Assessment of Fetal Wellbeing
Assessment of Fetal Wellbeing
Assessment of Fetal Wellbeing
Fetal Wellbeing
Ultrasound
Most commonly used diagnostic and
screening tool
Uses a transducer
Transabdominal vs transvaginal
Uses high-freq soundwaves which produce
image based on density of object
Bone = white
Soft tissue = grey
Fluid = black
Methods
?full bladder
Abdominal
Vaginal
Ultrasound
Limited U/S
Fetal presentation
Placental location
Confirmation of viability
Determine amniotic fluid volume
Diagnose multiple gestations
Guide amniocentesis
Comprehensive U/S
Above + looks at individual structures
Ultrasound
Gestational Age
Crown-Rump (CR) length at 6-10 wks
most accurate
Later in pregnancy
Measurement of BPD, head circumference,
abd circumference, femur length
Pregnancy Wellness
Growth comparisons
Amniotic fluid volume index (AFI)
Measurement of deepest pocket of fluid in
each quadrant, added together
Placental location and status
Doppler flow studies
Estimation of fetal wt
Ultrasound
Pregnancy Wellness
Biophysical Profile
Fetal “breathing”
Fetal movement
Fetal tone
AFI
FHR acceleration (measured with NST)
Scored as 0 or 2
Highest score is 10 (with NST)
Most significant is NST and AFI
Other tests
hCG: Qualitative or Quantitative
Charts to compare values
Peaks at about 60-90 days
CVS
Small sample of chorionic villi from edge
of developing placenta
Done ~ 10-12 weeks
risk of fetal complications if done
earlier
Other tests
MSAFP
Fetal protein produced by yolk sac then
fetal liver
If NTD, AFP levels
Most accurate during 15 – 16th week
Values based on wks gestation
Detects 85% of NTDs, false + 3-4% (?)
Low levels can indicate down syndrome
or “dying” fetus
Other tests
Amniocentesis
Procedure on pg 548
Can ID genetic disorders, chromosome
disorders
Best done about 15-20 wks
Also done toward end of pregnancy for
lung maturity
Lecithin and sphingomyelin (L/S ratio) 2:1
Phosphatidylglycerol (PG) present
Other tests
5 critical factors
Birth passage
Fetus
Relationship of Maternal Pelvis and
Presenting Part
Physiologic forces of labor
Psychosocial considerations
Birth Passage
Size of pelvis
Type of pelvis
Gynecoid
Android
Arthropoid
Platypelloid
Combination
Fetus
Fetal head
Largest and least flexible
Bones of the base of the cranium are
fixed; vault (frontal, parietal, occipital)
are not
Molding
Fetus
Sutures: Lambdoidal
suture
Frontal
Sagittal
Sagittal
Coronal suture
Lambdoidal
Coronal
suture
Frontal suture
Fetal attitude
Relation of fetal parts to one another
Normal: mod flexion of head, flexion of
arms onto chest, flexion of legs onto abd
Fetal lie
Relationship of the spine (cephalocaudal
axis) of the fetus to the spine of the mom
Longitudinal: parallel
Transverse: right angle
Fetus
Fetal presentation
Body part entering the pelvis (presenting
part)
Cephalic
Breech
Shoulder
Fetus
Fetal presentation: Cephalic
☺Vertex presentation
Most common
Head completely flexed on chest
Suboccipitobregmatic (Smallest diameter)
Occiput in presenting part
Military presentation
Fetal head neither flexed nor extended
Occipitofrontal diameter presents
Top of the head is presenting part
Fetus
Phases of contractions
Increment
Acme
Decrement
Relaxation
Uterine muscle rest
Rest for mom
Restores oxygenation to baby
Physiologic forces of labor
Frequency
Duration
Intensity
Physiologic forces of labor
Intensity:
indirect: palpation: mild, mod,
strong, subjective
direct: mmHg pressure with IUPC
objective
Physiologic forces of labor
Fears
Anxiety
Social support
Past experience
Knowledge
Physiology of Labor
Lightening
Braxton-Hicks contractions
Cervical changes (ripening)
Loss of mucous plug/Bloody show
ROM
Sudden burst of energy
diarrhea
Premonitory signs of Labor
Note on ROM
If ROM at home, told to come in
In term preg, ~80% will go into
spontaneous labor w/i 24 hrs; if no labor
w/i 12? Hrs, will induce
SROM vs AROM
☺Problem if ROM before engagement
Prolapsed cord
Problem if ROM before labor
infection
True vs False Labor☺
True False
