High Risk Pregnancy
High Risk Pregnancy
High Risk Pregnancy
1
HIGH-RISK PREGNANCY
A. DEMOGRAHPIC
FACTORS
1. Age
2. Weight
3. Height
B. SOCIOECONOMIC
STATUS
1.inadequate finances
2. overcrowding, poor
standard of housing, poor
hygiene
2
3. Nutritional deprivation
4. Severe social problem
5. Unplanned and
unprepared
pregnancy,specially
among adolescents
3
Adolescent Pregnancy:
Contributing Factors
Peer pressure
Self-esteem
Lack of role models
Gain attention
Media
Poverty
Rite of passage
4
Implications of Adolescent Pregnancy
Socioeconomic:
•reliance on welfare Fetal Health:
•cycle repeats itself •LBW
Maternal health:
•prematurity
•CPD
•resp complications
•PIH
•cp
•anemia
•cognitive deficits
•nut deficits
•death
mortality 5
Adolescent Pregnancy: Assessment
Risks
fundal height
# of sexual partners
knowledge of infant care/needs
family unit/support system
baseline VS/weight
6
C. OBSTETRIC HISTORY
1.Hx of infrtility or multiple gestation
2. Grand multiparity
3. prev. abortion or ectopic preg.
4. Prev losses
5. prev. operative OB,uterine or cervical
abnormalities
6.prev. abnormal labor
7
7. prev. high-risk infant
8. prev. hydatidiform mole.
D. CURRENT OB STATUS
1. Late or no prenatal; care
2. Maternal anemia
3. Rh sensitization
4.Antepartal bleeding
5.preg. Induce hypertension
8
6.Multiple gestation
7. pre-term- post term labor
8.Polyhydramnios
9. PROM
10. Fetus inappropriately large or
small,abnormality in test fetal well
being; abnormality in presentation
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E. MATERNAL MED.Hx/status
1. Cardiac or pulmonary dx
2. Metabolic ds.
3. Endocrine disoder
4. Chronic renal ds.
5.Chronic hypertension
6.Veneral and other infectious ds.
7.Major congenital disorder or rep.tract
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8. Hemoglobinopathies
9.Seizure disorder
10.Malignancy
11. Major emotional dis, mental
retardation
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Diagnostic test in high-risk
preg./prenatal det. Of fetal
well being
miscarriage
to examine the uterus and other pelvic
anatomy
to detect fetal abnormalities
13
Mid-trimester: (sometimes called the 18
to 20 week scan)
to confirm pregnancy dates
amniocentesis
to examine the fetal anatomy for
presence of abnormalities
to check the amount of amniotic fluid
15
Third trimester:
to monitor fetal growth
biophysical profile
to determine the position of a fetus
16
NON – STRESS TEST
Tocodynamometer records fetal movements and
Doppler ultrasound measures fetal heart rate to assess
fetal well-being after 28 weeks.
Nonstress test is basically performed to evaluate on how
the baby is doing inside. How often you need the test
will depend on your doctor; it can be every week or
more often till the baby is born.
17
• The test result is considered to be
normal or reactive if baby's heart beats
faster on two separate occasions during
the 20 minute span.
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• Results may also be non-reactive due
to baby's age; babies below week 32
have not hit the required maturity for
reactivity.
• The test time may be extended or the
mother may have to go for more
comprehensive tests like biophysical
profile
• If your doctor is absolutely sure that
baby is not thriving inside, the labor
may be induced
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Preparation
a. position- semi-fowler’s or left lateral
position slightly turn to the left
b. BP is check first
c. Explain
>procedure takes 30 to 60 mins long
>mother needs to activate”mark
botton”w/ each fetal movement
> does not need
hospitalization/ambulatory basis
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Who needs the nonstress
test..
• Women with preexisting medical conditions such as diabetes
• Women with pregnancy-induced medical conditions such as
hypertension
• Baby is less active than normal
• Baby is small for its age
• Amniotic fluid is either too much or too little
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• Women who have previously lost their
babies in the second half of their
pregnancies
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INTERPRETATION
A. Normal:Reactive- increased FHT
(acceleration) greater than 16 bpm /secor
more in 10 to 20 min. period w/ fetal
movement
B. Abnormal: Non reactive- No FHR
ACCELERATION WITH FETAL MOVEMENT
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INTERPRETATION
A. Normal : high –risk pregnancy
continue
b. Abnormal result: mother needs another
test, maybe biophysical profile
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OXYTOCIN CHALLENGE TEST(OTC) OR
CONTRACTION STRESS TEST (CST)
CST is based on the observation that during
contractions, blood flow to the placenta
lessens temporarily. An evaluation is done
on how the fetus handles this stress. (utero-
placental well being)
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Interpretation:
a. normal: negative – No late decelerations
of FHR w/ each of 3 contractions during a
10 minute interval
b. Abnormal: positive – with late
decelerations of FHR with 3 contractions in
10 minutes.
