Abruptio Placenta Case Scenario
Abruptio Placenta Case Scenario
Abruptio Placenta Case Scenario
CASE SCENARIO
A 25-year-old woman, gravida 2, para 1, reported to the hospital at 33 weeks of gestation
with severe abdominal pain. She had a BMI of 24 and no previous history of unfavorable
pregnancy. The abdominal pain had started three hours prior to admission, like a vague
posterior abdominal colicky pain. The patient and her husband did not relate physical efforts
prior the onset of the pain. She did not have abdominal surgery in the past. The course of
pregnancy was regular including fetal growth.
At the admission in the hospital the patient appeared pale, exhausted, unable to walk, and
maintaining an upright position. She had nausea and episodes of vomiting. At triage blood
pressure was 90–60 mmHg, heart rate was 120 beats per minute, and temperature was
37.1°C. Abdominal pain was deep, diffuse, with no signs of peritonitis or lateral renal colic.
Uterine growth was appropriate for gestational age and the organ was tender on palpation. On
vaginal examination cervix was closed and there was no vaginal bleeding or abnormal
vaginal discharges.
Ultrasonography showed a single fetus in transverse lie, with a normal amniotic fluid.
Placental location was clearly “anterior” with vast subchorionic hypoechogenic area
interpreted as a sign of abruptio placentae. Placental tissue was hyperechogenic and thicker
than normal. Fetal heart rate at ultrasound examination was 90 beats per minute. It was not
possible to visualize and interrogate uterine arteries in their typical anatomical sites by Color
Doppler and Pulsed Wave Doppler.
An emergency caesarean section under general anesthesia was performed. Abdomen was
opened by vertical incision. Peritoneal cavity contained approximately 100 milliliters of
coagulated blood. Hysterotomy was done on “the lower uterine segment.” At rupture of
membranes the amniotic fluid was clear. A male fetus in transverse lie was delivered by
breech extraction. The fetus was alive but pale and hypotonic. A large quantity of fresh clots
leaked from the uterus during the afterbirth. The placenta showed a large subchorionic
haematoma. The uterus was ischemic and extremely floppy. A superficial bleeding laceration
of the left part of the posterior wall that probably was the cause of the hemoperitoneum was
also observed. There were no myomas or uterine malformations. The posterior uterine
incision was closed by a standard two-layer suture. The uterus, after the administration of
uterotonic, was properly contracted and it was then replaced into the abdomen. The estimated
loss of blood was 1100 milliliters. The placenta was sent for pathologic examination that
confirmed the clinical diagnosis of abruptio.
Maternal conditions were stable after two hours. Haemoglobin concentration decreased from
10.8 g/dL at admission to 8.5 g/dL. White blood cell count was 19.000/milliliter, almost
halved from admission values (31.000/milliliter). C-reactive protein had always been
negative. Analysing the previous reports and ultrasound images in the patient’s pregnancy
record, it was confirmed that a posterior placental position had been observed at 22 weeks’
scan, exactly the opposite of the anterior position that has been seen by ultrasound at the time
of hospital admission.
The newborn at birth weighted 2200 grams, Apgar score was 1, 3, and 5 at 1, 5, and 10
minutes. A severe acute cerebral haemorrhage was diagnosed. The newborn was admitted at
Neonatal Intensive Care Unit (NICU), never recovered his vital functions, and died sixty days
after birth.