Conts Reg, ↑freq, dur, Irreg, short
intensity duration, mild
Pain Starts in back, Begins in abd
radiates to front
Cx Dilation/efface No change
change
Cont Does not decrease Decreases
change with rest or warm with rest,
bath; walking warm bath;
makes stronger walking slows
Stages of Labor ☺
Stage 1
Onset of regular contractions to
complete dilatation
Stage 2
Complete dilatation to birth
Stage 3
Birth of infant to birth of placenta
Stage 4
Birth of placenta to 1-4 hrs recovery
Stages of Labor ☺
Stage 1 divided into 3 phases
Latent phase: 0-3 cm
Primip 8.6 hrs
Multip 5.3 hrs
May have irreg contractions, short, mild –
mod
Excited, talkative, smiling
Active phase: 4-7 cm
Primip 4.6 hrs; dilation at least 1.2 cm/hr
Multip 2.4 ; dilation at least 1.5 cm/hr
u/c q 2-5 min, 40-60 sec, mod – strong
↑ anxiety, sense of hopelessness, fear of
loss of control
Stages of Labor ☺
Transition phase: 8-10 cm
Primip 3.6 hrs
Multip variable
u/c q 1 ½ - 2 min; 60-90 sec, mod – strong
Acutely aware of intensity of u/c, significant
anxiety, restless, can’t get comfortable, fears
being alone, yet may not want anyone to
touch her, hot-cold, apprehensive
Shiny schultze
Dirty duncan
Stages of Labor ☺
4th stage
Blood loss normal up to 500mL (vag del)
Hemodynamic changes ↓ BP, ↑ pulse
pressure, tachycardia
Uterus contracted and midline ~1/2 way
between symphysis and umbilicus.
Within 1st hour about level with umbilicus
Shaking, hunger, thirst
Bladder is hypotonic
Maternal Systemic Response to
Labor
Cardiovascular System
With strong u/c, stops or severely impedes
blood flow to uterus redistribution of 300-500
mL of blood to peripheral circ and ↑ in
peripheral resistance ↑ BP, ↓ pulse, ↑ CO by
~ 30%
When lying supine, CO, SV, BP and pulse ↑.
(Pushing also)
Immediately after birth, CO peaks at 80% ↑
over pre-labor then ↓s over 1st hour. Still has
elevation for ~ 24 hrs.
Maternal Systemic Response to
Labor
BP
Rises during 1st and 2nd stage
Fluid and electrolyte balance
Insensible water loss from sweating,
hyperventilation
Resp system
↑ O2 demand and consumption
Hyperventilation ↓PaCO2 and resp alkalosis
Acid base balance levels return to preg levels
by ~24 hrs; to norm w/i few weeks
Maternal Systemic Response to
Labor
Renal
↑ maternal renin, renin activity and
angiotensinogen
Polyuria is common
May have some hematuria
GI
Gastric emptying time prolonged
At risk if surgery needed
Fluids generally OK
Blood values
WBCs increased to 25-30,000
↓ glucose
Fetal Response to Labor
FHR changes: can cause
decelerations
Acid-Base: ↓ pH, ↑ PaCO2, ↓ PO2, ↑
base deficit
Hemodynamic: fetal and placental
reserves carry fetus thru anoxic
periods
Behavioral states: sleep/awake states
Fetal sensation: sensitive to light,
sound, touch
Chapter 23: Intrapartal
Nursing Assessment
Maternal Assessment
History
Obtained from mom and record
Include culture, educational needs, support
Intrapartal High Risk Screening
Excessive wt gain mult gestation
Abnormal presentation meconium fluid
Bleeding post dates
pre existing med cond drug use
Maternal Assessment
Contractions
Palpation: fundus, mild, mod, strong
Electronic: external
Uses tocodynamometer (no gel)
Placed on fundus
Measures freq and duration only
Methods of Evaluating Labor Progress
Contractions
Electronic: external
Advantages:
– Used on anyone
– Non-invasive
– Can be used intermittently
Disadvantages
– No intensity measurement
– Dependent upon women to remain fairly still
– Belt uncomfortable
– Maternal body size may make it diff to measure
contractions
Methods of Evaluating Labor Progress
Contractions
Electronic: internal
Uses IUPC
Advantages
– Accurately measures freq, duration, intensity, resting
tone
– Can use for amnioinfusion
Disadvantage
– ROM must have occurred
Methods of Evaluating Labor Progress
Cervical Assessment
VE: dilation, effacement, station,
presentation, position,
membrane status
Methods of Evaluating Fetal Status
Inspection of abd
Is the uterus longitudinal or transverse?
