Electrocardiogram
Electrocardiogram
Electrocardiogram
RAM 1. Rhythmicity
-ability to contract at a regular timing
-aka EKG 4 PHASES
-measures electrical impulse of the heart Stimulation
-Willem Einthoven (1893), meeting the dutch medical Transmission
society Contraction
-6 LEAD ECG does not include chest Relaxation
-12 LEAD ECG (1942)
-unipolar lead 1942 resulted to advancement of the 12- 2. Contractility
LEAD ECG -ability to contract (make each cell shorter or
longer) in response to stimuli
Dextrocardia – apex part of the heart points to the
right. The heart is positioned on the right side of the 3. Automacity
chest instead of its normal position on the left side -pacemaker ability
-ability to pace or to spontaneously initiate or
WHY TAKE ECG propagate an action potential
Chest pain/discomfort
SOB 4. Excitability
Nausea -to respond to stimuli with the strongest possible
Weakness contraction or none at all
When food poisoned -influenced by: hormonal, nutritional balance,
Palpitation adequacy of O2 supply, drug therapy, and product
Anxiety of infection
Abdominal Pain 5. Refractoriness
Fainting -ability of the Myocardiogram to prevent from
responding to a new stimuli
HEART 6. Extensibility
- Main organ monitored -stretched during diastole as the heart fills with
- 4 chambers (Atria & Ventricles) blood
o Atria – receives blood -the longer the fiber, the more or stronger
o Ventricles – pumps blood contraction
*right-oxygenated *Left-deoxygenated 7. Conductivity
-heart muscle fibers to conduct or transmit
SYSTOLIC BLOOD PRESSURE electrical impulse to the next cell
When ventricles contract
DIASTOLIC BLOOD PRESSURE 12-LEAD ECG
When ventricles starts to refill, pressure in arteries LIMB LEADS (6-LEAD)
decrease simultaneously the atria contract creating *remember your great boyfriend
pressure RA: Red right arm
LA: Yellow left arm
VAGUS NERVE/10th CRANIAL NERVE LL: green left leg
-informs/commands heart to pump RL: Black R leg- no reading
-sends electrical impulse/vagal stimulation/
BIPOLAR STANDARD LIMB LEADS
1. SA node – stimulation causes atria to contract -leads 1, II, III
2. Internodal tracts -augmemented unipolar leads
3. AV Node – depolarization -leads aVR ( L to R), a VL, aVF (all right)
4. Bundle of his – contracts septum ( divides heart L&R)
5. Bundle of branches UNIPOLAR PRECORDIAL LEADS/CHEST LEADS
6. Purkinji Fibers V1 – Red 4th ICS Right sternal border
V2 – Yellow 4th ICS Left sternal border
V3 – Green midway b/n V2 & V4
V4 – brown 5th ICS Left Mid clavicular space THE WAVAFORMS
V5 – black, left axillary line, lateral and P-wave
horizontal to V4 - Atrial depolarization (contraction)
V6 – Violet, Horizontal to V5, middle of axilla - Upon SA stimulation
- SA node product
Note: when putting ECG, Px should be still and free of
metal Q-wave
- Septum depolarization
ROLE OF ECG MACHINE - Product of AV node
-recognize & record electrical activities in the heart
-prints out in ECG paper made of small boxes R-wave
- Ventricular depolarization
.04 - Bundle fibers/branches
Amplitude -
measured 0.2 s
S-wave
in millivolts - Purkinji fibers depolarization
1s
T-wave
Duration/Time - Ventricular repolarization
PR INTERVAL
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