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E. BIOPHYSICAL PROFILE
A scoring combining ultrasound assessment of:
a. Fetal breathing
b. Fetal movement
c. Fetal tone
d. Reactivity of the heart rate
e. Amniotic fluid volume BPP could be
used to predict fetal well-being in high-risk
pregnancy
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Purpose
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EVALUATION OF FETAL
WELL-BEING
BIOPHYSICAL PROFILE
Surveillance of fetal well-being is based on 5 categories:
DETERMINATION OF SEX-CHROMATIN
Detects sex-linked disorders.( Hemophilia, Duchenne’s Muscular
Dystrophy, Color Blindness)
BILIRUBIN LEVEL
High in pregnancy then drops after 36 weeks’ gestation.
Increase in bilirubin needs evaluation for Rh incompatibility.
EVALUATION OF FETAL
WELL-BEING
URINARY/ SERUM ESTRIOL
• Assess placental functioning.
INTERPRETATION OF RESULTS:
• Sudden drop = fetal hypoxia
• Continuous drop = fetal compromise.
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SIGN MILD PREECLAMPSIA SEVERE PREECLAMPSIA
Hypertension 40 /90 or symmHg or more 160/110 or systolic increase
above the at or above the 160 or
Baseline; diastolic rise morethan 50 mmHg over
15mm Hg or more the baseline; diastolic rise
systolic inc. of 30 greater than 110 mmHg or
more on 2 readings taken 6
hours apart after bed rest
• severe dizziness
• halo vision, blind spots
• persistent vomiting
•Disorientation
Others Hypoproteinemia
Hemoconcentration
Hypernatremia
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TABLE 15–3 Deep Tendon Reflex Rating Scale
FIGURE 15–4 To elicit clonus, with the knee flexed and the leg
supported, sharply dorsiflex the foot, hold it momentarily, and then
release it. Normally the foot returns to its usual position of plantar
flexion. Clonus is present if the foot “jerks” or taps against the
examiner’s hand. If so, the number of taps or beats of clonus is recorded.
B. DIABETES MELLITUS
A chronic, metabolic disorder characterized
by a deficiency in insulin production by
islets of Langerhans resulting in improper
metabolic interaction of carbohydrates, fats.
Protein and insulin
51
RISK FACTOR
A. Family history
B. Rapid hormonal change in pregnancy
C. tumor/ infection of the pancreas
D. Obesity
E. Stress
52
Complication of dm in mother
Preeclampsia
Urinary tract infections
Future diabetes
Dystocia
Maternal mortality
Diabetic retinophaty
Diabetic nephropathy
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Fetus/ infant
Excess growth. Extra glucose will cross the
placenta, which triggers your baby's
pancreas to make extra insulin. This can
cause your baby to grow too large
(macrosomia). Very large babies are more
likely to become wedged in the birth canal,
sustain birth injuries or require a C-section
birth.
54
Low blood sugar (hypoglycemia).
Sometimes babies of mothers with
gestational diabetes develop low blood sugar
(hypoglycemia) shortly after birth because
their own insulin production is high. Severe
episodes of this problem may provoke
seizures in the baby. Prompt feedings and
sometimes an intravenous glucose solution
can return the baby's blood sugar level to
normal
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Respiratory distress syndrome. If your
baby is delivered early, respiratory distress
syndrome — a condition that makes
breathing difficult — is possible. Babies born
to women with gestational diabetes have
more breathing problems than do those born
to women without the problem, even at the
same gestational age. Babies who have
respiratory distress syndrome might need
help breathing until their lungs become
stronger.
56
Jaundice. This yellowish discoloration of the
skin and the whites of the eyes may occur if a
baby's liver isn't mature enough to break down
a substance called bilirubin, which normally
forms when the body recycles old or damaged
red blood cells. Although jaundice usually isn't
a cause for concern, careful monitoring is
important.