VE
U/S
Methods of Evaluating Fetal Status
Palpation of abd
Leopold’s maneuver
1st maneuver
– What is in fundus?
– Head is firm, round, moves independently of trunk;
buttocks is softer, symmetric, moves with trunk
2nd maneuver
– Where is the fetal back located?
– Back is firm smooth, hands and feet are irregular
3rd maneuver
– What is above the inlet (what presentation)?
4th maneuver
– Where is the brow and back of head (what position?)
Methods of Evaluating Fetal Status
Auscultation of FHR
Fetoscope, doppler, EFM
Heart tones heard best thru fetal back:
OA in lower quad
Breech above umb
Found lower and more midline as fetus descends
Listen before, during and after contraction
Intermittent monitoring has been found to be
just as effective as continuous for low risk.
Hosp policy determines who, when, how often
Methods of Evaluating Fetal Status
Variability:
Interplay between sympathetic and
parasympathetic nervous system
MOST important parameter of fetal well-being
May be decreased with fetal sleep (short-
term)
Fetal Heart Rate Pattern
Variability:
Long term variability
Rhythmic fluctuations of FHR
difference between lowest and highest FHR
Decreased: <6 BPM
Mod/avg: 6-25 BPM
Marked (saltatory): >25 BPM
Short-term variability
Difference between successive heartbeats as measured by
R-R waves.
Present or absent
Only measured with internal electrode
Indicator of fetal oxygen reserve
Fetal Heart Rate Pattern
Variability:
Sinusoidal
Indulating pattern with no short-term variability or
accels
Ominous sign
Psuedosinusoidal
Associated with med use
Fetal Heart Rate Pattern
Accelerations
15 x 15 or 10 x 10
Reassuring sign
Fetal Heart Rate Pattern
Early Decelerations
Associated with head compression
Waveform consistently uniform
Mirrors contractions
Onset is just before or early in contraction
Lowest level consistently at or before midpoint of
contraction
Range usually within 110-160
Can be single or repetitive
Benign or reassuring
Most often seen in 2nd or 3rd phase
Treatment:
NONE
Fetal Heart Rate Pattern
Late Decelerations
Associated with uteroplacental insufficiency
resulting in hypoxemia
Waveforms uniform, shape reflects
contractions
Onset is late in contraction and lowest level
consistently
after midpoint of cont (Depth not indicative of
threat)
Range usually within 110-160
Fetal Heart Rate Pattern
Late Decelerations (continued)
Can occur occasional, consistent,
gradually increasing, repetitive
May be caused by pathologic (myocardial
depression, calcified placenta, abruption) or
physiologic (supine, hypotension, tetanic
contractions)
Non-reassuring
Tx: correct cause
Position change O2
↑ fluid d/c pitocin
Fetal Heart Rate Pattern
Variable Decelerations
Associated with cord compression
Varies in onset, duration, intensity
and waveform
Generally drops and returns; abrupt with fetal insult
Onset not related to cont
May be single or repetitive
Usually benign
Tx: correct cause
VE to r/o prolapsed cord O2
Position change d/c pitocin
↑ fluid
Fetal Heart Rate Pattern
Prolonged Decelerations
Can be non-reassuring or benign
depending upon variability and
if returns to baseline
Tx: correct cause
Position change
↑ fluid
O2
d/c pitocin
Evaluation of FHR tracings
Resting tone
u/c: freq, duration, intensity
Baseline FHR: normal?
