukemia, including meningeal irritation and signs of increased ICP.
Down Syndrome, Fanconi anemia and Bloom
syndrome. f. General symptoms: weight loss, anorexia, vomiting
Malignant leukemia cells arise from precursor cells a. CBC may reveal normal, decreased or increased WBC
in blood-forming elements. count with immature cells (blasts), decreased RBCs, and
decreased platelets.
These cells can accumulate and crowd our normal
bone marrow elements, spill peripheral blood, and b. Bone marrow aspiration confirms the diagnosis by
eventually invade all body organs and tissues. revealing extensive replacement of normal bone marrow
elements by leukemic cells.
Replacement of normal hematopoietic elements by
leukemic cells results in bone marrow suppression, c. Lumbar puncture assesses abnormal cell migration to the
which is marked by a decreased production of CNS.
RBCs, normal WBCs, and platelets.
Nursing Diagnoses
Bone marrow suppression results in anemia from
decreased RBC production, predisposition to 1. Risk for injury
infection due to neutropenia, and bleeding
tendencies due to thrombocytopenia. These put 2. Risk for infection
the child at risk of death from infection or
3. Risk for trauma
hemorrhage.
d. Hepatosplenomegaly, bone pain, and lymphadenopathy 7. The child’s oral mucous membranes will remain intact.
9
FINALS MATERNAL LECTURE
9. The child will maintain appropriate growth and 4. Prevent trauma from bleeding and immobility.
development.
5. Ensure adequate hydration.
10. The child and family will receive adequate support.
6. Prevent mucosities.
11. The child and family will cope with the possibility of
death 7. Prevent pain.
1. Assist in ensuring partial or complete remission from the 8. Assist the family in coping with their child’s disorder.
disease by administering chemotherapy and by preventing or
minimizing, the complications of chemotherapy, radiation 9. Assist the child and family with the grieving process
and bone marrow transplant (BMT).
Follow guidelines and institutional policies for
administration. B. Wilm's Tumor (Nephroblastoma)
- Graft rejection or failure – fever, infection, decreased a. Stage I: tumor is confined in one kidney
blood count
b. Stage II: the tumor extends beyond kidney but can be
2. Monitor for, and minimize pediatric oncologic resected
emergencies
c. Satge III: the tumor has residual nonhematogenous tumor
3. Prevent infection. cells confined to the abdomen.
10
FINALS MATERNAL LECTURE
- monitor bowel sounds and assess for signs and e. Rapid changes in environmental temperatures
symptoms of intestinal obstruction resulting from abdominal
surgery f. Exercise
Pathophysiology
Asthma is a chronic, reversible, obstructive airway a. There is an initial release of inflammatory mediators from
disease, characterized by wheezing. bronchial mast cells, epithelial cells, and macrophages,
followed by activation of other inflammatory cells.
It is caused by a spasm of the bronchial tubes, or
the swelling of the bronchial mucosa, after b. Alterations of autonomic neural control of airway tone and
exposure to various stimuli. epithelial integrity occur and the increased responsiveness in
airway smooth muscle results in clinical manifestations
It is the most common chronic disease in (wheezing and dyspnea).
childhood. Most children experience their first
symptoms by 5 years of age. 2. Three events contribute to clinical manifestations.
11
FINALS MATERNAL LECTURE
b. Inflammation and edema of the mucosa 7. Provide child and family teaching
c. Production of thick mucus, which results in increased 8. Refer the family to appropriate community agencies for
airway resistance, premature closure of airways, assistance.
hyperinflation, increased work of breathing, and impaired
gas exchange.
3. If not treated promptly, status asthmaticus – an acute, D. Urinary Tract Infection (UTI)
severe, prolonged asthma attack that is unresponsive to the
usual treatment – may occur, requiring hospitalization. Description
f. Pregnancy
4. Explain the possible use of hyposensitization therapy.
g. Noncircumcision
5. Help the child cope with poor self-esteem.
12
FINALS MATERNAL LECTURE
i. Antimicrobial agents that alter normal urinary tract flora 1. Assess urinary status
2. Pyelonephritis usually results from an ascending infection Ø DM is a chronic metabolic disorder that results from either
from the lower urinary tract. It can lead to acute and chronic a partial or complete deficiency of insulin
inflammatory changes in the pelvis and medulla, with
Ø Type 1 DM is characterized by pancreatic beta cells
scarring and loss of renal tissue.
destruction leading to absolute insulin deficiency.