Type 2 diabetes later in life. Babies of
mothers who have gestational diabetes have a
higher risk of developing obesity and type 2
diabetes later in life. 57
Developmental problems. If you have
gestational diabetes, your child may have
an increased risk of problems with motor
skill development, such as walking,
jumping, or other activities that require
balance and coordination. An increased
risk of attention problems or hyperactivity
disorders also is a concern.
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CARDIAC DISEASE
This involves a variety of heart conditions
both congenital and acquired that complicate
pregnancy.
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TYPES OF SPONTANEOUS ABORTIONS
60
Spontaneous Abortion Management
61
Spontaneous Ab Mgmt, cont.
Incomplete Hospitalization
Before 14 wks – D&C + IV
Pitocin
After 14 wks – Pitocin or
Prostaglandins
Wait 3 to 5 wks for spont Ab
Missed
(93%)
Monitor for DIC
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Post Abortion Education
Bldg, cramping X 1-2 wks
vaginal rest X 1 wk
temp BID
f/u in 2 wks
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SITES OF ECTOPIC PREGNANCY
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S & S Ectopic Pregnancy
Missed Period
Abdominal Pain
Vaginal Spotting
Rupture Severe lower abd pain
↓ hCG levels
No gestational sac on U/S
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Surgical Management of
Ectopic Pregnancy
69
S&S Abruptio Placentae
•Vag bldg
(unless concealed)
•abd pain
U-act
•hemorrhage
•boardlike
abd
•late decels
•s&s shock
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Med Mgmt of Placental Abruption
Mom stable,
bleeding,
fetus immature
fetal distress
bedrest
tocolytics Emergency CS
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DIC
Placental Bleeding
Thromboplastin release
Clot formation (systemic response)
clotting factors (fibrinogen, plts, PTT, FDP)
inability to form clots
profuse bleeding 72
The Pathological Processes of Pre-eclampsia
73
S&S Pre-eclampsia
Rapid wt gain
edema of hands & face
proteinuria
hyperreflexic DTR’s
H/A, visual disturbances
epigastric pain
74
Treatment of Pre-eclampsia
75
S&S Eclampsia/HELLP Syndrome
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Treatment of Eclampsia/HELLP Syndrome
Bedrest
Meds
MgSO4
Valium or Phenobarb (if Mg not effective, not
within 2 hr of delivery)
Hydralazine (for severe ↑ B/P)
steroids to fetal lung maturity
Delivery
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Assessment: Hypertensive
Disorders of Pregnancy
Prenatal:
wt, B/P, U/A, H/A, visual disturbances
Hospitalized Ct:
daily wt
hourly u/o, dipstick urine Q4H
VS, FHR
LOC, DTR’s, H/A
clonus
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Risk Control Strategies for
Hypertensive Disorders of Pregnancy
Sz precautions
monitor for s/s Mg toxicity(RR<12, absent
DTR’s, sweating, flushing, confusion, B/P)
Ca gluconate @ BS
Mg levels
IV MgSO4 (should be “Y” connected to
another primary bag)
D/C MgSO4 for RR < 12 or absent DTR’s
renal function (30 mL/hr)
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Incompetent Cervix
S&S
•advanced cervical dilation
•low abd pressure
•bloody show
•urinary frequency
Treatment
•cerclage
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Premature Labor/Rupture of Membranes
S&S Treatment
contractions Tocolytics
cramps IV hydration
backache bedrest
diarrhea steroids, if needed
vag d/c abx, if needed
ROM
81
Nursing Care for PTL/PROM
Assessment Teaching
Thorough hx Infection Control
bleeding FMC
ROM
BPP (for PROM)
82
Postterm Pregnancy
S&S Treatment
Wt loss
uterine size
fetal surveillance
Meconium in AF NST, CST, BPP Q wk
mom monitors mvmt
Risks Induction
fetal mortality Pitocin (10-20U/L) @
cord compression 1-2 mU/min every 20-
mec asp 60 min
LGA shoulder dystocia
CS
episiotomy/laceration
depression
83
Disorders of Amniotic Fluid
Polyhydramnios Oligohydramnios
S&S Risks
uterine dist cord compression
dyspnea musculoskeletal
edema of lower extr deformities