Variability: STV and LTV
Changes from baseline: accels,
decels
Reassuring or non-reassuring
Evaluation of FHR tracings
Reassuring
Within normal range
STV present
LTV average or better
Accels
Evaluation of FHR tracings
Non-Reassuring
Not within normal range
STV absent
LTV minimal or absent
Absence of accels
Prolonged decelerations
Severe bradycardia
Other methods of fetal assessment
Scalp stimulation
Acoustic stimulation
Stimulation of abdomen
Fetal scalp blood sampling (pH)
Fetal oximetry
Cord blood analysis
Chapter 24: The Family in
Childbirth: Needs and Care
When do I go to the hospital?
ROM
Regular, frequent contractions
(primip q 5min for 1 hr; miltip q 6-8
min for 1 hr) VERY GENERAL
Vaginal bleeding
Decreased (absent) fetal movement
What is going to happen to me
when I arrive?
History
Physical
Assessment during 1st Stage
NOTE: general standards; individualized for
patient status and hospital policy
Latent:
VS q 1 hr; temp q 4 hrs unless ROM,
then q 1-2 hrs
U/C status q 1 hr
Fetal heart rate status q 30 min – 1 hr
Assessment during 1st Stage
NOTE: general standards; individualized
for patient status and hospital policy
Active:
VS q 1 hr, temp q 4 hrs unless ROM,
then q 1-2 hrs
U/C status q 1 hr
Fetal heart rate status q 15 -30 min
Assessment during 1st Stage
NOTE: general standards; individualized for
patient status and hospital policy
Transition:
VS q 1 hr; temp q 4 hrs unless ROM,
then q 1-2 hrs
U/C status q 1 hr
Fetal heart rate status q 15 min
Comfort during 1st stage
Anxiety
Keep informed
Establish rapport
Express confidence
Provide support
Comfort during 1st stage
Apgar Score☺
Heart rate
Resp effort
Muscle tone
Reflex irritability
Color
Initial Newborn Care
Physical Assessment
Gross inspection
Vital signs
Temp: >97.6 (1st temp rectally)
Pulse: 110-160, may be irregular
Resp: 30-60, irreg, abd breathing; no retractions,
nasal flaring, grunting. Lungs may be “wet”
Gestational Age Assessment (Dubowitz)
Newborn Identification
ID bands
Footprints/ mother’s fingerprint
Initiation of attachment
Nursing Care During 4th Stage of
Labor
Vs, uterus, bleeding q 15 min x 1 hr
Uterus: @ U, firm, midline
Lochia: rubra, small-mod
Bladder: atonic, fills rapidly, can displace
uterus, usually to Right uterine atony
Perineum: no hematoma, some swelling,
ice
Shaking, tired, hungry, thirsty
Nursing Care During Precipitous
Birth
Nurse delivery or unattended
Same care as previously mentioned
Important to remain calm, confident
Chapter 25
Pain Management During
Labor
The What, Whens and Hows of
Pain Management
Pain in 1st stage:
Dilatation of cervix
Hypoxia of myometrial cells
Stretching of lower uterine segment
Pressure on adjacent structures
Pain in 2nd stage:
Hypoxia of myometrial cells
Distention of vagina and perineum
Pressure on adjacent structures
Pain in 3rd stage:
Uterine contractions
Cervical dilatation
The What, Whens and Hows of
Pain Management
Factors affecting response to pain:
Preparation
Culture
Fatigue and sleep deprivation
Previous experiences
anxiety
The What, Whens and Hows of
Pain Management
40-45% receive epidural anesthesia
35-40% receive analgesia
Many receive combination
Systemic Analgesics
Maintaining normal vital signs and
homeostasis are important because they
affect fetal well-being
All systemic meds cross placental barrier
Fetal liver and kidneys are inadequate to
metabolize agents
Blood-brain barrier is more permeable at time of
birth
% of blood volume flowing to brain ↑s during
uterine stress so hypoxic fetus gets larger amt of
depressant drug