3. Recurrent or chronic infection results in increased fibrotic
Ø Type 2 DM usually results from insulin resistance
tissue and kidney contraction
c. Ureteral catheterization, bladder washout procedures, and 4. In a state of insulin deficiency, glucagon, epinephrine,
radioisotope renography may be needed to localize the GH, and cortisol levels increase, secondary to fat breakdown,
infection. stimulating lipolysis, fatty acid release, and ketone
production.
d. Renal ultrasound
13
FINALS MATERNAL LECTURE
1. Clinical Manifestations
- Polydipsia a. Fasting blood sugar (FBS) will reveal a level above 120
mg/dl accompanied by a random blood glucose level above
- Polyuria 200 mg/dl
14
FINALS MATERNAL LECTURE
- Recognize signs of hypoglycemia early, and be alert to a. A type of lesion, called an Aschoff body (a proliferating,
when blood sugar levels are at their lowest. fibrin-like plaque), forms on the heart valve causing edema
and inflammation.
- Offer a readily absorbed carbohydrate such as orange
juice, to alleviate early symptoms. b. When the healed area becomes fibrous and scarred, the
valve leaflets fuse (stenosis), causing inefficiency and
-Administer glucagon to the unconscious child. leakage.
5. Provide child and family teaching c. The mitral and aortic valves are affected most often.
Ø RF usually occurs in children between 5 and 15 years of - Polyarthritis – swollen, hot, painful joints (usually large
age, with peak incidence at 8 years of age. joints). Polyarthritis is the most common presenting
symptom and it occurs in about 75% of all cases of RF.
- Chorea – sudden aimless, irregular movements of the
Etiology
extremities; involuntary facial grimaces; speech
disturbances; emotional lability; muscle weakness; and
Ø The onset of RF usually occurs 2-6 weeks after an
movements that increase with stress and decreased with
untreated upper respiratory infection with group A beta-
rest. It occurs in about 10% of all cases.
hemolytic streptococci
- Erythema marginatum – clear-centered, transitory,
Ø It is believed that a genetic susceptibility to RF is
nonpruritic macules, with defined boarders. They are noted
associated with a state of immune hyperactivity to the
mostly on the trunk and proximal extremities. This occurs in
streptococcal antigens
about 5% of all cases.
Ø Exact etiology is unknown
- Subcutaneous nodules are non-tender lesions that may
persist, then resolve. They are located over bony
prominences. These rarely occur in RF.
Pathophysiology
1. The child becomes infected with group A beta-hemolytic
a. Minor Characteristics – fever, arthralgia and specific
streptococcal bacteria.
laboratory findings
2. Antibodies formed against these bacteria begin to
attack the connective tissue of the body, producing
inflammation, which affects the heart, joints, central nervous
Laboratory and diagnostic study findings
system and subcutaneous tissue.
a. Laboratory findings consistent with the Jones Criteria:
3. Cardiac involvement is characterized by carditis.
- Erythrocyte Sedimentation Rate (ESR) is elevated
15
FINALS MATERNAL LECTURE
- C-reactive protein (CRP) is elevated Ø It is one of the more common chronic diseases in
children.
- Acute-phase reactants
Ø The outcome is variable and unpredictable in individual
b. CBC will reveal transient anemia and elevated white blood children. Even in its most severe forms, JRA is rarely life-
count (WBC) threatening.
3. Promote rest by organizing nursing care to allow for Ø Neutrophils and macrophages ingest immune complexes,
adequate rest periods. releasing enzymes and damaging joints.i
4. Alleviate discomfort of fever and arthralgia Ø The synovial becomes inflamed, excessive fluid is
produced, and thickened villi and nodules are produced into
5. Prevent skin breakdown the joint cavity.
- Promote prevention of RF by encouraging proper - joint involvement is usually confined to the lower
evaluation and treatment of streptococcal infections. extremities.