Treatment
pulmonary hypoplasia
therapeutic
Treatment
amniocentesis amnioinfusion
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Risks of Multifetal Gestation
PIH
GDM
PPH
Anemia
UTI
PTL
Placenta previa
CS
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(Fetal) S&S Rh Incompatibility
Hyperbilirubinemia
jaundice
Kernicterus (severe neuro d.o. r/t bili)
anemia
hepatosplenomegaly
Hydrops fetalis
86
Sequence of Assessments for Rh Sensitization
Blood Test for Type & Rh Factor
Rh-positive
Rh-negative
No further testing
Indirect Coombs
- +
Repeat frequently Titer increasing
Give
RhoGAM Titer not increasing
amniocentesis ( bilirubin)
Elevated
continue to monitor No change
retest, U/S
retest prn
intrauterine transfusion or
early delivery 87
Management of Rh Incompatibility
Prenatal
•per algorithm
Prevention Postpartum
RhoGAM at 28 weeks direct Coomb’s
(unsensitized women RhoGAM to mom if
only) baby is Rh+ (within
72 hrs of birth)
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Hyperemesis Gravidarum
S&S Treatment
U/O IVF, TPN
wt loss antiemetics
ketonuria advance diet as tol
dry muc membranes
poor skin turgor
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Glucose Tolerance Test
Maternal Fetal
risk of: risk of:
PIH NTD’s
DKA Macrosomia or
Spont Ab IUGR
Polycythemia
hyperbilirubinemia
91
Treatment of Pre-existing DM
Team approach
Monitor glycosylated Hgb A
Diet: 50% carb, 20% prot, 30% fat
Insulin TID
Hourly glucoses during labor
NST’s weekly (starting at 28-30 wks)
Amnio ( lung maturity)
92
Effects of Gestational Diabetes
Maternal Effects Fetal Effects
UTI macrosomia
hydramnios hypoglycemia at
PROM/preterm labor birth
shoulder dystocia RDS
epis/lac
CS
HTN
93
Treatment of Gestational
Diabetes
technique
diet
s/s hypoglycemia
tremors, pallor, cold/clammy skin
s/s hyperglycemia
fatigue, flushed skin, thirst, dry mouth,
95
PPCM: Manifestations
dyspnea
edema, wt gain
chest pain
palpitations
jug vein distention
enlarged heart
spont ab, PTL
96
PPCM: Energy Management
Epidural
Activity restriction
Minimize anxiety
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PPCM: Cardiac Care
Meds
Sidelying, HOB ↑
Monitor VS, FHR, heart pressures
(Swan-Ganz)
Strict I/O
Assess lungs
98
PPCM: Patient Education
Avoid excessive wt gain/edema
Diet: 2200 cal, protein, NAS
rest/avoid exertion
avoid exposure to environmental
extremes
emotional stress
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Sickle Cell Disease
Maternal Effects Fetal Effects
pain IUGR/SGA
jaundice skeletal changes
Pyelonephritis
PIH/preeclampsia
leg ulcers
CHF
100
Systemic Lupus Erythematosis
Maternal effects Fetal effects
fatigue IUGR
muscle/joint pain preterm delivery
wt loss
rash
proteinuria Treatment
PIH/preeclampsia/HELLP •PO or IV Steroids
PG loss
101
AIDS
Maternal Effects Fetal Effects
vag candidiasis
Asymptomatic at birth
PID
Candidal diaper rash
genital herpes
thrush
HPV
diarrhea
PCP
recurrent bacterial
infections
FTT
dev delay
Treatment:
ZDV (zidovudine) during PG, L&D
ZDV to neonate for 6 wks
102
Which of the following socioeconomic factors
contributes to the high incidence of
adolescent pregnancy in the US?
103
Which genetic screening test for
chromosomal abnormalities provides an
older expectant couple with information
within the first trimester?
104
When caring for a woman with mild
preeclampsia, the nurse would be concerned
with which finding?
a. +4 proteinuria
b. +2 dependent edema in ankles
c. Blood pressure 156/100
d. +2 DTR’s, absent clonus
105
The nurse is preparing to infuse
magnesium sulfate to treat preeclampsia.
In implementing this order the nurse
understands the need to:
106
The primary expected outcome for care
associated with the administration of
MgSO4 would be met if the woman:
107
A primigravida at 10 weeks gestation reports
slight vaginal spotting without passage of tissue
and mild uterine cramping. When examined, no
cervical dilation is noted. The nurse caring for this
woman should:
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