Systemic Analgesics
Administration is usually when labor well
established and maternal and fetal
assessment within normal parameters
Systemic Analgesics
Narcotics
Butorphanol
agonist-antagonist
Usual dose 1-2 mg
30-40x more potent than Demerol; 7x more
potent than Morphine
Reverses analgesic effects of other opiods or
narcotics and precipitates withdrawal in drug
dependent women☺
Can cause resp depression in mom and baby
Narcan is reversal agent
Systemic Analgesics
Narcotics
Nalbuphine Hydrochloride
Same effects as Stadol
Usual dose 10 mg
Meperidine
Narcotic agonist
Usual dose 25 – 100mg
n/v big problem
Resp depression in mom and baby
Systemic Analgesics
Opiate Antagonist
Naloxone
Reverses depression and sedation from small
doses of opiates
Competes for opiate receptor sites
Has little agonistic effects so is drug of choice
when depressant is unknown because it will not
cause further depression
Resp depression can recur as it wears off
Dosage is wt based; can be given to mom or
baby
Systemic Sedatives
H1 receptor antagonists
Sedatives, anti-emetics, narcotic
potentiators
Promethazine
Hydroxazine
Diphenhydramine
Regional Analgesia and Anesthesia
Regional Anesthesia
Temporary and reversible loss of
sensation produced by injecting an
anesthetic agent into an area that brings
the agent into direct contact with the
nervous system
Epidural
Spinal
Combination
Regional Analgesia and Anesthesia
Lumbar epidural block
Local anesthetic injected into epidural space
Can be intermittent or continuous
Pain sensation vs motor sensation
Usually given when labor well established
Adv:
Good analgesia
Fully awake
Positive birth exp
Mother can rest
Disadv:
Maternal hypotension
Requires skilled persons to administer and manage
May have decreased ability to push
Regional Analgesia and Anesthesia
Lumbar epidural block
IV bolus☺
Positioned
Insertion
Care after placement
Adverse effects:
Hypotension
– Uterine displacement
– IV fluids increased
– O2
– Ephedrine
Inadequate anesthesia
Pruritis
Slight temp elevation
Regional Analgesia and Anesthesia
Spinal Block
For C/S
Anesthetic agent injected into
subarachnoid space
Immediately positioned after injection
Anesthesia is almost immediate
No direct effect on fetus
Complications:
Hypotension
Drug reaction
Spinal headache (controversial) Treatment
Total spinal block
Local Anesthesia
Pudendal Block
Anesthesia into area of pudendal nerve
Perineal anesthesia
Local
Anesthetic injected into perineum
Does not absorb systemically
General Anesthesia
May be needed for emergency
Not used as often for non-emergency
surgery
Complications:
Fetal depression
Some degree of uterine relaxation
Increased risk of gastric aspiration
Failure to establish airway
Other complications associated with resp
system
Analgesic and Anesthetic
Considerations for High Risk
PTL
Fetus more susceptible
Epidural preferred
Preeclampsia
Epidural preferred if hematology studies OK
DM
Epidural Ok
Watch closely for hypotension
Cardiac
Epidural with forceps
Bleeding
?epidural
?general
Chapter 26:
Childbirth at Risk: Intrapartal
Period
Psychologic Disorders
OP position:
Fetus must rotate 135° or occasionally
born in OP position
If born OP, increased risk of 3rd or 4th
degree laceration, broken symphysis
May use forceps or manual rotation
Positioning: knee chest, pelvic rocking
Fetal Malpresentation
Brow
Usually C/S recommended due to CPD
Perinatal morbidity and mortality:
Trauma: cerebral and neck compression; damage to
trachea and larynx
Tx: pelvimetry, oxytocin?, C/S
Face
