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DIETARY CONTROL

Dr/ Yasmine Ahmed Mortada Elmaraghy


Lecturer of pediatric dentistry and oral public health
Faculty of dentistry Assiut University
Diet
Diet refers to the customary allowance of food
and drink taken by any person from day to
day.
Nutrition
Nutrition is a science of how the body utilises food to meet the
requirements for development, growth, repairs and
maintenance or as the science of food and its relationship to
health.
Balanced diet

Is a diet which contains varieties of food in such quantities &


proportions that the need for energy, amino acids, vitamins, fats,
carbohydrates & other nutrients is adequately met for
maintaining health, vitality, and general well being & also makes
provision for short duration of leanness
Diet and dental caries

The effect of diet is considered in


two heading
•Systemic effect
•Local effect
Systemic effect
 i.e. before eruption, while the tooth is still forming.

 1-Before birth through placental circulation from mother to


fetus, which provides calcium and vitamin D for proper
development of teeth.

 2-After birth through general nutrition where the essential


nutrients are carried by the blood stream from the

digestive tract.
Local effect

After tooth eruption


Lodgment of fermentable carbohydrates around teeth
Diet component

Carbohydrates Protein Fats vitamins


CLASSIFICATION OF CARBOHYDRATES

Carbohydrates that are of special interest and


importance in nutrition are

Polysaccharides
Monosaccharides Disaccharides
Monosaccharides

Monosaccharides are the simplest carbohydrates and are classified


according to the number of carbon atoms in the chain,
e.g. glucose, fructose and galactose.
Disaccharides
Disaccharides consist of a linkage of two monosaccharide units.
Disaccharides, in general, have a sweet taste, water soluble and are
crystalline solids,
e.g. sucrose, lactose and maltose.
Polysaccharides

Polysaccharides are complex carbohydrates made up of many (more than


10) monosaccharides linked together.
Unlike sugars, these are tasteless. Some are used for storing energy and
others perform structural functions,
e.g. starch, glycogen and cellulose.
Sugars
The sugars most commonly found in an
average diet are sucrose, glucose, fructose,
lactose, and maltose, from which some regard
sucrose as the most cariogenic.
When assessing a patient’s diet, it is essential
to differentiate between natural and added
sugars.
Classification of Sugars

➢ Natural sugars are those intrinsically found in the structure of fresh fruits and
vegetables, milk, and dairy products.
➢ Natural sugars do not play a significant role in developing dental caries and
other non-communicable diseases. This is probably due to their protective
components (polyphenolic compounds, calcium, water, and fiber) and the
hardness of some fruits and vegetables, which stimulates saliva production.
➢ Furthermore, cow’s milk is classed as non-cariogenic. It may even protect
teeth from dental caries thanks to its high calcium and casein content.
Classification of Sugars

➢ Added sugars, also called free sugars by the WHO, are the sugars added to
foods and drinks by manufacturers or cooks and sugars present in edible
products other than fresh fruit, vegetables, milk, and grains, e.g., fruit juices,
honey, and syrups.
➢ Excessive intake of added sugars is closely linked to an extensive list of
systemic conditions, including dental caries, diabetes mellitus, obesity, and
cardiovascular diseases.
Starchy Food
➢ Dietary starches refer to a variety of food rich in starch (a polymeric
carbohydrate), including bread, pasta, potatoes, potato products, rice, oats,
breakfast cereals, and other grains.
➢ Dietary starches are of low cariogenicity.
CARIOGENICITY OF SUCROSE
Sucrose is the arch criminal of dental caries, it continues to be the most
common form of added sugar in the diet .

➢ Sucrose induces the smooth surface lesion more than any other carbohydrates,
especially when treated with Streptococcus mutans.
➢ Sucrose is the only carbohydrate diet degraded to glucans.
➢ Cariogenicity of sucrose does not relate to the ability to increase plaque, but ability
of Streptococcus mutans to colonize smooth surface in the presence of sucrose.
➢ Glucans limit the diffusion of acids away from tooth surface
Vipeholm study, 1954 :

they divided the participants into nine groups depending on the type of food
consumed.
1- Sucrose in solid forms (chocolate group, bread group and caramel
group) were given in different frequency and quantity. And they were given
in between meals.
2- In caramel group, 24 toffees and 48 toffees were given to one of the
groups.
Findings
1. Significant caries increase occurred when sucrose-containing snacks were taken

between meals-time factor.

2. More the number of times sucrose was ingested more increase in caries-
frequency.
3. Sticky forms of sucrose foods, which can retain high sugar levels, were more

cariogenic than those cleaned rapidly-consistency.

4. Sucrose containing solid foods were more cariogenic than liquid types-form.
THE BASIC STEPHAN CURVE

Describes the change in dental plaque pH in


response to a challenge

The challenge could be an inert substance placed


in the mouth with the aim of determining its effect
on plaque pH.
Example

Dental plaque is challenge with


fermentable carbohydrates

Asking volunteer to rinse the


mouth with 10 ml of 10% sucrose
solution for 10 seconds.

Average plaque samples were


removed at intervals and the PH
recorded.
Characteristically the Stephan Curve reveals a rapid drop in plaque pH, followed
by a slower rise until the resting pH is attained.
The time course varies between individuals and the nature of the challenge

However, pH recovery can


take anything between 15
and 40 minutes depending to
a large extent on the acid-
neutralizing properties of the
individual’s saliva
The initial rapid drop
in pH
It is due to the speed
with which plaque
microbes are able to
metabolize sucrose.
The rise in pH

The pH starts to rise after a few minutes


due to:
1. Acid by-products diffusing out of the
plaque.
2. Salivary bicarbonate diffusing into the
plaque and neutralizing the acid by-
products.
It normally takes at least 20 minutes for the
plaque pH to reach its resting value

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