Scientists Are Mapping The World's Largest Volcano: Advanced Reading
Scientists Are Mapping The World's Largest Volcano: Advanced Reading
Scientists Are Mapping The World's Largest Volcano: Advanced Reading
Reading
READING PASSAGE 1
You should spend about 20 minutes on Questions 1-12, which are based on Reading Passage 1 below.
(B) The team has begun making 3-D maps that offer the clearest look yet at the underwater mountain, which covers an area the
size of New Mexico. In the coming months, the maps will be refined and the data analyzed, with the ultimate goal of figuring out
how the mountain was formed.
(C) It's possible that the western edge of Tamu Massif is actually a separate mountain that formed at a different time, says
William Sager, a geologist at the University of Houston who led the expedition. That would explain some differences between
the western part of the mountain and the main body.
(D) The team also found that the massif (as such a massive mountain is known) is highly pockmarked with craters and cliffs.
Magnetic analysis provides some insight into the mountain’s genesis, suggesting that part of it formed through steady releases of
lava along the intersection of three mid-ocean ridges, while part of it is harder to explain. A working theory is that a large plume
of hot mantle rock may have contributed additional heat and material, a fairly novel idea.
(E) Tamu Massif lies about 1,000 miles (1,600 kilometers) east of Japan. It is a rounded dome, or shield volcano, measuring 280
by 400 miles (450 by 650 kilometers). Its top lies more than a mile (about 2,000 meters) below the ocean surface and is 50 times
larger than the biggest active volcano on Earth, Hawaii’s Mauna Loa. Sager published a paper in 2013 that said the main rise of
Tamu Massif is most likely a single volcano, instead of a complex of multiple volcanoes that smashed together. But he couldn’t
explain how something so big formed.
(F) The team used sonar and magnetometers (which measure magnetic fields) to map more than a million square kilometers of
the ocean floor in great detail. Sager and students teamed up with Masao Nakanishi of Japan’s Chiba University, with Sager
receiving funding support from the National Geographic Society and the Schmidt Ocean Institute.
(G) Since sharks are attracted to magnetic fields, the toothy fish “were all over our magnetometer, and it got pretty chomped up,”
says Sager. When the team replaced the device with a spare, that unit was nearly ripped off by more sharks. The magnetic field
research suggests the mountain formed relatively quickly, sometime around 145 million years ago. Part of the volcano sports
magnetic "stripes," or bands with different magnetic properties, suggesting that lava flowed out evenly from the mid-ocean ridges
over time and changed in polarity each time Earth's magnetic field reversed direction. The central part of the peak is more
jumbled, so it may have formed more quickly or through a different process.
(H) Sager isn’t sure what caused the magnetic anomalies yet, but suspects more complex forces were at work than simply
eruptions from the ridges. It’s possible a deep plume of hot rock from the mantle also contributed to the volcano’s formation, he
says. Sager hopes the analysis will also help explain about a dozen other similar features on the ocean floor, as well as add to the
overall understanding of plate tectonics.
Questions 1-8
Reading Passage 1 has eight paragraphs, A-H.
What paragraph has the following information? Write the correct letter, A-H, in boxes 1-8 on your answer sheet.
9. A large plume of……………………….. rock may have contributed additional heat and material.
10.Tamu Massif is a………………………………..or shield volcano.
11. Replacing the device with a ………………………….. didn't help, as that unit was nearly ripped off by more sharks.
12. Sager believes that the magnetic anomalies were caused by something more than……………………
from the ridges.
READING PASSAGE 2
You should spend about 20 minutes on Questions 13-28, which are based on Reading Passage 2 below.
Today we know much more how and why HIV – the virus that leads to AIDS – has become a global pandemic. Unsurprisingly,
sex workers unwittingly played a part. But no less important were the roles of trade, the collapse of colonialism, and 20th
Century sociopolitical reform.
HIV did not really appear out of nowhere, of course. It probably began as a virus affecting monkeys and apes in west central
Africa.
From there it jumped species into humans on several occasions, perhaps because people ate infected bushmeat. Some people
carry a version of HIV closely related to that seen in sooty mangabey monkeys, for instance. But HIV that came from monkeys
has not become a global problem.