16
FINALS MATERNAL LECTURE
and tissue involved in focusing the eye). Inflammation of iris 2. Administer prescribed medications (NSAIDS – ibuprofen,
alone is called anterior uvetitis or iritis. aspirin ( ASA), naproxen sodium)
- Systemic symptoms are mild and may include low-grade - Use assistive devices
fever, fatigue and slowed growth.
- Teach splint application
3. Encourage activities with family and peers.
4. Assist the family in meeting the child’s needs.
Laboratory and Diagnostic Study Findings
a. Systemic
D. Scabies
- CBC will reveal leukocytosis and anemia
Description
- ESR will be elevated
Ø Scabies is usually spread by close, prolonged skin-to-skin
- CRP will be elevated contact (e.g. holding hands), and is common in school-aged
children.
- RF (rheumatoid factor) is negative
Ø The mites and their eggs may live on clothes or bed linen
- ANA (antinuclear antibody) is negative for one to two days.
- ESR will be elevated Ø Treatment should be repeated one week after the first
treatment. Do not apply the treatment more than twice.
- ANA may be positive
Etiology
Ø Human scabies is caused by an infestation of the skin by
Nursing Management
the human itch mite (Sarcoptes scabiei var. hominis). The
17
FINALS MATERNAL LECTURE
microscopic scabies mite burrows into the upper layer of the Ø Scabies rash. A pimple-like (papular) itchy (pruritic)
skin where it lives and lays its eggs. “scabies rash” is also common.
Image Source: Dr. P. Marazzi / Photo Researchers, Inc. Ø Physical exam. Clinical findings include primary and
secondary lesions; primary lesions are the first manifestation
of the infestation and typically include small papules,
vesicles, and burrows; secondary lesions are the result of
18
FINALS MATERNAL LECTURE
rubbing and scratching, and they may be the only clinical effectiveness and inspecting for any signs and symptoms of
manifestation of the disease. adverse effects; and determine the appropriate pain relief
method.
Nursing Diagnoses
Evaluation
1. Risk for infection related to tissue damage.
Nursing goals are met for a patient with scabies as
2. Impaired skin integrity related to edema. evidenced by:
3. Acute pain related to injury to biological agents. 1. Patient remained free of infection, as evidenced
by normal vital signs and absence of signs and
4. Disturbed sleep pattern related to itchiness and symptoms of infection.
pain of lesions.
2. Patient and folks demonstrated an understanding
of plan to heal tissue and prevent injury.
Nursing Care Planning and Goals 3. Patient and folks described measures to protect
and heal the tissue, including wound care.
1. Patient remains free of infection, as evidenced by
normal vital signs and absence of signs and symptoms of 4. Patient described satisfactory pain control at a
infection. level less than 3 to 4 on a rating scale of 0 to 10.
4. Patient describes satisfactory pain control at a level less Ø It is a common childhood condition that can be passed
than 3 to 4 on a rating scale of 0 to 10. among friends and family.
19
FINALS MATERNAL LECTURE
Ø It is possible, but uncommon, to get lice by sharing The floor and furniture should be vacuumed,
personal belongings such as hats or hairbrushes. particularly where the infested person sat or lay.
However, spending time and money on
Ø Personal hygiene has nothing to do with getting head lice. housecleaning activities is not necessary to
prevent reinfestation by lice or nits that may have
Ø Head lice are at their most active at night, causing fallen off the head or crawled onto furniture or
irritability and difficulty sleeping for the human host. Itching clothing, due to the organism’s short lifespan.
of the scalp is not sufficient for diagnosis of active Head lice survive fewer than two days if they fall
infestation. off a person and cannot feed. Nits cannot hatch
and usually die within a week if they are not kept
Ø Evidence suggests that itching may not develop for at the same temperature as that found close to
several weeks or months after the initial infestation and may the scalp.
persist for days or weeks after successful eradication of the
head lice.
E. Impetigo
Ø Lice (pediculosis) and their eggs (nits) can infest the body Ø Impetigo is a highly contagious bacterial infection often
in any of the aforementioned locations but primarily choose found on and around the mouth of the child or elsewhere on
areas that have longer hair. In children, one is most likely to the face. It can also appear on the hands, neck, trunk,
find lice on the head and the live bugs tend to live near the buttocks, or extremities
nape of the neck and behind the ears. They lay their eggs at
the base of the hair shaft, where they can be seen as Etiology:
pearlescent tear drops
Ø Impetigo may be caused by Staphylococcus aureus (S.
aureus) and Streptococcus pyogenes (S. pyogenes) or both
(Watkins, 2005).
