Casea Omsk W Fas N Pal
Casea Omsk W Fas N Pal
Casea Omsk W Fas N Pal
Supervisor:
dr. H. Oscar Djauhari, Sp.THT-KL, M.Kes
Presented by:
Cecile 2013-061-142
Florensia 2014-061-158
CASE
Mrs. X, 25 years old came to hospital with chief
complaint: difficulty of smiling and dropping
of her left corner of mouth since three days
ago. She had a history of chronic suppurative
otitis media in her left ear.
IDENTITY
Name : Mrs. X
Age : 25 years old
Occupation : housewife
Address : Jl. Cikole, Sukabumi
COMPLAINT
Chief complaint: difficulty in smilling and
dropping of her left corner of mouth
Hypertension (-)
Maxillofacial : asymmetrical
Neck : lymphadenopathy (-)
FACIAL NERVE PHYSICAL EXAMINATION
Nervus VII
Facial expression : asymmetrical, left side
of the face slightly dropped
Raising eyebrows : normal/ difficult
Closing eyes : normal/left behind
Smiling : normal/left behind
WORKING DIAGNOSIS
Chronic suppurative otitis media auris sinistra
with complication of sinistra facial nerve
paralysis.
Differential Diagnosis
Primary tumor in the middle of left ear.
Workup
Bacteria culture and sensitivity test from the
ear discharge
Audiometry test
Head CT scan
Lab : Complete blood count including
differential count of white blood cells
MEDICATION
Topical antibiotic : ofloxacin ear drops 3mg/ml, 2
x 10 drops daily for 10 – 14 days in left ear
Oral antibiotic : Amoxicillin-clavulanic acid, 2 x
500 mg p.o. for 10 – 14 days
Steroid : Prednisone, 4 X 20 mg per day p.o.
Nerve decompression
CHRONIC SUPPURATIVE OTITIS MEDIA
(CSOM)
Definition
a persistent or intermittent infected discharge
through a non-intact tympanic membrane (ie,
perforation or tympanostomy tube)
Chronic perforation of the tympanic membrane can
occur without suppuration “inactive” CSOM
Epidemiology
prevalent in developing countries and is more
common in lower socioeconomic groups in the
developed world.
CHRONIC SUPPURATIVE OTITIS MEDIA
(CSOM)
Pathogenesis
most cases: CSOM occurs as a consequence of an episode
of AOM with perforation, with subsequent failure of the
perforation to heal.
continued presence of a middle ear effusion
degeneration of the fibrous layer of the tympanic
membrane.
P aeruginosa, S aureus, and the Proteus species.
Two main mechanisms by which a chronic perforation can
lead to continuous or repeated middle ear infections:
Direct contamination from external ear because the protective
physical barrier of the tympanic membrane is lost.
The loss of “gas cushion” in preventing reflux of nasopharynx
secretion results in the increased exposure of the middle ear to
nasopharynx bacteria.
CLINICAL FINDINGS
SYMPTOMS AND SIGNS
History of otorrhea, intermittent or continuous
Hearing loss
Discharge mucopurulent or bloodstained otorrhea
Edematous mucous membrane in the middle ear
Large perforation : identify the presence of ossicular
discontinuity
SPECIAL TESTS
A swab of the discharge : for culture and sensitivity test
An audiologic evaluation
Computed tomographic (CT) scans : demonstrating bony
anatomy and are essential if an intracranial extension of
the infection is suspected.
DIFFERENTIAL DIAGNOSIS
cholesteatoma
chronic granulomatous conditions
Wegener granulomatosis
mycobacterial infection
histiocytosis X
sarcoidosis
SURGICAL MEASURES
Tympanoplasty
Tympanomastoid surgery (tympanoplasty + cortical
mastoidectomy)
COMPLICATIONS
Sequelae
Tympanosclerosis (hyalinization of tympanic membrane)
Atelectasis of tympanic membrane (grossly retracted or
collapsed tympanic membrane)
Intratemporal Complications
Mastoiditis
Petroisitis (close relationship with CN. V and CN. VI
retroorbital pain, lateral rectus palsy)
Facial Nerve Paralysis
Suppurative Labyrinthitis
Intracranial Complications
Meningitis
Intracranial abscess
Lateral sinus thrombosis
Otic Hydrocephalus
FACIAL NERVE
FACIAL PALSY
DIFFERENTIAL DIAGNOSIS OF FACIAL
PARALYSIS
THANK YOU