We are more closely related to apes, like gorillas and chimpanzees, than we are to monkeys. But even when HIV has passed into
human populations from these apes, it has not necessarily turned into a widespread health issue.
HIV originating from apes typically belongs to a type of virus called HIV-1. One is called HIV-1 group O, and human cases are
largely confined to west Africa.
In fact, only one form of HIV has spread far and wide after jumping to humans. This version, which probably originated from
chimpanzees, is called HIV-1 group M (for "major"). More than 90% of HIV infections belong in group M. Which raises an
obvious question: what's so special about HIV-1 group M?
A study published in 2014 suggests a surprising answer: there might be nothing particularly special about group M.
It is not especially infectious, as you might expect. Instead, it seems that this form of HIV simply took advantage of events.
"Ecological rather than evolutionary factors drove its rapid spread," says Nuno Faria at the University of Oxford in the UK.
Faria and his colleagues built a family tree of HIV, by looking at a diverse array of HIV genomes collected from about 800
infected people from central Africa.
Genomes pick up new mutations at a fairly steady rate, so by comparing two genome sequences and counting the differences they
could work out when the two last shared a common ancestor. This technique is widely used, for example to establish that our
common ancestor with chimpanzees lived at least 7 million years ago.
"RNA viruses such as HIV evolve approximately 1 million times faster than human DNA," says Faria. This means the HIV
"molecular clock" ticks very fast indeed.
It ticks so fast, Faria and his colleagues found that the HIV genomes all shared a common ancestor that existed no more than 100
years ago. The HIV-1 group M pandemic probably first began in the 1920s.
Then the team went further. Because they knew where each of the HIV samples had been collected, they could place the origin of
the pandemic in a specific city: Kinshasa, now the capital of the Democratic Republic of Congo.
At this point, the researchers changed tack. They turned to historical records to work out why HIV infections in an African city in
the 1920s could ultimately spark a pandemic.
A likely sequence of events quickly became obvious. In the 1920s, DR Congo was a Belgian colony and Kinshasa – then known
as Leopoldville – had just been made the capital. The city became a very attractive destination for young working men seeking
their fortunes, and for sex workers only too willing to help them spend their earnings. The virus spread quickly through the
population.
It did not remain confined to the city. The researchers discovered that the capital of the Belgian Congo was, in the 1920s, one of
the best connected cities in Africa. Taking full advantage of an extensive rail network used by hundreds of thousands of people
each year, the virus spread to cities 900 miles (1500km) away in just 20 years.
Everything was in place for an explosion in infection rates in the 1960s.The beginning of that decade brought another change.
Belgian Congo gained its independence, and became an attractive source of employment to French speakers elsewhere in the
world, including Haiti. When these young Haitians returned home a few years later they took a particular form of HIV-1 group
M, called "subtype B", to the western side of the Atlantic.
It arrived in the US in the 1970s, just as sexual liberation and homophobic attitudes were leading to concentrations of gay men in
cosmopolitan cities like New York and San Francisco. Once more, HIV took advantage of the sociopolitical situation to spread
quickly through the US and Europe.
"There is no reason to believe that other subtypes would not have spread as quickly as subtype B, given similar ecological
circumstances," says Faria.
For instance, in 2015 there was an outbreak in the US state of Indiana, associated with drug injecting.
The US Centers for Disease Control and Prevention has been analyzing the HIV genome sequences and data about location and
time of infection, says Yonatan Grad at the Harvard School of Public Health in Boston, Massachusetts. "These data help to
understand the extent of the outbreak, and will further help to understand when public health interventions have worked."
This approach can work for other pathogens. In 2014, Grad and his colleague Marc Lipsitch published an investigation into the
spread of drug-resistant gonorrhoea across the US.
"Because we had representative sequences from individuals in different cities at different times and with different sexual
orientations, we could show the spread was from the west of the country to the east," says Lipsitch.
What's more, they could confirm that the drug-resistant form of gonorrhoea appeared to have circulated predominantly in men
who have sex with men. That could prompt increased screening in these at-risk populations, in an effort to reduce further spread.