Nursing Care: The nurse can instruct the parents that
visual inspection of the home, including clothes and
bedding, is important but it is often difficult to see lice
because of their small size Pathophysiology
Clothing and items that are not washable can be 2. Teach patient about wound care
dry-cleaned or sealed in a plastic bag and stored
for two weeks.
20
FINALS MATERNAL LECTURE
3. Educate the patient not to touch lesions and washing Ø Scoliosis may result from leg-length discrepancy, hip or
hands knee contractures, pain, neuromuscular disorders, or
congenital malformations. However it is usually idiopathic.
4. Educate caregiver on contact precautions
Ø Evidence points to a probable genetic autosomal dominant
5. Assess fever trait with incomplete penetrance; or to multifactorial causes.
8. Educate patient about the disease 1. Deformity progresses during periods of growth
(adolescent growth spurt) and stabilizes when vertebral
9. Educate about follow up because some patients may growth ceases.
develop glomerulonephritis
2. As the spine grows and the lateral curve develops, the
10. Educate caregiver to keep the child at home for 48 vertebrae rotate, causing the ribs and spine to rotate toward
hours after antibiotics have started because the infection is the convex part of the spine. Spinous processes rotate
highly contagious. toward the concavity of the curve.
3. The nose is a common reservoir and carriers can be 7. If significant scoliosis goes uncorrected, respiratory
treated with mupirocin (Bactroban Nasal) applied in the function is compromised and vital capacity is reduced;
nostrils. eventually, pulmonary hypertension, cor pulmonale and
respiratory acidosis may develop.
Ø It is a spinal deformity that usually involves lateral a. Scoliosis is asymptomatic most of the time and goes
curvature of the spine, spinal rotation and thoracic unrecognized until there some degree of deformity.
hypokyphosis.
b. The first signs of scoliosis include:
Ø The most common spinal deformity.
-Presence of a spinal curve
Ø During adolescence, scoliosis is more common in girls.
-asymmetry of scapula and extremities
Ø Untreated scoliosis may lead to back pain, fatigue,
disability, and heart and lung complications. -unequal distance between the arms and waist.
21
FINALS MATERNAL LECTURE
b. an MRI scan is used to evaluate the possibility of 1. Prevent physical and emotional trauma related to wearing
intraspinal pathology. a brace.
1. Functional Scoliosis- caused by poor posture and not by - Encourage verbalization of concerns and feelings.
spinal disease
- Assist the child in selecting clothing that will conceal the
Flexible and easily correctible brace.
2. Structural Scoliosis- anatomical change in shape of - Encourage positive aspects of wearing the brace
thorax or vertebrae including improved posture and symptom relief.
Hips and shoulders are uneven - Assist the child and family in developing the coping
skills.
Not easily correctible/ may need medical interventions
c. Promote normal growth and development by
It may be congenital encouraging self-care activities and peer socialization.
Neuromuscular scoliosis- result of muscle weakness or d. Provide family support by referring parents to social
imbalance services or an appropriate support group.
· Halo traction- used when there is associated weakness or d. Maintain skin integrity and prevent breakdown.
paralysis of the neck and truk muscle
e. Promote adequate bowel and bladder elimination.
Note: Curves up to 20 degrees do not require treatment.
- Prevent constipation by assessing bowel sounds.
- Prevent urinary complications by providing catheter
Nursing Management care.
22
FINALS MATERNAL LECTURE
- Promote adequate fluid and nutritional intake. Maintain Ø Soft tissue injuries usually accompany traumatic fractures
intravenous therapy until oral feedings are allowed. in adolescents involved in sports and adventurous activities.
- Provide the child and family with information about 1. Contusion- tearing of subcutaneous tissue results in
scoliosis and its treatment. Include information about the hemorrhage, edema and pain. Hematoma is evident.
equipment used during treatment.
2. Sprain- when ligament is torn or stretched away from
the bone at the point of trauma. Swelling, disability and pain
are major signs.