Perinatal morbidity and mortality:
Risk of CPD and prolonged labor, fetal edema,
swelling of neck and internal structures, petechiae,
ecchymosis
Tx: C/S in no progress
Fetal Malpresentation
Breech
Most common malpresentation
Frank breech most common
Risk of cord prolapse; fetal anomolies 3x
higher
If vag del: head trauma, fetal entrapment
Tx: external version (50-60% success), if
vag del: epidural, double set-up
Fetal Malpresentation
Shoulder
Version may be attempted
C/S
Compound presentation
Macrosomia
>4500 g
Obese 3-4x more likely to have
macrosomic baby
↑risk of perineal lacerations, PPH, infection
Most significant problem is shoulder dystocia
OB emergency permanent injury of brachial plexus,
fx clavicle, asphyxia, neurologic damage
Tx:
Assessment of adequacy of pelvis
McRobert’s maneuver
Suprapubic pressure
Woods Screw maneuver
Intentional breaking of clavicle
?C/S
Multiple Gestation
Two separate ova: dizygotic
One ova: monozygotic
If division occurs
w/i 1st 72 hrs: diamnionic, dichorionic
w/i 4th-8th day: diamnionic, monochorionic
9th – 13th day: monoamnionic, monochorionic
Only 50% of pregnancies diagnosed with twins
during 1st ∆ result in birth of 2 live infants
2nd ∆ loss associated with cong anomolies,
IUGR, chromosome abnormalities, cx
incompetence, twin-twin transfusion, PTL
Multiple Gestation
Mother at risk for:
SAB
Hypertension or preeclampsia
Anemia
Hydramnios
PPROM, IUGR, incompetent cx
PPH
Malpresentation
More physical discomforts
Multiple Gestation
Tx:
U/S to diagnose amnion/chorion, follow
growth, observe for twin-twin transfusion
Frequent office visits to monitor for
problems
Likely to deliver by C/S
Abruptio Placentae
Premature separation of normally
implanted placenta from the uterine wall
Very high mortality
Cause unknown but r/t
Maternal hypertension
Maternal trauma
Cigarettes, cocaine
Short umbilical cord, high parity
More common in Caucasian and African
American than Asian or Latin American
Abruptio Placentae
Classification
O=asymptomatic, diagnosed after birth
I=mild, most common
II=mod, both mom and baby show signs
of distress
III=severe, maternal shock and fetal
death likely
Abruptio Placentae
Types
Marginal-blood passes between fetal
membranes and uterine wall and escapes
vaginally; separation at periphery of
placenta
Central-separates centrally, blood trapped
between placenta and uterine wall. No
overt bleeding
Complete-massive vaginal bleeding in
presence of almost total separation
Abruptio Placentae
Tx
Continuous EFM (if baby alive)
Develop plan for birth
Maintain CV status/tx hypovolemic
shock
Follow blood coag studies/have blood
factors available
Placenta Previa
Improperly implanted in lower uterine
segment
Types
Low lying: close proximity to os, but doesn’t
reach it
Marginal: edge of placenta at margin of the os
Partial: internal os is partially covered by
placenta
Total: internal os completely covered
Placenta Previa
Cause unknown, but associated with
Multiparity
Increased age
Defective development of blood vessels in
decidua
Defective implantation of the placenta
Prior C/S
Smoking
Recent SAB or induced AB
Large placenta
Placenta Previa
Tx
Continuous EFM
Differential diagnosis
☺No vag exam until previa r/o (U/S,
other assessments)
Care depends on amt bleeding,
gestational age, assessment of fetus
Other Placental Problems
Review p. 727
Note re: infarcts and calcifications
As placenta matures calcifications
and infarcts
Calcification more often r/t age and diabetes
Infarcts more often r/t severe preeclampsia
and smoking
Prolapsed Cord