In other words, there is real power to studying pathogens like HIV and gonorrhoea through the prism of human society.
Questions 13-20
Do the following statements agree with the information given in Reading Passage 1?
In boxes 13-20 on your answer sheet, write
14. The most important role in developing AIDS as a pandemia was played by sex workers.
19. Human DNA evolves approximately 1 million times slower than HIV.
Questions 21-28
Complete the sentences below.
Write NO MORE THAN TWO WORDS from the passage for each answer.
Write your answers in boxes 21-28 on your answer sheet.
21. Scientists can place the origin of…………………………………. in a specific city.
22. Kinshasa was a very……………………………………. for young working men and many others willing to spend their
money.
23. In just 20 years virus managed to ………………………………………. to cities 900 miles away.
24. Belgian Congo became an attractive source of employment to French speakers when it
gained…………………………………………
25. HIV has spread quickly through the US and Europe because of the …………………………………………..
26. It is said that outbreak in Indiana was associated with ………………………………………..
27. The same approach as for HIV can work for………………………………………..
28. The form of gonorrhoea that is drug-resistant appeared to have ……………………………. in men who have sex with men.
READING PASSAGE 3
You should spend about 20 minutes on Questions 29-40, which are based on Reading Passage 3 below.
“They are an amazing species, living in extreme conditions, and great swimmers. Basically they are living engineering marvels,”
says research team member Dr Pirouz Kavehpour, professor of Mechanical and Aerospace Engineering at the University of
California, Los Angeles (UCLA). Birds’ feathers are known to have hydrophobic, or non-wetting, properties. But scientists from
UCLA, University of Massachusetts Amherst and SeaWorld, wanted to know what makes Antarctic penguins’ feathers extra ice
repelling.
“What we learn here is how penguins combine oil and nano-structures on the feathers to produce this effect to perfection,”
explains Kavehpour. By analysing feathers from different penguin species, the researchers discovered Antarctic species the
gentoo penguin (Pygoscelis papua) was more superhydrophobic compared with a species found in warmer climes – the
Magellanic penguin (Spheniscus magellanicus) – whose breeding sites include Argentinian desert.
Gentoo penguins’ feathers contained tiny pores which trapped air, making the surface hydrophobic. And they were smothered
with a special preening oil, produced by a gland near the base of the tail, with which the birds cover themselves. Together, these
properties mean that in the wild, droplets of water on Antarctic penguins’ superhydrophobic feathers bead up on the surface like
spheres – formations that, according to the team, could provide geometry that delays ice formation, since heat cannot easily flow
out of the water if the droplet only has minimal contact with the surface of the feather.
“The shape of the droplet on the surface dictates the delay in freezing,” explains Kavehpour. The water droplets roll off the
penguin's feathers before they have time to freeze, the researchers propose. Penguins living in the Antarctic are highly evolved to
cope with harsh conditions: their short outer feathers overlap to make a thick protective layer over fluffier feathers which keep
them warm. Under their skin, a thick layer of fat keeps them insulated. The flightless birds spend a lot of time in the sea and are
extremely agile and graceful swimmers, appearing much more awkward on land.
Kavehpour was inspired to study Antarctic penguins’ feathers after watching the birds in a nature documentary: “I saw these
birds moving in and out of water, splashing everywhere. Yet there is no single drop of frozen ice sticking to them,” he tells BBC
Earth. His team now hopes its work could aid design of better man-made surfaces which minimise frost formation.
“I would love to see biomimicking of these surfaces for important applications, for example, de-icing of aircrafts,” says
Kavehpour. Currently, airlines spend a lot of time and money using chemical de-icers on aeroplanes, as ice can alter the vehicles’
aerodynamic properties and can even cause them to crash.
Questions 29-33
Choose the correct letter, A, B, C or D.
Write the correct letter in boxes 29-33 on your answer sheet.
A. Hydrophobic properties
B. Extra ice repelling
C. Soft structures
D. Oil structures
Questions 34-40
Write ONLY ONE WORD from the passage for each answer.