B. Bone Tumor (Osteosarcoma)
3. Strain- microscopic tear of the muscle or tendon occurs
Ø It is a primary malignant tumor of the long bones over time and results in edema and pain.
involving rapidly growing bone tissue (mesenchymal matrix-
forming cells).
23
FINALS MATERNAL LECTURE
· General name given to infections spread through · primary amenorrhea include agenesis (no formation) of
direct sexual activities. the uterus, Turner’s syndrome (genetic disorder with pectus
excavatus, heart murmur, and short stature), imperforate
· The occurrence of STI in a prepubertal stage must hymen, and constitutional delay.
prompt investigation for possible sexual abuse
· Certain medications can cause amenorrhea, including
Nursing Interventions chemotherapy and medroxyprogesterone acetate (Depo
Provera), which is given as a contraceptive injection.
· Review structures of reproductive system
· Review personal hygiene
Signs and Symptoms
· Discuss values and decision making, possible sexual
behaviors and consequences 1. Primary amenorrhea: the patient may exhibit
abnormalities in body habitus, suggestive of delayed
· Discuss prevention of pregnancies and STIs puberty. The Tanner stages of sexual characteristic
development may show delays.
2. Secondary amenorrhea
E. Amenorrhea
a. Signs and symptoms of pregnancy include mastalgia
Ø Amenorrhea refers to the absence of menses.
(breast tenderness); breast enlargement; nausea and
possibly vomiting, especially in the early morning;
Ø Primary amenorrhea is when no menses occur by the age
gastrointestinal upset; and urinary frequency. On
of 17.
examination, the uterus may be enlarged and Chadwick’s
sign (blue or violaceous cervix) may be present, a probable
Ø Secondary amenorrhea implies that menses have been
sign of pregnancy that becomes evident about the fourth
established, but have ceased for a minimum of 3 months.
week of gestation.
Causes:
b. hypothyroidism, the patient may have dry skin, dry hair,
· corpus luteum cyst fatigue, hoarseness, constipation, and an enlarged thyroid
gland. In hyperthyroidism, the patient may exhibit oily skin
· lactation and hair, diaphoresis, tachycardia, diarrhea, and a goiter
(enlarged thyroid gland).
· menopause (premature or normal)
c. Patients with polycystic ovarian syndrome may have
· hypothyroidism or hyperthyroidism hirsuitism (excessive facial and bodily hair) and obesity.
Corpus luteum cysts tend to cause pain in the lower
· chemotherapy quadrants that may be intermittent in nature, as some cysts
resolve spontaneously. Other cysts grow and may rupture,
· polycystic ovarian syndrome (PCOS) causing significant lower quadrant abdominal pain and even
peritoneal signs of rebound, guarding, and rigidity.
· diabetes mellitus
Diagnosis
· stress
a. Genetic testing - to determine disorders such as
· excessive exercise Turner’s syndrome.
F. Dysmenorrhea
24
FINALS MATERNAL LECTURE
Ø Discomfort in the lower abdomen and may radiate to the b. Counseling on diet and lifestyle
lower back or down the legs.
c. Encouraging exercise
Ø May be accompanied by nausea and vomiting
Types:
H. Eating Disorders
1. Primary- there is no evidence of pelvic abnormality;
affects 50 % of menstruating females and is the leading Ø Anorexia Nervosa (purging or withholding),
cause of short term recurrent school absenteeism in
adolescent girls Ø Bulimia Nervosa (binging and purging)
2. Secondary- pathologic condition is identified Ø Is a form of self-starvation seen mostly in adolescent girls
Ø Secondary dysmenorrhea most commonly results from · Failure to maintain minimum body weight for age and
endometriosis height
Ø Vitamins B and E and high level of omega 3 fatty acids · Dysfunctional family- controlling, rigid and imposing
G. Obesity
25
FINALS MATERNAL LECTURE
2. Weakness
14. Depression
4. Takes in larger amounts & over long periods of time.
Assessment Findings
1. A brief period of hospitalization may be necessary to 1. Physical: fatigue, repeated health complaints, red and
correct severe malnutrition and electrolyte imbalance glazed eyes, and a lasting cough
26
FINALS MATERNAL LECTURE
4. School: decreased interest, negative attitude, drop in abuse, especially if there is a strong family and genetic
grades, many absences, truancy, and discipline problems history of abuse. Two tools that can be used in the
identification of substance abuse are the CRAFFT (Knight,
5. Social problems: new friends who are less interested in Sherritt, Shrier, Harris, & Chang, 2002) and the CAGE
standard home and school activities, problems with the law, (Ewing, 1984). Both of these tools use simple acronyms to
and changes to less conventional styles of dress and music assist in the evaluation of drinking or drug use.
Complications: 2. Help the child and family find community resources that
may help conquer the substance abuse problem. Research
1. Delirium tremens & alcohol syndrome. indicates that nearly 80% of adolescents with substance
abuse receive treatment. There are many different types of
2. Korsakoff’s psychosis treatment, but the most promising appears to be a family-
based approach, as it shows the best outcomes for reduction
3. Wernicke’s syndrome
in substance abuse in adolescents. Family treatment means
that the entire family receives psychoeducation regarding
4. Peripheral neuropathies
substance abuse.
5. Hepatitis, cirrhosis, pancreatitis
6. Anemia
J. Suicide
Ø It is a deliberate self-injury with the intent to end one’s
life.
Stage 1:
Diagnosis of s substance use and abuse is based b. Family factors include conflict, parental rejection or
on the physical, emotional and social factors exhibited by the hostility, divorce and separation, relocation, unrealistic
child. A thorough family history is essential along with parental expectations, and parental indifference.
information about the child’s physical and emotional health.
c. Adolescent factors include hopelessness, depression,
substance abuse, impulsivity, difficulty tolerating frustration,
feelings of self-loathing of guilt, thought disorder, physical or
Nursing Care body image problems, gender identity concerns and a
perfectionist personality.
1. Assess drug and alcohol use in children. The nurse is in
an ideal position to identify adolescents at-risk for substance
27
FINALS MATERNAL LECTURE
· Insomnia
· Giving away cherished possessions • Organ donation questions, such as “How do you leave your
body to a medical school?”
· Preoccupation with death or death themes (e.g.
music, art, movies with death themes) • Sudden, unexplained elevation of mood. Mood elevation
may indicate that the individual has reached a decision about
· Statement of intention to commit suicide the suicide and feels relief.
Ø In younger adults, depression can be manifested by • A statement such as, “This is the last time you will see
behavioral problems me.”
· Self-destructive behavior
28
FINALS MATERNAL LECTURE
• Preference for art, music, and literature with themes of care. Ensure that the adolescent understands that he or she
death must cease or not implement this destructive behavior.
• Recent increase in interpersonal conflict with significant - Arrange for counseling and hospitalization if necessary;
others refer the adolescent and the family to a professional
therapist who will work with them through the crisis.
• Running away from home
2. Assist the family and friends in coping with loss. After a
• Recent experience of a friend or famous person committing completed suicide occurs, counsel the adolescent’s family
suicide and friends to help them understand work through their
grief.
• Inquiring about the hereafter
• Asking for information (supposedly for a friend) about
suicide prevention and intervention Other Interventions
• Almost any sustained deviation from the normal pattern of 1. Client expresses feelings of depression to health care
behavior providers or other adults, saying she will contact support
person should the desire to commit suicide become
overwhelming
Ø Ineffective individual coping 3. Try to find out the things in the child’s life that are still
viewed as important.
Ø Hopelessness
4. Since adolescent resort to suicide as a method of solving
Ø Altered family processes
their problems, helping them in this area can be an
intervention strategy.
Planning and Outcome Identification:
5. Help adolescents speak honestly about thoughts of suicide
1. An adolescent who has made suicide gestures or
and the problems that have led them to think that death is a
attempts will display improved self-esteem, positive
solution.
behaviors, and more effective coping and problem-solving
strategies.
6. A period of observation in a hospital setting is desirable
after a suicide attempt to prevent the adolescent from
2. Family and friends of an adolescent who committed a
inflicting personal injury again and to allow assessment in a
completed suicide will work through their grief and resolve
neutral setting, away from the stress that precipitated the
the loss over time.
attempt.
7. Antidepressant medicine alone, may be of little value in
treating depressed adolescents.
29
FINALS MATERNAL LECTURE
· achieving in school
Outcome evaluation
30